RAH
- pulm HTN
- Congenital Heart Dz
- Tricuspid/PV dz
- PE
- RVH
LAH
- HTN
- AMI
- Mitral/Aortic valve stenosis
- LVH
Pacemaker types
- Dual/single chamber
- Fixed-rate mode
- Demand/escape mode
- Implantable cardioverters
Brugada syndrome
- congenital
- men
- asymptomatic
- tx: cardioverter-defibrillator
- SUDDEN DEATH
Wellens Syndrome
- Unstable angina
* stenosis of proximal LAD –> infarction
Long QT syndrome
- genetic/med
* asymptomatic OR syncope/seizure/sudden death
Sinus Bradycardia
- Meds
* athletes
Sinus arrythmia
*respiration
*Junctional Escape Rhythm
*Sinus arrest, so AV junction takes over (HR 40-60)
Ventricular Escape Rhythm (Idioventricular rhythm)
Ventricles pace w/o SA/AV node (20-40 HR)
*probably unconscious (brain can’t thrive)
PAC
*Increased Epi/caffeine/cocaine/albuterol/ B ag/meth/dig
*Hyperthyroid
SA/AV NODE IRRITATED
PVC
- low O2
- Hypokalemia
- Injury (infxn, MI, inflammation)
SVT =
- atrial tach
* junctional tach
SVT
Reentry Tach of AV node
SVT most important is to
Differentiate supraventricular from ventricular (Tx difference)
V tach
- ischemic heart dz
- Myocardial scar (old MI) –> ectopic electrical focus
- Hemodynamic compromise
Torsades de Pointes
two competitive, irritable foci in the ventricular myocardium
- HypoK
- HypoMg
- Hypocalcemia
- K blockers
- Congenital long QT syndrome (female)
Atrial flutter
long AV node refractory period –> stops atrial signal from going to ventricles (V rate slower than A rate)
A fib
Atria not conducting/contracting w/ any regularity
V fib
*Cardiac arrest
PEA Pulseless Electrical Activity
Dying heart generates electrical impulses (but NO PULSE)
Asystole
- death
* leads fell off
1st degree Heart block
- up vagal tone (atheletes)
- Meds (antiarrythmics)
- MI
- electrolyte disturbance
2nd Degree Heart Block type 1 (wenchebach)
- Atheletes
* electrolytes disturbance
