Chronic Dizziness Flashcards

(62 cards)

1
Q

What is the key feature of vestibular neuritis in a chronic dizzy patient?

A

A single, disabling attack of vertigo lasting several days.

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2
Q

How long does the vertigo in vestibular neuritis typically last?

A

A few days.

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3
Q

What characterizes benign paroxysmal positional vertigo (BPPV)?

A

Multiple brief episodes of rotational vertigo, typically lasting seconds, triggered by position changes such as looking up or turning over in bed.

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4
Q

How long do BPPV attacks usually last?

A

Only a few seconds.

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5
Q

What are common triggers for BPPV episodes?

A

Looking up, lying down, or turning over in bed.

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6
Q

Can BPPV recur or persist for years?

A

Yes, patients can have recurrent, undiagnosed, or untreated BPPV for decades.

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7
Q

What are the typical features of migrainous vertigo?

A

Recurrent attacks of vertigo lasting minutes to a few days (usually hours), often associated with migrainous symptoms like headache, photophobia, and visual aura.

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8
Q

Are there any abnormalities between attacks in migrainous vertigo?

A

No, there are no interictal (between attacks) abnormalities.

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9
Q

What are the characteristic features of Ménière’s disease (endolymphatic hydrops)?

A

Attacks of auditory distortion, ear fullness, tinnitus, and vertigo.

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10
Q

What is the long-term outcome of Ménière’s disease?

A

Progressive unilateral audiovestibular failure.

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11
Q

Which vertigo cause presents with both hearing and balance symptoms?

A

Ménière’s disease

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12
Q

Which vertigo cause is most likely if vertigo is brief and position-related?

A

Benign paroxysmal positional vertigo (BPPV).

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13
Q

Which cause of vertigo presents as a single, prolonged episode without hearing loss

A

Vestibular neuritis.

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14
Q

Which cause of vertigo is linked to migraine symptoms such as photophobia and aura?

A

Migrainous vertigo.

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15
Q

What are the two systems that mediate vestibular control of eye movements and perception?

A

A brainstem system and a perceptual (presumably cortical) system.

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16
Q

In acute vertigo, how are eye movements and perception related?

A

They are strongly coupled

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17
Q

In chronic vertigo, how do eye movements correlate with symptoms?

A

Poorly correlated—eye movements and symptoms often don’t match.

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18
Q

What psychological factors are often associated with long-term vestibular symptoms?

A

Anxiety and depression

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19
Q

Why is it difficult to separate chronic vestibular migraine from anxiety and depression?

A

Because the conditions are often deeply intertwined.

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20
Q

What secondary problems can develop from chronic vestibular dysfunction?

A

Neck muscle pain, stress, fatigue, and chronic anxiety.

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21
Q

How does inactivity (from bed rest, fear, or anxiety) affect vestibular compensation?

A

It delays and impairs full compensation.

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22
Q

Which tests are part of functional assessment in dizziness evaluation?

A

Romberg test, gait assessment, tandem gait, and heel-to-toe gait.

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23
Q

What is the main distinction between chronic dizziness and true gait unsteadiness?

A

Chronic dizziness is a subjective sense of imbalance (felt “in the head”), while gait unsteadiness causes objective balance problems noticeable to others.

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24
Q

How might a patient describe chronic dizziness?

A

As feeling “drunk” or light-headed, but others usually do not notice any imbalance.

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25
How might a patient with gait unsteadiness describe their symptoms?
They and others notice clear loss of balance or unsteady walking.
26
How can falls help distinguish disequilibrium from chronic dizziness?
Patients with disequilibrium tend to fall, whereas those with chronic dizziness rarely do.
27
Is MRI alone sufficient for diagnosing disequilibrium?
No. MRI is not a substitute for thorough neurological history and examination.
28
What are classic symptoms of bilateral vestibular failure (BVF)?
Oscillopsia during walking and balance problems in the dark.
29
What does “oscillopsia” mean?
The visual perception that the environment moves or bounces during head motion.
30
What are common causes of bilateral vestibular failure?
Gentamicin toxicity, meningitis, and idiopathic causes.
31
What feature helps distinguish gentamicin-induced from meningitic bilateral vestibular failure?
Gentamicin patients are typically not deaf, while meningitic patients usually are.
32
What is the typical course of idiopathic bilateral vestibular failure?
Slowly progressive balance difficulty and oscillopsia during walking, running, or riding.
33
Can bilateral vestibular failure develop after episodes of vertigo?
Yes, some patients have paroxysmal vertigo or oscillopsia that gradually progresses to chronic BVF.
34
How is bilateral vestibular failure diagnosed clinically?
With bedside tests like the head thrust (head impulse) test.
35
How do patients without vertigo or disequilibrium typically describe their symptoms?
As vague, chronic dizziness without true spinning or unsteady gait.
36
Why should orthostatic hypotension be checked in elderly dizzy patients?
Because dizziness may be due to postural drops in blood pressure, especially with antihypertensive medications.
37
What are good predictors of a cardiovascular cause for dizziness in older adults?
Syncope, light-headedness, and dizziness that improves with sitting or lying down.
38
What are predictors of a cardiovascular cause for dizziness in older adults?
Syncope, light-headedness, dizziness relieved by sitting or lying down, pallor during symptoms, precipitation by prolonged standing, and coexisting vascular disease.
39
Why may dizziness history be unreliable in the elderly?
Age-related cognitive decline can impair recall and even the perception of dizziness.
40
What is an example of a lost sensory percept in the elderly?
Older patients may not perceive dizziness during caloric testing despite strong nystagmus.
41
What common vestibular condition should always be checked for in elderly patients with vague disequilibrium?
Benign Paroxysmal Positional Vertigo (BPPV).
42
What investigations are often done for patients with vague imbalance?
Blood tests (for anaemia, thyroid or endocrine disorders, diabetes, hypoglycaemia), brain imaging, and vestibular function tests.
43
When investigations are negative and the history is non-specific, what diagnosis is often made?
Psychogenic dizziness.
44
What are symptoms suggesting hyperventilation-related dizziness?
Perioral or distal paraesthesia, anxiety episodes, and a sense of light-headedness.
45
Why is the active hyperventilation test generally unreliable?
Because hyperventilation causes dizziness in everyone
46
When is the hyperventilation test considered useful?
When the patient recognises their typical dizziness symptoms during the test.
47
What are the four main components of managing a chronic dizzy patient?
1. Treat the specific vestibular condition (e.g., BPPV, migraine). 2. Short-term nonspecific pharmacological treatment for vertigo and nausea. 3. Physical rehabilitation. 4. Information, counselling, and reassurance.
48
What is the goal of treating the specific vestibular condition?
To address the underlying cause, such as repositioning manoeuvres for BPPV or migraine therapy for migrainous vertigo.
49
Why is it important to treat migraine in patients with vertigo?
Migraine can both cause vertigo and be triggered by vertigo.
50
Why should long-term use of vestibular suppressants be avoided?
They interfere with vestibular compensation — the brain needs to sense vertigo to trigger adaptation.
51
What is the principle behind vestibular compensation?
The brain adapts to imbalance signals; to trigger this, it must experience some vertigo — “no vertigo, no compensation.”
52
What systems interact to maintain balance?
The vestibular, visual, proprioceptive, cerebellar, cortical, brainstem, and spinal systems.
53
What is vestibular compensation?
A plastic CNS process that restores functional symmetry after a unilateral vestibular lesion.
54
What is sensory substitution in vestibular loss?
When patients use visual and proprioceptive cues to compensate for reduced vestibular input.
55
What problem can arise from overreliance on visual input for balance?
Visual vertigo — dizziness triggered by visual motion (e.g., crowds, scrolling, moving patterns).
56
What is visual vertigo and how is it treated?
Dizziness due to visual motion stimulation; treated with specialised vestibular rehab including optic flow techniques.
57
What is visual vertigo?
A form of chronic dizziness worsened by visually busy environments (e.g. supermarkets, traffic, films, or moving patterns).
58
What causes visual vertigo?
Excessive visual dependence following vestibular injury (sensory substitution gone too far). Migraine is a known predisposing factor.
59
What is motorist disorientation syndrome?
A condition where patients feel that the car is tilting or veering while driving, often due to visual-vestibular mismatch.
60
What vestibular structure may be involved in symptoms during curves while driving?
The otolith system, which senses linear acceleration — damaged otoliths can cause disorientation when turning or cornering.
61
How is motorist disorientation syndrome treated?
• If vestibular origin: vestibular rehab with visuo-vestibular conflict and optic flow training. • If psychogenic origin: psychiatric therapy (CBT + medication). • If mixed: combination of both.
62
What is phobic postural vertigo?
Episodes of subjective unsteadiness in patients with normal balance on examination, often linked to anxiety or obsessive traits.