Stuttering
Involuntary repetitions of sounds and syllables, sound prolongations, blocks
Determining Stuttering
Nonverbal Components of Stuttering
Secondary behaviors or accessory characteristics may accompany speech disfluencies.
* Include eye blinking, facial grimacing or tension, and exaggerated movements of the head, shoulders, and arms.
* Exaggerated movements are used to keep listeners engaged and to distract speakers from their speech.
* Interjected speech fragments (e.g., “…that is to say…” “…like…”) are also considered secondary characteristics.
* Secondary behaviors are adopted in an effort to reduce instances of stuttering. * Some behaviors are so habituated that they are permanently associated with stuttering. The more someone stutters, the more these behaviors occur.
Neurogenic Stuttering
Typically associated with neurological disease or trauma and is acquired after childhood. Usually seen in adults.
* Neurogenic stuttering does not improve with repeated readings or singing.
* Prognosis for neurogenic stuttering is usually poor.
Developmental Stuttering
The most common form of stuttering; begins in the preschool years.
* Developmental stuttering usually occurs on content words, whereas they can occur on function words in neurogenic stuttering.
* People who have developmental stuttering frequently exhibit secondary behaviors and fear and anxiety about speaking, whereas individuals with neurogenic stuttering do not.
* Developmental stuttering occurs on the initial syllables of words, whereas neurogenic stuttering can be more widely dispersed throughout the utterance.
* Onset of developmental stuttering is between 2 and 5 years of age and is gradual with severity increasing with age.
The developmental framework has age groups.
Younger Preschool Years 2-3
Older Preschool Years 4-6
School-Age
Older Teens and Adults
Organic Theory
Physical cause for stuttering
Behavioral Theory
Stuttering is a learned response to conditions external to the individual.
Diagnosogenic Theory
Overly concerned parents react negatively to a child’s normal speech hesitations and repetitions, causing anxiety in the child and increased stuttering.
Psychological Theory
Stuttering is a neurotic symptom treated appropriately by psychotherapy. Stuttering is highly related to emotions because anxiety can create disfluency which may cause more anxiety.
Covert Repair Hypothesis
Stuttering is a reaction to a flaw in the speech production plan. Poorly developed phonological encoding skills cause errors in the speech plan. Stuttering is a “normal” repair reaction to an abnormal phonetic plan.
Demands and Capacities
Stuttering develops when the demands to produce fluent speech exceed the child’s physical and learned capacities. Fluency depends on motor skills, language production, maturity, and cognitive development. Children who stutter presumably lack one or more of these capacities. The DCM is a tool for understanding the forces that contribute to stuttering. More demands, less abilities.
The Packman and Attanasio 3-Factor Model
Suggests that there are three factors that cause moments of stuttering: a deficit in the neural processing of language and inherent instability of the speech production system; triggers, or certain features of spoken language that are associated with greater speech motor demands that negatively affect an already unstable speech production system; and modulating factors, such as physiological arousal in an individual that can alter the threshold at which a stuttering moment occurs. Processing of language, motor abilities, physiological arousal.