3 major essential elements for ruminants?
calcium
magnesium
phosphorous
4 essential trace elements for ruminants?
copper
cobalt
selenium
iodine
what is the difference between primary and secondary tract element deficiencies?
primary: levels of mineral in the diet are inadequate
secodnary: levels in the diet are normal, absorption and uptake from diet are inadequate
what are some (4) mehtods of direct supplementation?
-inclusion in compound feeds
-free access minerals
-medication of water supply
-injectable compounds
ADD/DELETE/CONCISE ON CLINICAL SIGNS OF COPPER TOXICITY!! RLLY CHANGE TH WSIGNS!
which are more susceptible to copper deficiency: cattle or sheep?
which is more common: primary or secondary copper deficiency?
cattle more susceptible
secondary more common 90-99% copper eaten passes through rumen unabsorbed.
what are some elements that antagonise copper, thus leading to a high level of secondary deficiencies in rumeninants?
-sulpher
-iron
-molybdenum
-also incurrent disease and genetics (texels get toxicity, blackface get deficiency etc)
major organ for storing copper? contains 70% of reserves
liver
-clinical signs of deficiency is only after both liver and blood levels drop
major transporter protien for copper?
caeruloplasmin
copper deficiency clinical signs: usually in young or old cattle?
young
after weaning, 3-12 mo
what are some (5) clinical signs of copper deficiency?
-depigmentation
-dry, sparse hair caot
-epiphysis thickening
-spontaneous fractures
-joint stiffness,lameness
- anaemia
-swayback
copper poisoning in sheep is usually due to no supplementation during what stage?
which sheep are more susceptible: housed or outdoor?
-no supplementation during rpegnancy
-housed=lower risk
congenital sign of copper deficiency in sheep?
usually affects front limbs or hind limbs?
swayback (aka enzoonotic ataxia)
-hind limbs
delayed swayback is usually how long after birth?
2-8 wks after birth
-less severe, precipitated by stress. progressive and irreversable
clinical signs of copper deficiency in sheep? (other than swayback)
-depigmentation of fleece
-defective keratinisation: poor wool quality
one UNSUITABLE form of copper deficiency diagnosis?
two SUITABLE forms of copper deficiency diagnosis?
UNSUITABLE: dietary levels. due to molybednum, sulphur, iron effects
SUITABLE: plasma/serum levels, liver levels, etc
copper supplemntation inclusio in concentrate feeds is banned in which animals?
banned in sheep
what are some (3) INDIRECT methods of copper supplementation
(skipped DIRECT, thats water supplementation/injectable etc…)
-application of copper salts on pasture
-minimise dietary antagonists
-genetic selection in sheep
cobalt is necessayr for the synthesis of _________
vit B12
vit B12 is necessary for two types of metabolism. name them
propionate metabolism
one-carbon metabolism
cobalt requirements: which is bigger:-sheep vs cattle requirement?
sheep requirement>cattle req
clinical signs of cobalt deficiency in lambs?
-terminal anaemia
-watery ocular discharge
-empty pot bellied appearance
-ovine white liver disesae
-hepatic enecepalopathy
-(no specific signs in adults)
clinical signs ovine white liver disease?
PM signs?
clinical sings: unresponsive neuro disease
PM: fatty infiltration of liver