What is another name for Mobitz I?
Wenckebach or 2nd-degree AV block type I.
What is the hallmark ECG feature of Mobitz I?
Progressive lengthening of the PR interval until a QRS complex is dropped.
Where does Mobitz I usually occur in the conduction system?
At the AV node.
How does the rhythm appear in Mobitz I?
Atrial rhythm regular; ventricular rhythm irregular due to dropped beats.
What happens to the PR interval after a dropped QRS in Mobitz I?
It resets to normal and begins to lengthen again.
How do the P waves look in Mobitz I?
Normal in appearance; always present.
Is the QRS complex typically narrow or wide in Mobitz I?
Narrow (<0.12s) unless there is pre-existing bundle branch block.
What common causes can trigger Mobitz I?
Myocardial infarction (esp. inferior), ischaemia, medications (beta-blockers, calcium channel blockers, digoxin).
How do you clinically remember Mobitz I versus Mobitz II?
“Mobitz I = PR gets longer, longer, drop. Mobitz II = PR constant, sudden drop.”
What symptoms might a patient with Mobitz I present with?
Often asymptomatic; if symptomatic → dizziness, syncope, palpitations.
How serious is Mobitz I compared to Mobitz II?
Mobitz I is usually benign; Mobitz II carries higher risk of complete block.
What treatment is usually required for Mobitz I if asymptomatic?
No treatment, just monitoring.
What is the first step if Mobitz I causes symptomatic bradycardia with low cardiac output?
Atropine administration.
What may be required if Mobitz I does not respond to atropine and symptoms persist?
Temporary pacing.
What happens to Mobitz I during exercise or sympathetic stimulation?
It may improve because conduction through the AV node speeds up.