a. High RI is specific for rejection
b. Reverse end diastolic flow suggestive of renal vein thrombosis
c. Lymphocele collects radiotracers
d. Lymphocele occcus in the first two days post transplant.
e. PSV 1m/s is consistent with renal artery stenosis
b. Reverse end diastolic flow suggestive of renal vein thrombosis
*IVM: sneaky units for e. We normally think about PSV in cm/s.
PSV equal to or greater than 180cm/sec is suggestive renal artery stenosis.
c. Pneumocystis
a. Call the referer
a. Ganglioglioma 35% calcify, commonly cause seizures
*LW:
Sub acute thyroiditis, likely referring to Sub acute De Quervain granulomatous thyroiditis, which shows low uptake thyroid scans.
Functioning thryoid adenoma are hot on uptake scan with colder background parenchyma.
Previously:
a. Low up take in initial phases of subacute thyroiditis
Subacute thyroiditis- measle mumps coxackie- hypothyroidism- self limiting- U/S: hypo echoic, hypo vascular, small or large thyroid- Tc 99m : decreased uptake
but you can also have low background uptake in toxic adenoma?? ummm
d. Benign and malignant phaeochromocytoma chang et al cancer imaging 2016.
**SCS: this is recommended to assess for synchronous/ Metastatic disease best evaluated on Ga 68 DOTATATE scans (high level of somatostatin receptors), F-18 FDG may also be used.
Definition of malignant Phaeos is metastases. Evidence: [Robbies: “both capsular and vascular invasion, as well as cellular pleiomorphism may be encountered in some benign lesions. Therefore definite diagnosis of malignancy is based on the presence of metastases” ]
Rule of 10% tumour. thus 10%
Malignant.
Random trivia from Robbies: “one traditional 10% rule that has since been modified pertains to familiar cases… as many at 25% harbour a germ line mutation”
Hibernoma: Rare benign fatty tumour. Arise from vestigial Brown Fat. FDG PET avid, thus can’t distinguish from malignant lesion.
a. Atropine
also put their feet up (probably vas-vagal)
StatDx:
Hypotension with bradycardia (vasovagal): If unresponsive to fluids and supplemental oxygen: 0.6-1.0 mg atropine IV
a. Unruptured ectopic
8-10 weeks post LMP so assume pregnant
*AJL - I favour normal ovarian stimulation.
If it was hyperstimulation then they would need to say enlarged, free fluid etc. TOA doesn’t have this appearance (simple cysts). PCOS is bilateral AND the question say undergoing IVF rather than pre-IVF. It’s a bit of a weird question though, especially with only one ovary doing anything…
*LW: unsure of this….
If assume undergoing IVF, means recieving IVF ovulatory drugs, this would be a bilateral appearance, which doesn’t make sense if left ovary normal.
Ovarian hyperstimulation and PCOS are also bilateral processes.
Options are thus;
- Normal ovarian stimulation, in absence of full blown IVF drugs (unlikely the other ovary didn’t respond at all to drugs). Maybe in pre IVF work up this may be a possibility (i’m now stretching the imagination of the question)
- Tubo ovarian abscess: although it appears as a mass, multiple anechoic cysts is not classic…..
Previous answer:
d. Normal ovarian stimulation.
a. Chondrosarcoma
a. Liposarcoma
*ESG weakly favour ruptured XGP due to fever
MM - Agree with Ed
c. Too many neutrons beta minus
b. Too many protons beta plus note:
Alpha decay
- the process in which an alpha particle (containing two neutrons and two protons) is ejected from the nucleus.
- An alpha particle is identical to the nucleus of a helium atom.
RD226 -> Rn 222 + helium
Beta decay
- type of radio-active decay
- depends on how many protons and neutrons
If too many neutrons (beta minus decay)
- neutron -> protons + B - (electron)
If too many protons (beta positive decay)- protons -> neutrons + B+ (positron)
a. MDT discussion then rebiopsy probably
*AJL - agree with above. Though it’s tricky as the classic line is ‘needs repeat biopsy.’
In reality these go for discussion in MDT (or results are given in MDT) for radiology-pathology correlation and then go for surgical excision. I can’t find anything which specifically says rebiopsy before MDT discussion. (Let me know if you have found something else or think something else.)
Paper from 2016 lays it out: For Radiologically suspicious but pathologically benign lesions it says ‘The findings are discussed with referring physician and pathologist, a repeat biopsy in form of open surgical biopsy should be done.’
(https://www.alliedacademies.org/articles/concordant-versus-discordant-ultrasound-guided-breast-biopsy-results-how-they-effect-patient-management.pdf)
c. Neutrophilia
Around 25% of RCC patients will develop a paraneoplastic syndrome
19-21:- hypercalcemia (20%)
- hypertension (20%)
- polycythemia: from erythropoietin secretion (~5%)
- Stauffer syndrome: hepatic dysfunction not related to metastases
- feminisation
- limbic encephalitis
c. Desmopressin toxicity
Desmoresspin = ADH - used in treating DI and nocturnal diuresis
a. Uncinate herniation can result in pontine haemorrhage
b. Uncinate haemorrhage can result in 4th CN compression
c. Something about cingulate gyrus in parasaggital herniation
a. Uncinate herniation can result in pontine haemorrhage
d. Artchitectural distortion
b. Dense linear calcification
d. SAR I think
Specific absorption rate
Increased tissue heating secondary to multiple 180°-pulses may limit FSE use in infants and small children.
note improve MRI SNR by
note improve SNR by
a. Defect is small 2 – 4cm
b. Sequestration
e. Cryptococcus
MM - statdx - 4 imaging patterns: Gelatinous pseudocysts, leptomeningeal enhancement, miliary enhancing parenchymal/leptomeningeal nodules, cryptococcoma