Perio final exam notes Flashcards

(275 cards)

1
Q

1. Clinical morphology of the periodontium

Components of periodontium

A
  • Gingiva
  • Periodontal ligament
  • Cementum
  • Alveolar bone(Alveolar bone proper/bundle bone and alveolar process
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2
Q

1. Clinical morphology of the periodontium

Types of mucosa

A
  • Masticatory mucosa
  • Specialised mucosa
  • Lining mucosa
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3
Q

1. Clinical morphology of the periodontium

Masticatory mucosa covers

A

Gingiva and hard palate

Is keratinised

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4
Q

1. Clinical morphology of the periodontium

Specialised mucosa covers

A

Dorsum of tongue

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5
Q

1. Clinical morphology of the periodontium

Lining mucosa covers

A
  • Soft palate
  • Inside of lips
  • Floor of mouth
  • Vestibulum
  • Ventral surface of tongue
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6
Q

1. Clinical morphology of the periodontium

Microscopic anatomy of gingiva-types of epithelium

A
  • Oral epithelium
  • Oral sulcular epithelium
  • Junctional epithelium
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7
Q

1. Clinical morphology of the periodontium

Epithelium that faces the oral cavity

A

Oral epithelium

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8
Q

1. Clinical morphology of the periodontium

Epithelium that faces the tooth without being in contact

A

Oral sulcular epithelium

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9
Q

1. Clinical morphology of the periodontium

The epithelium that is in contact with the tooth

A

Junctional epithelium

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10
Q

1. Clinical morphology of the periodontium

Oral epithelium characteristics

A
  • Keratinised
  • Stratified
  • Squamous epithelium
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11
Q

1. Clinical morphology of the periodontium

Oral epithelium cell layers

A
  1. Stratum basale(cuboidal cells)
  2. Stratum spinosum(thickest layer)
  3. Stratum granulosum
  4. Stratum corneum(keratinised)
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12
Q

1. Clinical morphology of the periodontium

Types of connective tissue fibres

A
  • Collagen fibres
  • Reticulin fibres
  • Oxytalan fibres
  • Elastic fibres
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13
Q

1. Clinical morphology of the periodontium

Types of collagen fibres

A
  • Circular fibres
  • Dento-gingival fibres
  • Dento-periosteal fibres
  • Transeptal fibres
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14
Q

1. Clinical morphology of the periodontium

Periodontal ligament fibres

A
  • Alveolar crest fibers(ACF)
  • Horizontal fibres(HF)
  • Oblique fibres(OF)
  • Apical fibres(AF)
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15
Q

1. Clinical morphology of the periodontium

Types of cementum

A
  • Acellular-extrinsic fibre cementum
  • Cellular mixed stratified cementum
  • Cellular intrinsic fibre cementum
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16
Q

2. Mucosa around teeth

Mucosa around the teeth

A

Gingiva

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17
Q

2. Mucosa around teeth

Gingiva type of mucosa

A

Masticatory

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18
Q

2. Mucosa around teeth

Types of gingiva

A
  • Free gingiva
  • Attached gingiva
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19
Q

2. Mucosa around teeth

Free gingiva location

A

Runs from gingival margin to the free gingival groove in area of CEJ

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20
Q

2. Mucosa around teeth

Attached gingiva location

A

From free gingival groove to mucogingival junction

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21
Q

2. Mucosa around teeth

Biological width

A
  • Gingival sulcus
  • Junctional epithelium
  • Connective tissue attachment

Distance established by junctional epithelium and connective tissue attachment(inc, sulucs)

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22
Q

3.Peri-implant mucosaImplants

Normal epithelium histologically

A
  • Well keratinised gingiva and continuous w/ JE
  • Supra alveolar tissue 1mm high
  • PDL present
  • Principle connective tissue fibers attached to root cementum
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23
Q

3.Peri-implant mucosa

Peri-implant mucosa histologically

A
  • Keratinised gingiva surrounding implant
  • Thinner JE at abutment level
  • Greater biological width- 2.8mm
  • Connective tissue contact instead of attachment
  • Vascular supply to peri-implant mucosa limited due to lack of PDL
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24
Q

4. Clinical bone morphology

Two types of bone formation

A
  • Intramembranous
  • Endochondrial(Cartilage precursor)
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25
# 4. Clinical bone morphology Bone definition
* **Specialised** connective tissue * **Mineralised** organic matrix, collagenous and non collagenous **proteins** * Main component **hydroxyapatite**
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# 4. Clinical bone morphology Bone cells
* Osteoblasts * Osteoclasts
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# 4. Clinical bone morphology Classifications of bone by
* Misch * Lekholm and Zarb
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# 4. Clinical bone morphology Misch bone classification
* **D1 Dense cortical bone** * Anterior mandible * Very hard; excellent primary stability but poor vascularity (slow healing) **D2 Thick cortical bone w/dense trabecular bone** * Posterior mandible, anterior maxilla * Ideal bone for implants (best balance of strength and vascularity) * **D3 Thin cortical bone w/ dense trabecular bone** * Anterior maxilla, posterior mandible * Moderate initial stability; good vascularity **D4 Very thin cortical bone w/ low-density trabecular bone** * Posterior maxilla * Poor initial stability; highest failure risk; * may need wider or longer implants, or bone grafting
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# 4. Clinical bone morphology Lekholm and Zarb classification
* Type 1: mostly **compact** bone; * Type 2: **thick** **cortical** w/ **trabecular** core * Type 3: **thin** **cortical** w/ **more** trabecular bone; * Type 4: **thin** **cortical** w/ **low**-**density** trabecular core.
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# 5. Fundamentals of implantology-classifications, terminology Dental Implantology
* **Clinical dental speciality=>** * Studies **theoretical foundations** + **practical applications** of **implants** for the **restoration** of chewing apparatus and its functions=> * **Damaged** as a result of **partial/complete edentulation** + the associated **processes**
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# 5. Fundamentals of implantology-classifications, terminology Dental implants
* **Medical devices** made of **alloplastic** biomaterial=> * Placed **permanently** in contact with the **internal** environment of the body=> * in order to **restore** the functions and activities of the **damaged** dentition
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# 5. Fundamentals of implantology-classifications, terminology Osseointegration
* A state in which a **clinically asymptomatic, rigid fixation** of the **alloplastic** material in the bone is achieved and maintained=> * During **functional loading**
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# 5. Fundamentals of implantology-classifications, terminology List the most commonly used implant materials.
* Grade 4 commercially **pure** **titanium** (cpTi) * **Titanium**-**zirconium** alloys (Ti-Zr) * **Ti-6Al-4V.**
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# 5. Fundamentals of implantology-classifications, terminology What is the recommended minimum distance between an implant and a natural tooth?
* **1.5 mm**=> * to **preserve** lateral biologic width and **prevent** bone loss around adjacent structures.
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# 5. Fundamentals of implantology-classifications, terminology What is the expected normal bone loss around implants in the first year and annually thereafter?
* <1.5 m during the first year * **~0.2 mm** per year afterwards (typical guideline).
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# 5. Fundamentals of implantology-classifications, terminology What is the minimum inter-implant distance to preserve interproximal bone height?
**3 mm** between adjacent implants.
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# 5. Fundamentals of implantology-classifications, terminology What are recommended minimal buccal/lingual bone thicknesses around implants?
* Minimum **1 mm** buccal/lingual=> * in aesthetic zone aim for **2 mm** buccal to **preserve** emergence profile and buccal bone.
38
# 5. Fundamentals of implantology-classifications, terminology What are safe vertical distances for implant apex from vital structures?
* Minimum **1 mm** from the **nasal** **floor** and at least **2 mm** above **inferior** **alveolar** **nerve** * implant body about **5 mm** **anterior** to mental foramen.
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# 5. Fundamentals of implantology-classifications, terminology What is primary stability and what does it depend on?
* **Mechanical** stability at implant placement=> * dependent on bone **density/quality, implant shape**, and **surgical** **technique**.
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# 5. Fundamentals of implantology-classifications, terminology What is secondary stability?
* **Biological** stability achieved **after** bone healing (osseointegration)=> * influenced by **bone** **quality**, **implant** **surface**, **patient** **health**, and **loading** **protocol**.
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# 5. Fundamentals of implantology-classifications, terminology Name the two mechanisms of osseointegration.
* **Distance osteogenesis** (bone grows from existing bone toward implant) * **Contact osteogenesis** (new bone forms on implant surface).
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5. Fundamentals of implantology-classifications, terminology What are the two main forms of titanium used in implants?
1. Commercially **pure** **titanium** (cp Ti) — Grades I–IV 2. **Titanium** **alloy** (Ti-6Al-4V) — Grade V
43
# 5. Fundamentals of implantology-classifications, terminology What is the composition of Ti-6Al-4V alloy?
* Titanium: ~90% * Aluminum: 6% * Vanadium: 4%
44
# 5. Fundamentals of implantology-classifications, terminology What is the typical composition of zirconia implants?
* ZrO₂: **94–97%** * Y₂O₃ (yttria): **3–5%** (for stabilization)
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# 5. Fundamentals of implantology-classifications, terminology What are the main advantages of zirconia implants?
* Tooth-coloured (aesthetic) * Biocompatible * Corrosion-resistant
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# 5. Fundamentals of implantology-classifications, terminology What is a healing abutment and what is its function?
* A healing abutment (aka healing cap or cover screw) is **transmucosal component placed on implant**=> * allows **shaping** and **maturation** of peri-implant **soft tissues** before prosthetic procedures - Provides **biological** **seal** during healing phase. ##Footnote Used during second-stage surgery (or immediately in a one-stage approach).
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# 5. Fundamentals of implantology-classifications, terminology What is the difference between a healing abutment and a cover screw?
* Cover Screw=> * **Two**-stage surgery * Keeps implant **sealed** under gingiva during osseointegration * Healing Abutment=> * **One**-stage surgery or **after** second-stage uncovering * Transmucosal component **shaping** soft tissue for future prosthesis.
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# 5. Fundamentals of implantology-classifications, terminology When is the healing abutment placed?
* **One-stage protocol**: placed immediately after implant insertion * protruding through soft tissue. * **Two-stage protocol**: placed during second-stage surgery * after uncovering implant once osseointegration is confirmed (≈3–6 months after placement).
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# 5. Fundamentals of implantology-classifications, terminology What components are used for making an implant impression?
- **Impression coping**/**transfer**: connects to implant to record its position. - **Healing abutment:** temporarily removed. - **Implant analog:** attached to coping in impression for lab model. - **Laboratory analog:** embedded in stone model to replicate implant. - **Tray:** open or closed type, custom or stock.
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# 5. Fundamentals of implantology-classifications, terminology Describe the closed tray (indirect) impression technique.
1. **Remove** healing abutment. 2. **Place** closed-tray impression coping and hand-tighten. 3. Make **impression** w/ **PVS** or **polyether**. 4. **Remove** impression; coping **stays** in mouth. 5. **Unscrew** coping, **attach** to implant analog, and **insert** into impression. 6. **Pour** master cast. Advantages: **simple**, **quick**, **no** tray modification. Disadvantages: **less** accurate for multiple or angulated implants.
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# 5. Fundamentals of implantology-classifications, terminology Describe the open tray (direct) impression technique.
1. **Perforate** tray above implant for screw access. 2. **Remove** healing abutment and **place** open-tray coping. 3. **Try** in tray; ensure coping screw **projects** through. 4. **Inject** light-body around coping, **fill** tray w/ heavy-body, **seat** tray. 5. After set, **unscrew** coping screws through tray holes. 6. **Remove** tray w/ coping inside impression. 7. **Attach** implant analogs and pour cast. Advantages: **high** accuracy, ideal for **multiple** implants. Disadvantages: **more** **time**-consuming and **technique**-sensitive.
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# 5. Fundamentals of implantology-classifications, terminology What impression materials are used for implant impressions?
- **Addition silicone (PVS):** most accurate, dimensionally stable. - **Polyether**: excellent detail and rigidity, best for multiple implants. - **Custom trays** recommended to minimize distortion and improve accuracy.
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# 5. Fundamentals of implantology-classifications, terminology Components of implant
* Implant body * Abutment * Crown
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# 5. Fundamentals of implantology-classifications, terminology Processes to improve implant microdesign
* Additive processes * Subtractive processes
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# 5. Fundamentals of implantology-classifications, terminology Additive processes
* Modify implant structure by **adding** materials to surface
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# 5. Fundamentals of implantology-classifications, terminology Examples of additive processes
* Mineral coatings * Plasma spraying * Biocoating with growth factors * Fluoride and particulates * Cements containing Ca-phosphates * Sulphates or carbonates * Hydroxyapetite ## Footnote Hydroxyapetite shown to enhance initial bone cell adaptation or proliferation
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# 5. Fundamentals of implantology-classifications, terminology Examples of subtractive processes
* Machining * Acid etching * Sand blasting or combination
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# 6. implant tissue interface. Osseointegration Tissues of healed implant site
* Edentulous ridge covered with **masticatory mucosa**-2-3mm thick * Masticatory mucosa covered w/ **keratinised epithelium** * Outer cortical plates **lamellar bone** and enclose **cancellous bone** * Bone marrow very **vascular** ## Footnote -masticatory mucosa has connective tissue rich in collagen and fibroblasts firmly attached to bone -Cancellous bone contains trabecullae of lamellar bone embedded in bone marrow -Bone marrow contains adipocytes and pluri-potent mesenchymal cells as well
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# 6. implant tissue interface. Osseointegration Process of osseointegration
* **Clot** formation * **Ingrowth** of vessels and migration of leukocytes and mesenychmal cells * **Granulation tissue** replaced with provisional matrix * **Fibroplasia** and **angiogenesis** starts * **New bone** formation seen in **first week** of healing * Newly formed woven bone **projects** from lateral wall of cut bony bed * New bone formation also on **implant surface** * In following weeks trabeculae of woven bone **replaced** with mature bone ## Footnote -Clot formation stumulated by thrombocytes->signals->fibroblasts -Provisional matrix rich in vessels, mesenchymal cells and fibres
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# 6. implant tissue interface. Osseointegration Newly formed woven bone projects from lateral wall of cut bony bed
* Distance osteogenesis/ Appositional bone formation ## Footnote will be replaced by mature bone by remodelling
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# 6. implant tissue interface. Osseointegration New bone formation on implant surface
Contact osteogenesis ## Footnote at a distance from parent bone
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# 7. Periodontal proprioception and osteoperception What is periodontal proprioception?
* The ability of CNS to **detect** and **interpret** tooth **loading direction of force** and **micro**-**movement**=> * via **mechanoreceptors** in the periodontal ligament (PDL).
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# 7. Periodontal proprioception and osteoperception What is osseoperception?
* The ability to perceive **mechanical** stimuli through an **osseointegrated** **implant**=> * despite the **absence** of a **periodontal** **ligament**.
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# 8. Epidemiology of periodontal disease Silness & Löe Plaque Index (PI)
Measures plaque thickness at gingival margin
65
# 8. Epidemiology of periodontal disease Löe & Silness Gingival Index (GI)
Measures severity of gingival inflammation
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# 8. Epidemiology of periodontal disease Sulcus Bleeding Index (SBI – Mühlemann & Son)
Measures bleeding after probing
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# 8. Epidemiology of periodontal disease Ainamo & Bay Bleeding Index (BOP)
* Measures presence of bleeding on probing * 0 = no bleeding, 1 = bleeding * % bleeding sites reflects current gingival inflammation
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# 8. Epidemiology of periodontal disease O’Leary Plaque Control Record (PCR)
* Measures presence of plaque * % = plaque surfaces ÷ total surfaces × 100 * <20% = good plaque control * Used for patient motivation & monitoring
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# 8. Epidemiology of periodontal disease PSI (Periodontal Screening Index)
Periodontal screening by sextant ## Footnote Determines need for full periodontal charting
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# 8. Epidemiology of periodontal disease CPI (Community Periodontal Index – WHO)
Epidemiological periodontal assessment ## Footnote Used mainly for population studies, not detailed diagnosis
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# 9. Oral biofilms and calculus Stages in the formation of dental plaque
1. Acquired pellicle 2. Initial collonisation 3. Secondary colonisation 4. Maturation and biofilm growth 5. Plaque mineralisation and calcification
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# 9. Oral biofilms and calculus Acquired pellicle
* Formed **instantly** after brushing * **Salivary** and **glycoproteins** * **Thin** film that is attachment site for bacteria
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# 9. Oral biofilms and calculus Intial colonisation
* Within a few hours * **S. Sanguis, S. Oralis**
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# 9. Oral biofilms and calculus Secondary colonisation
* **24-72 hours** * More **diverse** microorganisms including **actinomyces, preveotella and fusobacterium** * Adhere to initial colonisers
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# 9. Oral biofilms and calculus Maturation and biofilm growth
* After **a few days** * Porphyromonas gingivalis, Tannerella forsythia, and Treponema denticola * Gram **negative** and **positive** bacteria
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# 9. Oral biofilms and calculus Plaque Mineralization and Calcification
* Over **longer** period * Biofilm **hardens** due to salivary **minerals**
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# 9. Oral biofilms and calculus Calculus composition
* **Inorganic** (mineral) components * **Organic** (cellular and extracellular matrix) components
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# 9. Oral biofilms and calculus Inorganic component of calculus (mineral)
* **40-60%** depending on location * **Calcium phosphate crystals** organised into four mineral phases=>**Octacalcium phosphate, hydroxyapatite, whitlockite** and **brushite**
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# 9. Oral biofilms and calculus Organic component of calculus
* **85%** cellular and **15%** extracellular matrix * **Cellular density** within calculus **very high** * Extracellular matrix mostly **proteins and lipids** ## Footnote 200,000,000 cells per milligram -Cells primarily bacterial, some fungal-candida and species of archaea
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# 10. Periodontal infection Bacterial complexes
* Green * Orange associated * A-a * Orange * Red
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# 10. Periodontal infection Bacteria in green complex
* E.corrodens * Capnocytophagia species -C.gingivalis -C.Ochracea -C.Spitigena ## Footnote E= Eikenella
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# 10. Periodontal infection Bacteria in orange associated complex
* C.rectus(Cr) * E.nodatum(En) ## Footnote C=Campylobacter E=Eubacterium
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# 10. Periodontal infection Bacteria in A-a complex
A.Actinomycetemcomitans(Aa)
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# 10. Periodontal infection Bacteria in orange complex
* P.Intermedia(Pi) * P.micra(Pm) * F. nucleatum(Fn) ## Footnote Pi=Prevotella Pm- Parvimonas Fn=Fusobacterium
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# 10. Periodontal infection Red complex bacteria
* P.gingivalis(Pg) * T.forysthia(Tf) * T.denticola ## Footnote Pg=Porphyromonas Tf=Tannerella Td-Treponema
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# 12. Pathogenesis of periodontitis Phases of Gingivitis/Periodontitis
1. **Initial** lesion (clinically healthy gingiva) 2. **Early** lesion (early stages of gingivitis) 3. **Established** lesion chronic gingivitis) 4. **Advanced** lesion (gingivitis progesses to periodontitis)
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# 13. Modifying factors of periodontitis Most important modifying factors in periodontal disease
* Diabetes * Hormonal=>Puberty, pregnancy, menopause, oral contraceptives * Tobacco smoking * Osteoporosis * HIV * Leukemia
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# 13. Modifying factors of periodontitis Medications with an effect on periodontal disease
* **CCB**- Nifidipine * **Phenytoin**-Anticonvulsant * **Cyclosporin**-Immunosuppresant
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# 13. Modifying factors of periodontitis Smoking effect on periodontal disease
* **Vasoconstriction**->False Bop * **Deeper** pocket depths-AL * **Decreased** salivary flow-xerostomia * **Fibrotic gingival appearance** w/ little inflammation and edema * **Poorer** oral hygiene * **Impaired** PMN phagocytosis * **Poorer healing**
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# 13. Modifying factors of periodontitis Diabetes effect on periodontal disease
* **Decreased** salivary flow * **Predisposition** to candidiasis, burning mouth, abcess formation * **Impaired healing**=>poor leukocyte function, reduction in growth and proliferation of CT=> * Hyperglycemic environment
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# 13. Modifying factors of periodontitis HIV effect on periodontal disease
* **Impaired immune system**=>infection susceptibility * **Dry mouth** * Red band **gingivitis** * PD bone **loss**
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# 13. Modifying factors of periodontitis Leukemia effect on periodontal disease
* Increased **bleeding** * **Paleness** of mucosa * Gingival **hyperplasia**, **petechiae** and **hemorrhages** ## Footnote Petechia=>small red or purple spot on the skin, conjunctiva, retina, and mucous membranes=>haemorrhage of capillaries
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# 14.Occlusal trauma and periodontal disease What is occlusal trauma and does it cause periodontitis?
* Injury to periodontal tissues from **excessive occlusal forces**. * **Does NOT** initiate gingivitis or periodontitis but **can accelerate destruction** if inflammation is present.
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# 14.Occlusal trauma and periodontal disease Primary vs secondary occlusal trauma?
* Primary: **excessive force** on tooth w/ **normal periodontal support** → mobility and widened PDL but no attachment loss. * Secondary: **normal** or **excessive** **force** on tooth w/ **reduced** **support** → increased mobility and faster attachment loss if inflammation present.
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# 14.Occlusal trauma and periodontal disease Acute vs chronic occlusal trauma?
* Acute: **sudden force** (e.g. high filling) → pain on biting tenderness and sudden mobility. Chronic: **gradual forces** (e.g. bruxism) → mobility, fremitus wear facets and tooth migration.
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# 14.Occlusal trauma and periodontal disease Classic radiographic sign of occlusal trauma?
Widened periodontal ligament (PDL) space ± thickened lamina dura.
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# 14.Occlusal trauma and periodontal disease Key management principle for occlusal trauma with periodontitis?
* Control inflammation first w/ **OHI** and **scaling/root planing** then **occlusal adjustment or night guard** if needed. * Exception: adjust immediately if acute high restoration.
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# 15. Non plaque induced inflammatory gingival diseases Non plaque induced factors
* Bacterial * Viral * Fungal * Gingival manifestation of immunosuppresion * Gingival lesion of genetic origin * Mucocutaneous disorders * Allergic lesions * Traumatic lesions
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# 16. Plaque induced gingival diseases-plaque induced gingivitis, Definition of plaque induced gingivitis
Inflammatory lesion from reaction between dental plaque biofilm and host immune response
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# 16. Plaque induced gingival diseases-plaque induced gingivitis, Modifying factors of plaque induced gingivitis
🔹Smoking 🔹Changed in sex hormones 🔹Malnutrition 🔹Specific diseases and conditions 🔹Systemic drugs
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# 16. Plaque induced gingival diseases-plaque induced gingivitis, Natural Plaque retentive factors
* Calculus * Caries * Orthodontic anomalies * Deep pits and fissures * Crowded teeth * Recession * Furcations * Fractures * Radices * Frenulum attachments(papilla and papilla penetrating) * Exogenous tooth discolouration * Mucogingival abnormalities(congenital or acquired defects)
102
# 16. Plaque induced gingival diseases-plaque induced gingivitis, Iatrogenic plaque retentive factors
* Caused by dental treatment * Poorly adapted restorations(crowns, bridges, fillings) * Fixed orthodontic appliances and retainers * Mouth breathing-> xerostomia/hyposalivation
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# 17. Plaque induced gingival diseases, modulated by sex hormones Types of plaque induced gingival diseases modified by sex hormones
* **Puberty** associated gingivitis * **Menstrual cycle** associated gingivitis * **Pregnancy** associated gingivitis
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# 17. Plaque induced gingival diseases, modulated by sex hormones Pregnancy effect on periodontal disease
* Increaed **plasma hormone levels**(increased bleeding, gingival overgrowth, exudate and GCF) * Estrogen affects **salivary peroxidases** * Increased growth of **seletive pathogens** * Estrogen increases **inflammation** * Estrogen affects degree of **keratinisation of CT ground substance** * **Pregnancy granuloma** can develop * **Gingivitis** in response to low plaque levels ## Footnote -salivary perioxidases active against mo -Increased risk in 2nd and 3rd trimesters
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# 17. Plaque induced gingival diseases, modulated by sex hormones Puberty effect on periodontal disease
* Steroid hormones=>increased inflammatory mediators * Gingivitis ## Footnote Gingivitis in repsonse to relatively low levels of plaque
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# 17. Plaque induced gingival diseases, modulated by sex hormones Menstrual cycle effect on periodontal disease
* Overt gingival changes rare w/ menstrual cycle * Increased exudate/inflammatory exudate * Not clinically observable
107
# 19. Plaque induced gingival diseases, modified by systemic medication Diabetes effect on Gingivitis
* Decreased **salivary flow** * Predisposition to **candidiasis**, **burning** **mouth**, **abscess** **formation** * **Impaired** **healing**=>poor leukocyte function * Reduction in **growth and proliferation of CT**=>hyperglycemic environment ## Footnote * Level of diabetic control important in determining extend of periodontal disease
108
# 19. Plaque induced gingival diseases, modified by systemic medication Leukemia effect on gingivitis
* **Abnormal proliferation of leukocytes** in blood and marrow * Increased **bleeding** * **Paleness** of mucosa * **Swollen**, **glazed**, **spongy** tissues=>deep **red** to **purple** * **Gingival** **enlargement**=>interdental papilla folllowed by marginal and attached
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# 19. Plaque induced gingival diseases, modified by systemic medication Drugs that can cause gingival enlargement
* Anticonvulsants(pheyntoin, sodium valproate) * Immunosuppressants(cyclosporin A) * Calcium channel blockers(Nifidepine, verapamil) ## Footnote -Phenytoin causes gingival enlargment in 50% of users
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# 20. Periodontitis- classification of periodontal diseases New Periodontitis classification
1. Periodontal health, gingival diseases and conditions 2. Periodontitis 3. Periodontal manifestations of systemic diseases and developmental and acquired conditions 4. Peri-implant diseases and conditions
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# 21. Necrotising periodontal disease Types of Necrotising periodontal diseases
* Necrotising gingivitis * Necrotising periodontitis * Necrotising stomatitis ## Footnote -Acute -Recurrent -Chronic
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# 21. Necrotising periodontal disease Necrotising gingivitis
* Destructive inflammatory gingival condition * **Limited to gingival tissue** w/ no loss of attachment
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# 21. Necrotising periodontal disease Necrotising Periodontitis
* Loss of attachment(gingiva, PDL, Alveolar bone)
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# 21. Necrotising periodontal disease Necrotising stomatitis
Progession of the disease beyond the MGJ
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# Necrotising periodontal diseases Acute phase treatment
* **Eliminate** disease activity=>scaling if possible * **Hydrogen** **peroxide** use * Chemical plaque control=>**CHX 0.2% x2 daily** * **Antibiotics**=>**Metronidazole** **250mg** **x3**/**day**, **penicillins**, **tetracyclines** * HIV patients=>**antimycotic**->**miconazole** * **Regular** **apppoitments** and motivation ## Footnote patient should use gentle burshing technqiue
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# 21. Necrotising periodontal disease Periodontal abscess definition
**Localised purulent inflammation** of periodontal tissues
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# 21. Necrotising periodontal disease Types of abscess
* **Gingival abscess**=>marginal gingiva and interdental tissues * **Periodontal abscess**=>infection localised to periodontal pocket=> * Destruction of PDL and alveolar bone * **Pericoronal abscess=>**associated w/ crown of partially erupted tooth * **Periapical abscess**=>Forms at root tip * **Acute** * **Chronic**
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# 21. Necrotising periodontal disease Clinical presentation of gingival abscess
* **Localised acute** inflammatory lesion * **Red, smooth** and sometimes **painful** swelling * Aetiology-**Plaque, trauma, foreign body** impaction ## Footnote Involves marginal and interdental gingiva
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# 21. Necrotising periodontal disease Causes of Periodontal abscess
* In patients w/ **untreated periodontitis** * Major cause of tooth loss * Can occur=>after **periodontal surgery, recurrent disease, tooth perforation, foreign body** impaction * **Diabetes** predisposing factor
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# 21. Necrotising periodontal disease Antibiotic options for periodontal infections
* **Amoxicillin**=>500mg-1g(loading dose) then 500mg x3/day for 3 days->re-evaluation after 3 days * **Penicillin** **allergy**=> **clindamycin**-600mg loading dose then 300mg x4/day for 3 days * **Azithromycin**-1g loading dose then 500mg x4/day for 3 days ## Footnote Re-evaluation to dettermine need for continued use
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# 23.Biological complications in implantology, peri-implant mucositis Peri-implant disease
Inflammatory process in tissues surrounding implant
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# 23.Biological complications in implantology, peri-implant mucositis Peri-implant mucositis
* **Reversible** inflammatory process=> * Soft tissue **surrounding** functioning implant
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# 23.Biological complications in implantology, peri-implant mucositis Peri-implantitis
Inflammatory process characterised by **loss of peri-implant bone**
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# 23.Biological complications in implantology, peri-implant mucositis Treatment of peri-implant mucositis
* Patient hygiene routine * Subgingival debridement * Irrigation * Antimicrobial mouthwash-CHX
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# 23.Biological complications in implantology, peri-implant mucositis Treatment of peri-implantitis
* Non surgical periodonatal therapy * GBR and bone graft techniques * Bone implant removal if bone loss extended to apical half
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# 25. X-ray examination methods in patient with periodontal disease Why are radiographs used in periodontology?
* To assess **alveolar** **bone** **loss** * **pattern** of destruction * **calculus**/**overhangs** * **furcation** involvement * **disease** **progression** and **treatment** **outcomes**. They show past destruction not current activity.
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# 25. X-ray examination methods in patient with periodontal disease Which radiograph is most important for periodontal assessment?
* **Bitewing** radiographs. * Best for **interproximal** and **crestal** bone levels. * **Vertical** **bitewings** are preferred in **periodontitis**.
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# 25. X-ray examination methods in patient with periodontal disease What are bitewing radiographs best at showing in periodontology?
* **Early** to **moderate** interproximal bone loss * **crestal** bone height * **CEJ** position * **calculus** and **overhanging** restorations.
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# 25. X-ray examination methods in patient with periodontal disease When are periapical radiographs indicated in periodontology?
* **Advanced** periodontitis assessment of **root** **morphology** * **vertical**/**angular** defects * **furcation** **involvement** * **periapical** pathology and **trauma** from occlusion.
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# 25. X-ray examination methods in patient with periodontal disease What is the role of panoramic radiographs in periodontology?
* Provides a **general** **overview** of dentition and bone levels=> * is **not** **accurate** for early or detailed periodontal diagnosis.
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# 27. Examination of patients with implant treatment Examination viva answer for assessment of implants
* “Implant examination includes **history**, **soft** **tissue** assessment, **probing** for **bleeding** and **pocket** **depth**, **mobility** testing, and **radiographic** **evaluation** of bone levels. * Healthy implants are **immobile** with **no** **bleeding** * Peri-implant mucositis **shows inflammation w/out bone loss** * while peri-implantitis shows **inflammation w/ progressive bone loss and possible mobility.**
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# 29. Diagnosis and prognosis Types of prognosis
* Good/secure * Doubtful * Irrational to treat ## Footnote -Simple therapy and secure abutments for function -Comprehensive therapy -Extracted
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# 29. Diagnosis and prognosis Factors used in different types of prognosis
* Periodontal * Endodontic * Dental * Functional
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# 29. Diagnosis and prognosis Periodontal doubtful prognosis
* Furcation involvement=1 or 2 * Angular bony defects * Horizontal bone defect reaching 2/3 of root
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# 29. Diagnosis and prognosis Periodontal irrational prognosis
* Recurrent abcess * Combined endo and periodonal lesion * Attachment loss to apex * Furcation involvement= 3
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# 29. Diagnosis and prognosis Endodontic doubtful prognosis
* Large post or screw obturations * Incomplete root canal * Peri-apical pathology
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# 29. Diagnosis and prognosis Endodontic irrational to treat prognosis
Perforations in apical half of root
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# 29. Diagnosis and prognosis Dental doubtful prognosis
* Extensive root caries * Doesn’t extend to root canal
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# 29. Diagnosis and prognosis Dental irrational to treat prognosis
* Extensive root caries=>extends to root canal * Vertical root fractures 2/3 of root * Oblique fractures into middle third of tooth
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# 29. Diagnosis and prognosis Functional irrational to treat prognosis
* Third molars only without antagonist * Grade 3 mobility * Periodontitis and caries
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# 29. Diagnosis and prognosis When re-evaluation done
6 week to 6 months
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# 29. Diagnosis and prognosis What is done during re-evaluation
* Periodontal risk assessment * Full periodontal status
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# 29. Diagnosis and prognosis Information contained in periodontal risk assessment
* Age * Number of implants * Number of sites per tooth/implant * No. of bop positive sites * No. of sites w/ pocket depths >5mm * No. of missing teeth * % alveolar bone loss * Systemic genetic diseases * Smoking(environmental factors)
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# 29. Diagnosis and prognosis Environmental factors include
* Non smoker * Former smoker * Occasional smoker * Smoker * Heavy smoker
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# 30.Treatment sequence Aim of systemic phase
1. Identify conditions, diseases and medications that **contribute to periodontal disease** 2. Identify conditions, diseases and medications that **can endanger the patient during treatment** 3. Diseases and conditions that are **dangerous to medical staff**
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# 30.Treatment sequence Number of groups of conditions, diseases and medications
3
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# 30.Treatment sequence Group 1 conditions
* Puberty * Pregnancy * Menopause
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# 30.Treatment sequence Group 1 diseases
* Diabetes * Vitamin C deficiency * Leukemia
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# 30.Treatment sequence Group 1 medications
* Immunosuppressants-Cyclosporin * CCB-Nifidipine * Anticonvulsants-Phenytoin
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# 30.Treatment sequence Group 2 conditions
Allergies(latex/asthma)
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# 30.Treatment sequence Group 2 diseases
* Endocarditis=> * Debridement may exacerbate condition
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# 30.Treatment sequence Group 2 medications
Anticoagulants(aspirin)
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# 30.Treatment sequence Group 3 conditions
Psychological
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# 30.Treatment sequence Group 3 diseases
* All infectious diseases * Viral hepatitis * Covid
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# 30.Treatment sequence Aim of hygienic phase
* To achieve a clean infection free oral cavity free of plaque and calculus * Removal of all plaque retentive factors * Extraction of irrational to treat teeth * Debridement-Scaling and root planing
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# 30.Treatment sequence Treatment sequence is according to
Ramfjord
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# 30.Treatment sequence When epithelium regenerates
10-14 days
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# 30.Treatment sequence When connective tissue regenerates
6 weeks
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# 30.Treatment sequence When bone reforms
6 weeks to 3 months
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# 30.Treatment sequence When periodontal healing takes place
up to 6 months
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# 30.Treatment sequence Aim of corrective phase
* **Restore function and health of periodontium=>** * Surgical treatment * Orthodontics * Prosthetic treatment * Conservative(RCT) * Periodontal surgery * Implant placement * Re-evaluation after surgery
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# 30.Treatment sequence Aim of maintainence phase
Prevent re-infection and disease recurrence
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# 30.Treatment sequence Maintainence phase includes
* Anamnesis * Extra and intra-oral status * Indexes * Scaling * Periodontal status * Periodontal risk assessment
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# 30.Treatment sequence How patient recall is dettermined
* High risk-3 months * Medium risk- 6 months * Low risk- 12 months
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# Mechanical control of the supragingival plaque. Brushing methods
* **Horizontal** * **Vertical** (Leonard) * **Circular** (Fones) * **Bass** (Sulcular) * **Stillman** (vibratory * **Charters** (interdental areas) * **Roll technique** * **Scrubbing** * **Modified bass/stillman**
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# Mechanical control of the supragingival plaque. Bass (circular) brushing technique
* Effective for **subgingival** plaque removal * Brush **oblique** towards apex * Filament tips directed into sulcus at **45 degree** angle * **Short strokes** w/ back and forth motion keeping brush filaments in sulcus * On **lingual** surfaces of **anteriors**-brush **vertical**
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# Mechanical control of the supragingival plaque. Stillman(vibratory) brushing technique
* **Massaging**, **stimulating** gingiva and cleaning **cervical** areas * **Recession** * Brush head **oblique** towards apex * Filaments **partly** on gingival margin and on tooth surface * **Light** pressure w/**rotary** movements ## Footnote Maintain contact of filaments w/ tooth surface
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# Mechanical control of the supragingival plaque. Charters brushing technique
* Cases w/ **receded** interdental papillae->Filaments penetrate area * Brush **oblique** w/ filaments towards **occlusal** and **insicsal** surfaces * **Light** pressure to force filaments into interproximal embrassure
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# Mechanical control of the supragingival plaque. Roll brushing technique
* Brush head in **oblique** direction towards apex * Filaments **partly** at gingival margin and on tooth surface * Sides of filaments **pressed** lightly against ginigva * brush head **rolled** over gingiva and teeth in **occlusal** direction
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# Mechanical control of the supragingival plaque. Modified bass/stillman brushing technique
* **After** activation of brush in back and forth direction-> **rolled** over gingiva and teeth in **occlusal** direction * Filaments can then **penetrate** interdentally
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# Chemical control of supragingival plaque Classification of elements that inhibit the formation of supra-gingival plaque-according to mechanism of action
* Antiadhesive * Antimicrobial * Plaque removal * Antipathogenic
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# Chemical control of supragingival plaque Antiadhesive
* Acts at **pellicule surface** * Interferes w/ bacterial **matrix** formation
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# Chemical control of supragingival plaque Antimicrobial
* Inhibits **primary** plaque proliferation * **Bactericidal**
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# Chemical control of supragingival plaque Plaque removal
* destroy bacterial matrix * **Mechanical** removal-brush combined w/**antimicrobial** mouthwash
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# Chemical control of supragingival plaque Types of antiplaque agents and medications
* Antibiotics * Enzymes * Bis bigaunide antiseptics * Quaternary ammonium compounds * Phenols+essential oils * Natural products * Flourides * Metal salts * Oxidising agents * Detergents * Alcohols * Salicylamide ##Footnote Angry Elephants Bring Quite Powerful New Fancy Magical Old Drunk Angry Scientists
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# Chemical control of supragingival plaque Antibiotics
* **Antimicrobial** * Penicillin * Vancomycin
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# Chemical control of supragingival plaque Enzymes
* **Plaque removal** * **Antimicrobial** * Protease * Lipase * Oxidase
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# Chemical control of supragingival plaque Bisguanide antiseptics
* **Antimicrobial** * Chlorhexidine
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# Chemical control of supragingival plaque Quaternary ammonium compounds
* **Antimicrobial** * Cetylpyridium chloride
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# Chemical control of supragingival plaque Phenols+essential oils
* **Antimicrobial** * **Anti-inflammatory** * Thymol * Eucalyptol * Triclosan
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# Chemical control of supragingival plaque Natual products
* **Antimicrobial** * Sanguinarine
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# Chemical control of supragingival plaque Flourides
* **Antimicrobial** * Sodium flouride
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# Chemical control of supragingival plaque Metal salts
* **Antimicrobial** * Tinto * Zinc
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# Chemical control of supragingival plaque Oxidising agents
* **Antimicrobial** * Hydrogen peroxide
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# Chemical control of supragingival plaque Detergents
* **Antimicrobial** * Sodium sulphate
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# Chemical control of supragingival plaque Alcohols
* **Inhibition** * Delmopinol
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# Non surgical therapy-manual and machine tools Hand instrumentation advantages
* **Good** tactile sensation * **Minimal** aerosal production * Modified curettes=>**increase** efficiency ## Footnote -Modified curettes- w/extended shanks =>deep pockets -Mini-bladed currettes=>narrow pockets developed to increase efficiency
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# Non surgical therapy-manual and machine tools Parts of hand instruements
* Working part(blade) * Shank * Handle
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# Non surgical therapy-manual and machine tools Curette use
**Scaling** and root **debridement**
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# Non surgical therapy-manual and machine tools Curette structure
* **Spoon** shaped blade * **Two** cutting edges * Two edges united by **rounded** toe ## Footnote Length and angulation of shank, dimensions of blade differ between different brands
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# Non surgical therapy-manual and machine tools Sickle use
**Supragingival** debridement or scaling in **shallow** pockets
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# Non surgical therapy-manual and machine tools Sickle structure
* **Curved** or **straight** blade * **Triangular** in cross section * **Two** cutting edges
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# Non surgical therapy-manual and machine tools Hoe use
* **Supragingival** scaling * Blade can be positioned at **four** different inclincations in relation to shank-**F, L, D, M**
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# Non surgical therapy-manual and machine tools Hoe structure
* **One** cutting edge * Blade turned at **100 degree** angle to shank w/ cutting edge beveled at **45 degree angle**
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# Non surgical therapy-manual and machine tools Periodontal files use
**Smoothing** roots in areas of stubborn deposits
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# Non surgical therapy-manual and machine tools Two main groups of mechanical scalers
1. **Ultrasonic** scalers 2. **Sonic** scalers ## Footnote 1. Convert **electrical** **current** into mechanical energy in form of high frequency **vibrations** 2. Use **air** **pressure** to create **mechanical** vibration
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# Non surgical therapy-manual and machine tools Vibration Frequency of sonic scalers
3000-8000 Hz
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# Non surgical therapy-manual and machine tools Vibration Frequency of Ultra-sonic scalers
18000-45000
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# Non surgical therapy-manual and machine tools Types of ultrasonic scalers
1. **Piezoelectric** scaler 2. **Magnetostrictive** scaler ## Footnote 1. **Alternating** electrical current causes **dimensional** change in handpiece-> vibrations 2. Current produces **magnetic** **field** that causes instrument **vibration**
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# Non surgical therapy-manual and machine tools Piezoelectric scaler vibration frequency
25000-30000 ## Footnote Can reach upto 45000
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# Non surgical therapy-manual and machine tools Magnetostrictive scaler vibration frequency
25000-30000 ## Footnote can reach upto 45000
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# Non surgical therapy-manual and machine tools Pattern of vibration for Piezoelectric scalers
Linear ## Footnote Only lateral side of scaling tip used
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# Non surgical therapy-manual and machine tools Pattern of vibration for Magnetostrictive scalers
Elliptical ## Footnote All surfaces of scaling tip are active
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# Non-surgical therapy - technique of instrumentation in debridement. Scaling
* Procedure that aims to remove **plaque** and **calculus** from tooth surface * **Supra** and or **subgingival** instrumentation
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# Non-surgical therapy - technique of instrumentation in debridement. Two stages of debridement
* Root scaling * Root planning
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# Non-surgical therapy - technique of instrumentation in debridement. Root scaling
* Removal of **soft** and **hard** deposits on the root * Can be done with **hand** and **ultrasonic** instruments
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# Non-surgical therapy - technique of instrumentation in debridement. Root planning
Removal of **necrotic** **cementum** and **dentine** to achieve **smooth** root
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# Non-surgical therapy - technique of instrumentation in debridement. Optimal angle between cutting edge of curette and tooth
80 degrees
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# Non-surgical therapy - technique of instrumentation in debridement. Obtuse angle when using curette
**Cratering** and **roughening** of root surface
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# Non-surgical therapy - technique of instrumentation in debridement. Acute angle when using curette
**Ineffective** removal and **burnishing** of subgingival calculus deposits
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# Non-surgical therapy - technique of instrumentation in debridement. Procedure for use of curette
* LA * Check root surface w/ probe * Instrument held in **modified** **pen** **grasp** and **inserted** into pocket * On insertion- face of blade **parallel** to and in **light** **contact** w/ root surface * Proper **finger** **rest**(fulcrum) * After insertion to base of pocket-instrument **turned** to cutting position(shank **parallel** to long axis of root) * Blade moved in **coronal** direction * Probe inserted again to check for calculus
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# 37. Non-surgical therapy Aim of non surgical therapy
* **Eliminate biofilm** from tooth surfaces * Create environment that enables host to **prevent microbial colonisation** w/ oral hygiene methods
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# 37. Non-surgical therapy Methods used in non surgical therapy
* Hand instruments * Ultrasonic and sonic scalers * Ablative laser therapy * Reciprocating instruments
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# 38. Gingivectomy and gingivoplasty What is a gingivectomy?
**Surgical removal** of gingival tissue to eliminate **periodontal pockets.**
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# 38. Gingivectomy and gingivoplasty What is gingivoplasty?
**Surgical reshaping of gingiva** to create normal physiological contours.
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# 38. Gingivectomy and gingivoplasty What is the key difference between gingivectomy and gingivoplasty?
Gingivectomy removes tissue to eliminate pockets while gingivoplasty reshapes gingiva for contour.
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# 38. Gingivectomy and gingivoplasty What is the main indication for gingivectomy?
* **Suprabony** periodontal pockets. * **Gingival enlargement**=> inflammatory or drug induced.
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# 38. Gingivectomy and gingivoplasty When is gingivoplasty indicated?
For **reshaping** **gingival** **contour** and **aesthetic** improvement.
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# 38. Gingivectomy and gingivoplasty What is a major contraindication for gingivectomy?
Poor plaque control.
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# 38. Gingivectomy and gingivoplasty What are advantages of gingivectomy?
* Pocket elimination * improved access for cleaning and reduced inflammation.
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# Surgical methods of access - ENAP, apically displaced flap, modified tec What does ENAP stand for?
Excisional New Attachment Procedure.
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# Surgical methods of access - ENAP, apically displaced flap, modified tec What is the main goal of ENAP?
To **remove pocket epithelium** and promote **new** **attachment**.
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# Surgical methods of access - ENAP, apically displaced flap, modified tec What type of incision is used in ENAP?
Internal bevel incision.
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# Surgical methods of access - ENAP, apically displaced flap, modified tec What is removed during ENAP?
**Diseased** pocket epithelium and **inflamed** tissue.
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# Surgical methods of access - ENAP, apically displaced flap, modified tec When is ENAP indicated?
* **Moderate** periodontal pockets * need for **new** **attachment** w/ minimal gingival removal.
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# Surgical methods of access - ENAP, apically displaced flap, modified tec What is an apically displaced flap?
A periodontal flap repositioned **apically** to **reduce** **pocket** **depth**.
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# Surgical methods of access - ENAP, apically displaced flap, modified tec What is the main disadvantage of an apically displaced flap?
Root exposure and sensitivity.
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# Surgical methods of access - ENAP, apically displaced flap, modified tec What is the modified Widman flap?
A **conservative periodontal flap** surgery for **access** and **root** **debridement**.
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# Surgical methods of access - ENAP, apically displaced flap, modified tec What is the main goal of the modified Widman flap?
Access for **scaling** and **root** **planing** w/ minimal gingival removal.
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# Surgical methods of access - ENAP, apically displaced flap, modified tec How many incisions are used in the modified Widman flap?
Three incisions.
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# Surgical methods of access - ENAP, apically displaced flap, modified tec What is the first incision in modified Widman flap?
Internal bevel incision.
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# Surgical methods of access - ENAP, apically displaced flap, modified tec What is the second incision in modified Widman flap?
Crevicular incision to the bone.
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# Surgical methods of access - ENAP, apically displaced flap, modified tec What is the third incision in modified Widman flap?
Interdental incision to remove pocket lining.
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# Surgical methods of access - ENAP, apically displaced flap, modified tec What happens after flap reflection in modified Widman flap?
* Granulation tissue removal * scaling and root planing.
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# Surgical methods of access - ENAP, apically displaced flap, modified tec How is the flap positioned after modified Widman flap surgery?
Repositioned to its original position and sutured.
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# Treatment of furcation involvement Furcation classifications
* Class 0 * Class 1 * Class 2 * Class 3
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# Treatment of furcation involvement Class 0 furcation
No furcation involvement
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# Treatment of furcation involvement Class 1 furcation
Horizontal direction, probe travels less than 3mm ## Footnote through one or two entrances
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# Treatment of furcation involvement Class 2 furcation
Horizontal direction, probe travels more than 3mm ## Footnote -Probe doesnt go through whole furcation -Only in one entrane
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# Treatment of furcation involvement Class 3 furcation
Horizontal, probe travels more than 3mm making through and through lesion ## Footnote two or more entrances
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# Treatment of furcation involvement Subclasses of furcation in class 3
1. Less than 3mm in vertical direction 2. 3-6mm in vertical direction 3. More than 6mm in vertical direction
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# Treatment of furcation involvementTreatment of furcation involvement Probe used for assessment of furcations
* Nabers probe * 1 for upper * 2 for upper and lower
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# Treatment of furcation involvement Treatment of F1
* Scaling and root planing=> * Furcation plasty
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# Treatment of furcation involvement Treatment of F2
* Furcation plasty * Tunnel preparation * Root resection * Tooth extraction * GTR for mandibular molars
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# Treatment of furcation involvement Furcation plasty
* **Elimination of interradicular defect** * Tooth substance **removed**(odontoplasty) * Alveolar crest **remodelled** at level of furcation entrance(osteoplasty) * Mainly at **buccal** and **lingual** furcations
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# Treatment of furcation involvement Tunnel preparation
* Furcation area **widened**-removal of interradicular bone * Alveolar bone crest **recontoured** * **F2 and F3 in mandibular molars** ## Footnote F2 and F3 in mandibular molars(short root trunk, wide seperation angle, long divergence between mesial and distal roots)
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# Treatment of furcation involvement Root seperation
Sectioning of **root complex** and **maintenaince** of all roots
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# Treatment of furcation involvement Root resection
**Sectioning** and **removal** of **one** or **two** roots of multi-rooted tooth
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# Endodontic periodontal lesions Endo-perio lesion
* Conditions where **pulp** and **periodontium** diseased **simultaneously** * Disease may arise from **one** or **both** sites
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# Endodontic periodontal lesions Treatment of endo-periodontal lesions
* Endodontic treatment * Periodontal therapy following endodontic treatment if necessary
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# Endodontic periodontal lesions Diagnostic features of perio-endo lesion
* Occurence of sudden pain * Marginal inflammation * Increased probing depth * Suppuration * Increased mobility * Loss of fibrous attachment
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# 43.Antibiotics in periodontal therapy What are the main antibiotic groups used in periodontology?
* Penicillins * Metronidazole-Ntroimidazoles * Tetracyclines * Macrolides * Cindamycin * Cephalosporins and local antibiotics.
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# 43.Antibiotics in periodontal therapy Which antibiotic group is most commonly used for general periodontal infections?
Penicillins especially amoxicillin.
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# 43.Antibiotics in periodontal therapy Which antibiotic group is most effective against anaerobic periodontal bacteria?
Metronidazole nitroimidazole group.
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# 43.Antibiotics in periodontal therapy Which antibiotic group concentrates well in gingival crevicular fluid?
Tetracyclines.
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# 43.Antibiotics in periodontal therapy What is the gold standard antibiotic combination for aggressive periodontitis?
Amoxicillin and metronidazole.
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# 43.Antibiotics in periodontal therapy What is the main concern when prescribing penicillins?
Allergic reactions.
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# 43.Antibiotics in periodontal therapy Which antibiotics belong to the tetracycline group?
Tetracycline, doxycycline and minocycline.
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# 43.Antibiotics in periodontal therapy Which antibiotics belong to the macrolide group?
Azithromycin, erythromycin and clarithromycin.
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# 43.Antibiotics in periodontal therapy When are macrolides used in periodontology?
In **penicillin** **allergic** patients and **aggressive** **periodontitis**.
261
# 43.Antibiotics in periodontal therapy When is clindamycin used in periodontal treatment?
For **severe** **infections** or **penicillin** **allergic** patients.
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# 43.Antibiotics in periodontal therapy Give an example of a cephalosporin used in dentistry?
Cephalexin.
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# 43.Antibiotics in periodontal therapy Give examples of local periodontal antibiotics?
Minocycline, doxycycline gel and chlorhexidine chips.
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# 44. Regenerative therapy.Guided tissue regeneration Guided Tissue Regeneration (GTR)
* **Regenerating** periodontal structures **lost** due to periodontal disease=> * **Barrier** **membranes** direct growth of **new** bone and periodontal ligament=> * On **root** **surface** previously exposed due to disease
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# 44. Regenerative therapy.Guided tissue regeneration Restitutio ad integrum" in the context of GTR
* **Complete** **restoration** of periodontal tissue to **original state=>** * **Formation** of alveolar bone, functionally aligned periodontal ligament, and new cementum
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# 44. Regenerative therapy.Guided tissue regeneration Materials used for barrier membranes in GTR
* **Non-absorbable materials**=> * Polytetrafluoroethylene (PTFE) * Expanded polytetrafluoroethylene (e-PTFE)=> * Often reinforced w/ titanium * Perforated titanium foil * **Bioabsorbable materials**=> * **Natural** materials=>Collagen membranes from porcine or bovine origin * **Synthetic** materials=> Polylactic acid, copolymers of polylactic acid, polyglycolic acid, PEG
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# 44. Regenerative therapy.Guided tissue regeneration Synthetic bioabsorbable membranes
* **Polylactate** acid and its copolymers * **Polyglycolic** acid and its copolymers * **Polyglactin** (Vicryl) * **PEG**
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# 44. Regenerative therapy.Guided tissue regeneration Natural bioabsorbable membranes
* **Porcine** origin=>Jason membrane and Collprotect membrane * **Bovine** origin=>Geistlich Bio-Gide®
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# 44. Regenerative therapy.Guided tissue regeneration Types of bone replacement grafts used in GTR
* **Autologous grafts** (autografts)=>patient’s own body * **Isografts**=>genetically identical species * **Allografts**=>different individuals of same species * **Xenografts**=>different species * **Synthetic grafts** (alloplasts)=> synthetic materials * **Composite grafts**=>combination of different graft materials
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# 44. Regenerative therapy.Guided tissue regeneration Autografts
* **Same** individual * Osteogenic (4 weeks) osteoconductive and osteoinducive (2-4weeks) potential * **Extraoral**=> * Iliac crest, tibia, fibula, ribs * **Intraoral**=> * Chin, exostoses, torus * Ramus, tuberosity
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# 44. Regenerative therapy.Guided tissue regeneration Isografts
* Genetically identical species
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# 44. Regenerative therapy.Guided tissue regeneration Allografts
* **Same** species * **Osteoinductive** potential * **DFDBA**=>deminineralised freeze dried bone allograft * **FDBA**=>freeze dried bone allograft * **Frozen**
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# 44. Regenerative therapy.Guided tissue regeneration Alloplasts
* **Synthetic** * **Osteoconductive** * Hydroxyapatite * Calcium phosphate cements(CPC) * Beta-tricalcium phosphate(TCP) * Biphasic alloplastic materials * Bioactive glasses * Synthetic polymers
274
# 44. Regenerative therapy.Guided tissue regeneration Xenografts
* **Different** species * **Osteoconductive** and **osteoinducive** potential * Bovine derived * Porcine derived * Coralline calcium carbonate
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# 44. Regenerative therapy.Guided tissue regeneration Potential characteristics of bone replacement grafts
* **Osteogenesis**=>new bone by cells in graft * **Osteoinduction**=>new bone through differentiation of mesenchymal cells into osteoprogenitor cells * **Osteoconduction**=>new bone by providing a scaffold for bone growth