Define Female Genital Mutilation.
Female genital mutilation (FGM) involves surgically changing the genitals of a female for non-medical reasons
Where is FGM common?
What are the types of FGM?
How do we identify cases of FGM?
It is important to recognise risk factors for FGM to identify and ideally prevent cases from occurring.
Two key risk factors to bear in mind are coming from a community that practise FGM and having relatives affected by FGM.
There are scenarios where it is worth considering the risk of FGM:
Women may also present with the complications of FGM.
What are the short and long term complications of FGM?
Immediate complications include:
Long term complications include:
What does the Female Genital Mutilation Act 2003 say?
How should we think of managing FGM?
How should we manage FGM in under 18s?
It is mandatory to report all cases of FGM in patients under 18 to the police.
Other services should also be contacted:
How should we manage FGM in over 18s? What is the role of de-infublation?
A de-infibulation surgical procedure may be performed by a specialist in FGM in cases of type 3 FGM. This aims to correct the narrowing or closure of the vaginal orifice, improve symptoms and try to restore normal function.
Is re-infibulation allowed?
Re-infibulation (re-closure of the vaginal orifice) could be requested after childbirth. Performing this procedure is illegal.
Define Asherman’s syndrome
Asherman’s syndrome is where adhesions (sometimes called synechiae) form within the uterus, following damage to the uterus.
What causes Asherman’s syndrome? What are the complications associated with it?
Intrauterine synechiae or adhesions are caused by trauma to the endometrium. This can result from repeated uterine cavity surgery e.g. dilatation and curettage (D&C), postpartum haemorrhage procedures and infection.
It can also occur after uterine surgery (e.g. myomectomy) or several pelvic infection (e.g. endometritis).
Endometrial curettage (scraping) can damage the basal layer of the endometrium. This damaged tissue may heal abnormally, creating scar tissue (adhesions) connecting areas of the uterus that are generally not connected. There may be adhesions binding the uterine walls together, or within the endocervix, sealing it shut.
These adhesions form physical obstructions and distort the pelvic organs, resulting in menstruation abnormalities, infertility and recurrent miscarriages.
Adhesions may be found incidentally during hysteroscopy. Asymptomatic adhesions are not classified as Asherman’s syndrome.
How does Asherman’s syndrome present?
How does Asherman’s syndrome present?
What are the investigations for Asherman’s syndrome?
What is the management of Asherman’s syndrome?
The aim of the treatment is to:
Hysteroscopic adhesiolysis - intrauterine synechia divided using hysteroscopic scissors.
Myometrial scoring – uterine reconstruction is performed using a knife electrode to increase the intrauterine dimensions.
MedEd
Surgical breakdown of intrauterine adhesions (hysteroscopic adhesiolysis) + insertion of paediatric Foley catheter or IUCD to prevent re-formation
2 cycles of cyclical oral oestrogen and progesterone given after to aid endometrial proliferation