Abnormal studies Flashcards

(19 cards)

1
Q

Discuss one explanation for MDD (e.g., cognitive - negative cognitive style, in onset and relapse of depression).

A

Alloy et al

Aim: To investigate the role of cognitive style in the onset and relapse of depression.

Method: Non-depressed college freshmen, some with a history of depression, were assessed for cognitive style and followed longitudinally with regular mood and cognitive processing assessments.

Results: Students with a high-risk cognitive style showed significantly higher rates of developing and relapsing into depression, alongside biased processing of emotional information.

Conclusion: A negative cognitive style is implicated in both the initial development and recurrent episodes of depression.

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2
Q

Explain a cognitive explanation of the etiology of depression (e.g., cognitive style).

A

Alloy et al

Aim: To investigate the role of cognitive style in the onset and relapse of depression.

Method: Non-depressed college freshmen, some with a history of depression, were assessed for cognitive style and followed longitudinally with regular mood and cognitive processing assessments.

Results: Students with a high-risk cognitive style showed significantly higher rates of developing and relapsing into depression, alongside biased processing of emotional information.

Conclusion: A negative cognitive style is implicated in both the initial development and recurrent episodes of depression.

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3
Q

Explain a cognitive explanation of the etiology of depression (e.g., rumination).

A

Nolen Hoeksema

Aim: To investigate the role of rumination in depressive symptoms.

Method: A large community sample of adults was interviewed multiple times over a year, with assessments of depression and rumination.

Results: Individuals experiencing or maintaining depression exhibited higher rumination scores compared to those without a history of depression or those who recovered.

Conclusion: Rumination is associated with both the presence and persistence of depressive symptoms.

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3
Q

Explain a sociocultural explanation of the etiology of depression (e.g., social factors and stressful life events)

A

Brown and Harris

Aim: To examine the link between social factors, stressful life events, and depression in women.

Method: Women from South London underwent semi-structured interviews about their daily lives and any depressive episodes, with life events independently rated for severity.

Results: Depressed women frequently experienced adverse life events, and social class influenced depression risk, with contributing factors categorized as protective, vulnerability, and provoking.

Conclusion: Social factors and life stress are significantly linked to depression, with social class playing a role in susceptibility.

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4
Q

Explain a biological explanation of the etiology of MDD (e.g., gene-environment interaction)

A

Caspi et al

Aim: To investigate the gene-environment interaction of the 5-HTT gene in relation to depression.

Method: A large sample of participants was genotyped for the 5-HTT gene, completed a stressful life events questionnaire, and were assessed for depression.

Results: Individuals with one or more short 5-HTT alleles showed more depressive symptoms and suicidal ideation when exposed to increased stressful life events.

Conclusion: The interaction between genetic predisposition and environmental stressors significantly increases the likelihood of developing depression.

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5
Q

Evaluate a research method (e.g., twin studies) in the study of the etiology of disorders

A

Kendler et al

Aim: To investigate the heritability of major depression, including gender differences and changes in influencing factors over time.

Method: A large number of twin pairs from a national registry. Telephone interviews were conducted to assess lifetime major depression using established diagnostic criteria.

Results: Higher concordance rates for major depression were found in women compared to men. Identical twins showed significantly higher concordance rates than non-identical twins.

Conclusion: Major depression shows higher heritability in women, suggesting some gender-specific genetic risk factors.

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6
Q

Discuss one explanation for eating disorders (e.g., sociocultural - media influence)

A

Becker et al

Aim: To investigate the impact of television’s introduction on disordered eating attitudes and behaviors in Fijian adolescent girls.

Method: Two groups of Fijian adolescent girls were studied using a standardized questionnaire and interviews, one before widespread TV exposure and another a few years later.

Results: The later group, exposed to television for longer, showed significantly higher rates of disordered eating attitudes and related behaviors.

Conclusion: The introduction of television appears to contribute to the adoption of Westernized beauty ideals and an increase in disordered eating among Fijian girls.

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7
Q

Evaluate a research method (e.g., longitudinal/quasi-experimental design) in the study of the etiology of disorders

A

Caspi et al

Aim: To investigate the gene-environment interaction of the 5-HTT gene in relation to depression.

Method: A large sample of participants was genotyped for the 5-HTT gene, completed a stressful life events questionnaire, and were assessed for depression.

Results: Individuals with one or more short 5-HTT alleles showed more depressive symptoms and suicidal ideation when exposed to increased stressful life events.

Conclusion: The interaction between genetic predisposition and environmental stressors significantly increases the likelihood of developing depression.

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8
Q

Discuss prevalence rates with regard to depression (e.g., in relation to social factors)

A

Brown and Harris

Aim: To examine the link between social factors, stressful life events, and depression in women.

Method: Women from South London underwent semi-structured interviews about their daily lives and any depressive episodes, with life events independently rated for severity.

Results: Depressed women frequently experienced adverse life events, and social class influenced depression risk, with contributing factors categorized as protective, vulnerability, and provoking.

Conclusion: Social factors and life stress are significantly linked to depression, with social class playing a role in susceptibility.

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9
Q

Discuss one explanation for MDD (e.g., sociocultural - life events).

A

Brown and Harris

Aim: To examine the link between social factors, stressful life events, and depression in women.

Method: Women from South London underwent semi-structured interviews about their daily lives and any depressive episodes, with life events independently rated for severity.

Results: Depressed women frequently experienced adverse life events, and social class influenced depression risk, with contributing factors categorized as protective, vulnerability, and provoking.

Conclusion: Social factors and life stress are significantly linked to depression, with social class playing a role in susceptibility.

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10
Q

Evaluate the effectiveness of treatments for MDD (e.g., comparing therapies and medication)

A

Elkin et al

Aim: To compare the effectiveness of different treatments for Major Depressive Disorder (MDD).

Method: Patients with MDD were randomly assigned to different active treatments or a placebo, receiving treatment for a set period with various symptom assessments.

Results: All active treatments were similarly effective and superior to placebo, with medication showing faster initial response and therapies offering interpersonal benefits, though long-term remission was generally low.

Conclusion: Various treatments for MDD are effective, but specific benefits and long-term remission rates vary.

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11
Q

Discuss one explanation for MDD (e.g., biological - gene-environment interaction)

A

Caspi et al

Aim: To investigate the gene-environment interaction of the 5-HTT gene in relation to depression.

Method: A large sample of participants was genotyped for the 5-HTT gene, completed a stressful life events questionnaire, and were assessed for depression.

Results: Individuals with one or more short 5-HTT alleles showed more depressive symptoms and suicidal ideation when exposed to increased stressful life events.

Conclusion: The interaction between genetic predisposition and environmental stressors significantly increases the likelihood of developing depression.

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12
Q

Discuss a biological explanation of the etiology of MDD (e.g., genetic and environmental factors).

A

Kendler et al

Aim: To investigate the heritability of major depression, including gender differences and changes in influencing factors over time.

Method: A large number of twin pairs from a national registry. Telephone interviews were conducted to assess lifetime major depression using established diagnostic criteria.

Results: Higher concordance rates for major depression were found in women compared to men. Identical twins showed significantly higher concordance rates than non-identical twins.

Conclusion: Major depression shows higher heritability in women, suggesting some gender-specific genetic risk factors.

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13
Q

Discuss prevalence rates with regard to depression (e.g., cultural differences in presentation)

A

Parker et al

Aim: To compare the self-reported symptom identification and help-seeking motivations for depression between Chinese and Caucasian patients.

Method: Depressed out-patients from Malaysian Chinese and Australian Caucasian backgrounds completed a symptom questionnaire and identified their primary reason for seeking professional help.

Results: Chinese patients were more likely to report somatic symptoms as their primary reason for seeking help, and less likely to identify cognitive/emotional symptoms, despite both groups reporting a similar number of total somatic symptoms.

Conclusion: Cultural norms influence the presentation and reporting of depressive symptoms, with Chinese culture encouraging somatic expression over emotional/cognitive acknowledgment.

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14
Q

Discuss a cognitive explanation of the etiology of depression (e.g., rumination and neural predictors).

A

Farb et al

Aim: To investigate neural predictors of depressive relapse and how formerly depressed individuals respond to emotional challenges.

Method: Formerly depressed patients and healthy controls viewed emotional movie clips while their brain activity was tracked using fMRI, and their cognitive modes and depression severity were assessed via questionnaires.

Results: Formerly depressed patients who relapsed showed increased activity in a frontal brain region when faced with sadness, and this activity was linked to higher rumination.

Conclusion: Differences in brain responses to emotional challenges, particularly increased frontal lobe activity linked to rumination, predict future depressive relapse in formerly depressed individuals.

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15
Q

Discuss a sociocultural explanation of the etiology of eating disorders (e.g., media influence)

A

Becker et al

Aim: To investigate the impact of television’s introduction on disordered eating attitudes and behaviors in Fijian adolescent girls.

Method: Two groups of Fijian adolescent girls were studied using a standardized questionnaire and interviews, one before widespread TV exposure and another a few years later.

Results: The later group, exposed to television for longer, showed significantly higher rates of disordered eating attitudes and related behaviors.

Conclusion: The introduction of television appears to contribute to the adoption of Westernized beauty ideals and an increase in disordered eating among Fijian girls.

16
Q

Discuss the role of culture in the treatment of disorders (e.g., Taoist Cognitive Therapy for GAD)

A

Chang et al

Aim: To investigate the efficacy of Taoist Cognitive Therapy (TCT) for Chinese Americans diagnosed with Generalized Anxiety Disorder (GAD).

Method: A small sample of Chinese Americans with GAD received a course of TCT, with symptom reduction and related psychological measures assessed before, during, after treatment, and at follow-up.

Results: Most GAD-only participants experienced significant reductions in anxiety and worry, with associated improvements in depression and cognitive flexibility, although daily functioning improvements varied.

Conclusion: Taoist Cognitive Therapy shows promise as an effective and culturally adaptable cognitive therapy for GAD, suggesting that cognitive therapy can be culturally sensitive.

17
Q

Discuss the role of culture (e.g., power distance and self-control) in the treatment of disorders.

A

Zhang et al

Aim: To investigate how power distance influences consumer spending and impulsive buying, particularly for “vice” products.

Method
Study 1: Participants were primed for either high or low power distance, then given a hypothetical amount of cash to purchase snack items.

Study 2: Similar priming was used, but participants were offered a mix of “vice” (unhealthy) and “virtue” (healthy) products.

Results
Study 1: High power distance individuals spent less money and bought fewer items.

Study 2: High power distance individuals spent less on “vice” products and bought fewer of them, with no significant difference for “virtue” products.

Conclusion: Power distance influences impulsive buying of “vice” products, suggesting that higher power distance is associated with greater self-control.

18
Q

Discuss the effectiveness of a biological treatment for OCD (e.g., factors influencing SSRI response).

A

Park et al

Aim: To investigate the relationship between patients’ beliefs about worry and the effectiveness of SSRI treatment for OCD.

Method: A sample of OCD patients from South Korea completed a questionnaire on their beliefs about worry before starting a four-week course of SSRI medication, with symptom reduction tracked.

Results: Younger age and a belief that “worrying helps to cope” were identified as key predictors of a slower or less effective early response to SSRI treatment.

Conclusion: Positive beliefs about worry are associated with a delayed response to SSRI treatment for OCD, suggesting they may create psychological resistance and highlighting the potential benefit of combined cognitive and pharmacological approaches.