Eyelids Flashcards

(47 cards)

1
Q

List the eyelid tissue layers from anterior to posterior.

A
  1. Skin.
  2. Orbicularis muscle.
  3. Tarsal plate (fibrous).
  4. Levator muscle.
  5. Muller muscle.
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2
Q

The eyelid is anatomically divided into sections. How many and what are they called? What are they separated by?

A
  1. 2 sections.
  2. Lamellae.
  3. Gray line - outermost margin of the orbicularis muscle.
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3
Q

Where do the upper and lower lid sit in relation to the limbus?

A
  1. Upper - 2mm below the superior limbus.
  2. Lower - at the level of the inferior limbus.
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4
Q

What does the anterior lamella contain?

A

Skin and orbicularis muscle.

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5
Q

What does the posterior lamella contain?

A

Tarsal plate and conjunctiva.

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6
Q

What is the orbital septum?

A

Fibrous sheet that originates from the fascia and periosteum of the orbital rim.

As the septal sheet descends to the aperture between the upper and lower lids, it becomes thicker and forms the tarsal plates.

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7
Q

What is the function of the tarsal plates?

A

Maintain the shape of the eyelids.

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8
Q

Why are the preaponeurotic fat pads posterior to the orbital septum useful?

A

Useful for categorising the depth of lacerations. If the fat pads are visible, then it is a deep laceration involving the posterior lamella.

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9
Q

How many muscles of eyelid retraction are there? What are they?

A
  1. 3 muscles.
  2. Levator palpebrae superioris (main), Muller’s muscle and frontalis muscle.
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10
Q

Which nerve is the levator muscle innervated by?

A

CN3.

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11
Q

Which nerve innervates the frontalis muscle?

A

CN7 (facial nerve).

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12
Q

What innervates the Muller’s muscle?

A

Sympathetic innervation.

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13
Q

Which muscle is used in eyelid closure? Which nerve innervates this muscle?

A

Orbicularis oculi, CN7.

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14
Q

What are the 3 afferent limbs of the blink reflex?

A
  1. Corneal stimulus via CNV1.
  2. Light stimulus via CN2.
  3. Auditory stimulus via CN8.
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15
Q

What is the efferent limb of the blink reflex?

A

Orbicularis oculi muscle via CN7.

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16
Q

What is Bell’s phenomenon? What is a poor Bell’s phenomenon a risk factor for?

A
  1. Normal physiological finding where the globe rotates up and out during forced lid closure.
  2. Lagophthalmos - inability to fully close the lid.
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17
Q

What do the canthal tendons do? Where do they attach?

A
  1. Keep the eyelid structure stable.
  2. They attach to the tarsus of the upper and lower eyelids.
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18
Q

How many canthal tendons are there in each orbit?

A

2 - a medial and a lateral.

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19
Q

What is the palpebral fissure?

A

Gap between the upper and lower eyelid margins.

20
Q

Describe the palpebral fissure abnormality for thyroid eye disease and state the mechanism for this.

A
  1. Retraction.
  2. Muller contraction.
21
Q

Describe the palpebral fissure abnormality for Horner syndrome and state the mechanism for this.

A
  1. Ptosis.
  2. Muller relaxation.
22
Q

Describe the palpebral fissure abnormality for CN7 palsy and state the mechanism for this.

A
  1. Lagophthalmos.
  2. Orbicularis weakness.
23
Q

Describe the palpebral fissure abnormality for CN3 palsy and state the mechanism for this.

A
  1. Ptosis.
  2. Levator relaxation.
24
Q

What is the risk of lagophthalmos?

A

Exposure keratopathy.

25
What are the types of lid reconstruction (for lacerations)?
1. Direct closure - use of surrounding skin to close the excision. 2. Flaps - rotation of local skin to close the excision with intact blood supply. 3. Grafts - transplant of distant skin.
26
What is the priority order of skin graft site?
1. Other lid. 2. Pre-auricular. 3. Post-auricular. 4. Underarm.
27
Can you repair both lamellae with grafts? Why?
No because there would be no blood supply.
28
What are the techniques used for anterior lamella repair?
1. Anterior advancement - incide and stretch tissue over the laceration. 2. Transposition - move tissue from the other lid. 3. Rotation - rotated skin from the cheek. 4. Glabella - rotated diamond-shaped forehead skin.
29
What are the techniques used for posterior lamella repair?
1. Hughes flap - flap taken from the upper lid. 2. Free tarsal graft from fellow eye - tissue taken from the other eye.
30
Which lamella is repaired first in a full thickness laceration of the lid?
Posterior (conjunctiva and tarsus) first then anterior (orbicularis and skin).
31
What is blepharitis? What is the typical infection that causes it?
1. Chronic inflammation of the eyelid. 2. Typically associated with Staph aureus.
32
What is affected by anterior blepharitis?
Base of the eyelashes.
33
What are the 2 subtypes of anterior blepharitis?
1. Seborrhoeic (excessive secretions). 2. Staphylococcal (direct infection).
34
What is affected by anterior blepharitis?
Meibomian glands.
35
Which type of blepharitis has a better response to treatment? Why?
Anterior blepharitis because it occurs at the surface level.
36
Describe the presentation of blepharitis.
1. Bilateral crusting of the lids and lashes. 2. Foamy tear film and meibomian cysts are seen specifically with posterior blepharitis. 3. Lashes appear normal in posterior blepharitis.
37
What should recurrent unilateral blepharitis be investigated for?
Sebaceous cell carcinoma.
38
What is the associated condition for staphylococcal blepharitis?
Atopic dermatitis.
39
What is the associated condition for seborrhoeic blepharitis?
Seborrhoeic dermatitis.
40
What is the associated condition for posterior blepharitis?
Acne rosacea.
41
What is the management of blepharitis?
1. Lid hygiene. 2. Warm compress. 3. Topical lubrication and tetracyclines.
42
What is meibomian gland dysfunction?
Chronic disorder of the meibomian glands that overlaps with posterior blepharitis, characterised by duct obstruction and abnormal glandular secretions which result in a characteristically foamy tear film.
43
Describe the pathology of the meibomian gland dysfunction.
- Meibomian glands secrete a lipid layer which contributes to the tear film to keep it stable. - In MGD: secretion of the meibomian oil is obstructed → stagnation within glands → inflammation → staphylococcal infection → chronic inflammation and scarring.
44
What is meibomianitis? What is it associated with?
1. Type of MGD where inflammation is marked. 2. Associated with acne rosacea and worse in the mornings with thick secretions and duct inflammation.
45
Describe the presentation of meibomian gland dysfunction.
1. Foamy tear film. 2. Crusty eyelashes. 3. Gritty irritated eyes.
46
What are the investigations and results of MGD?
1. Tear film breakup time of <5 seconds → tear film instability. 2. Fluorescein staining of the cornea shows corneal epithelial damage.
47