-prils
Mech
-Impedes production of AII
Effect
-No vasoconstrict/ADH secretion
-sartans
Mech
-block AII receptor (AT1 type)
Effect
-no vasoconstrict/ADH secretion
-olols
Mech
-block B1 receptor
Effect
-impede SNS effects
atropine
Mech
-blocks cholerginic-muscarinic receptor
Effect
-impede PSNS effects
amlodipine
Mech
-blocks Type L Ca2+ channels IN VSM
Effect
-vasodilation
epi/norepi
Mech
-binds to beta 1 (and 2)
Effect
-ionotropic (and SNS)
digoxin
Mech
- block Na+/K+ ATPase
- increase intracellular Na+
- NCX stops working because wants to trade intracellular Ca+ for extracellular Na+
Effect
-ionotropy
furosemide
Mech
- impairs Na, Cl, K reabsorption in kidney loop
- more water into urine
Effect
-diuretic
hyrochlorothiozide
Mech
- impairs Na+ resorption in kidneys
- more water into pee
- probably weaker than furosemide
Effect
- diuretic
- other BP lowering effects
venapril/diltiazem
Mech
- block type-L Ca2+ channels in HEART
- intracellular Ca2+ decrease
Effect
-negative ionotropy
albuterol
Mech
-short acting B2 agonist
Effect
- brionchiolar smooth muscle relaxation
- easier to breathe
salmeterol
Mech
-long acting B2 agonist
Effect
- brionchiolar smooth muscle relaxation
- easier to breathe
fluticasone
Mech
-glucocorticoid receptor agonist
Effect
- anti-inflammatory
- decreased blood flow to an inflamed area
name ace inhib
-prils
name ARB’s
-sartans
name cholinergenic-muscarinic antagonist
atropine
name beta blockers
-olols
name calcium channel blockers
amlodipine
name 3 postive ionotropes
epi, norepi, digoxin
name a loop direutec
furosemide
name a thiazide diuretic
HCTZ
name two calcium channel blockers
vermapril and dilimiazem
name two beta receptor agonists
-difference?
-terols
albuterol, salmeterol
name a glucocorticoid receptor agonist
fluticasone
