CCRN Multisystem Flashcards

(35 cards)

1
Q

clinical findings in neurogenic shock are related to which of the following

A

Loss of SNS innervation

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2
Q

when SNS innervation is lost what happens

A

no alpha or beta receptors function resulting in loss of vascular tone (alpha) and a decrease in heart rate, contractility, and conductivity (beta)

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3
Q

colloids do what

A

increase intravascular colloidal oncotic pressure

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4
Q

cardiac index is decreased, SVR is increased, and urine output is decreased in what form of shock

A

hypovolemic and cardiogenic

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5
Q

what differentiates hypovolemic and cardiogenic shock

A

Decreased PAOP in hypovolemic shock

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6
Q

a pulse can be felt at the radial site, the SBP is at least

A

80

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7
Q

Pulse is felt at brachial artery then the carotid; what are the SBPs

A

70, 60

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8
Q

early septic shock looks like this

A

Increased cardiac output and index, decreased RAP and PAOP, and a decrease in SVR

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9
Q

methanol causes what

A

metabolic acidosis

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10
Q

in a toxin situation, what are PNS effects

A

constricted pupils, brady cardia, hypotension, drooling

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11
Q

PNS is to

A

cholinergic

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12
Q

SNS is to

A

adrenergic

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13
Q

this shock shows, increased CO, SVR decreased, and SvO2 is increased

A

early septic shock

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14
Q

at levels greater than 10 mcg/kg/min this drug exerts pure alpha effects, causing peripheral vasoconstriction (ie cool, pale skin, slow capillary refill, and decreased UOP; what drug is it

A

dopamine

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15
Q

The RAAS system is maladaptive in which type of shock

A

cardiogenic

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16
Q

what is the preferred crystallolid in hypovolemic shock

17
Q

what is NOT a common pathophysiologic manifestation seen in patients with septic shock

A

tissue hypoxia resulting from decreased oxygen delivery

18
Q

This drug may cause QT interval prolongation and may cause torsades

A

Amitriptyline

19
Q

what is common in all forms of shock

A

decrease in cellular oxygenation

20
Q

what is an indication to stop treatment of bowel irrigation in toxic ingestions

A

the rectal effluent is clear

21
Q

to block cholinergic effects, give a what

A

anticholingergic such as atropine

22
Q

what organ system is most common to fail in severe sepsis

23
Q

exaggerated inflammation, excessive coagulation, and impaired fibrinolysis for what

24
Q

a patient with a normal serum lactate could also have

A

renal failure, due to it being an accumulation of nonvolatile acids

25
what is the difference between anaphylactoid and anaphylactic reaction
anaphylactoid reaction does not require previous exposure to the antigen
26
methanol causes what issue with regard to an ABG and why
metabolic acidosis with increased anion gap
27
what is the classic picture of early septic shock
increased CI/CO, decreased RAP and PAOP, and decreased SVR
28
what does the RAAS system do
causes vasoconstriction (increases afterload) and retention of sodium and water (increases preload) all leading to more work for the heart
29
which toxicity causes metabolic acidosis because aspirin is acetylsalicylic acid
salicylate toxicity
30
acid base imbalance in aspirin toxicity
respiratory alkalosis and metaboolic acidosis
31
T/F: renal failure patients have normal serum lactate
true
32
the most specific characteristic of distributive shocks such as septic shock is a decrease in what
SVR ( indicator for afterload)
33
the PAOP is decreased in what type of shock but elevated in which other type
hypovolemic; cardiogenic
34
why is SvO2 increased in septic shock
because the tissue has a difficult time extracting the oxygen from the blood
35
earliest indication of hepatic failure
hypoglycemia (liver performs functions of glycogenolysis and gluconeogenesis)