SDEC—Hypertensive Urgency (no acute end-organ damage), SDEC Inclusion
Severely elevated BP (e.g., ≥180/110–120) without acute end‑organ damage, asymptomatic or mild headache/anxiety
SDEC—Hypertensive Urgency (no acute end-organ damage), Red Flags → Admit/Escalate
Hypertensive emergency evidence: neuro deficit, chest pain/ACS, pulmonary oedema, aortic dissection symptoms, encephalopathy, AKI/oliguria, eclampsia, papilloedema, refractory BP
SDEC—Hypertensive Urgency (no acute end-organ damage), Key History
Duration of hypertension, adherence
SDEC—Hypertensive Urgency (no acute end-organ damage), Exam – Positive Findings
Very high BP on repeated measures (proper cuff/position), fundus exam (AV nicking vs papilledema)
SDEC—Hypertensive Urgency (no acute end-organ damage), ECG / Imaging
ECG for LVH/ischemia, CXR if pulmonary oedema suspected
SDEC—Hypertensive Urgency (no acute end-organ damage), Biomarkers/Labs
U&E (renal function, K+), urinalysis (protein/haematuria), FBC, consider TFTs/catecholamines only if indicated.”
SDEC—Hypertensive Urgency (no acute end-organ damage), First-Hour Actions
Confirm BP correctly, rest quietly 20–30 min and recheck
SDEC—Hypertensive Urgency (no acute end-organ damage), Initial Management
Gradual reduction over days—not hours. Start/uptitrate oral agents (e.g., amlodipine, ACEi/ARB, thiazide). Avoid rapid large drops (>25% MAP in 24–48 h). Counsel adherence/salt/alcohol
SDEC—Hypertensive Urgency (no acute end-organ damage), Discharge Criteria
Asymptomatic, BP improving/stable with plan
SDEC—Hypertensive Urgency (no acute end-organ damage), Safety-Net & Follow-Up
Return for chest pain, neuro symptoms, dyspnoea, visual changes, confusion, vomiting. GP/HTN clinic review in ≤1 week with home BP log
SDEC—Hypertensive Urgency (no acute end-organ damage), Clinical Pearls
25–30% of ‘severe BP’ normalises after 30 minutes’ rest. Treat the patient, not the number—avoid IV drugs in urgency
SDEC—Hypertensive Urgency (no acute end-organ damage), Decision Rules
Non-specific, disposition based on the absence of end‑organ damage and follow‑up reliability
SDEC—Musculoskeletal Chest Wall Pain / Costochondritis,SDEC Inclusion
Pleuritic or focal chest wall pain reproducible on palpation/movement, normal ECG/troponin or alternative cause excluded
SDEC—Musculoskeletal Chest Wall Pain / Costochondritis, Red Flags → Admit/Escalate
Features suggestive of ACS/PE/aortic dissection/pneumothorax, fever with toxicity
SDEC—Musculoskeletal Chest Wall Pain / Costochondritis, Key History,
Localised sharp pain worse with movement, deep breath, coughing, recent strain/infection/cough
SDEC—Musculoskeletal Chest Wall Pain / Costochondritis, Exam – Positive Findings
Point tenderness over costochondral joints or chest wall musculature, pain reproduced by movement/press
SDEC—Musculoskeletal Chest Wall Pain / Costochondritis, ECG / Imaging
ECG normal, CXR if pneumonia/pneumothorax suspected
SDEC—Musculoskeletal Chest Wall Pain / Costochondritis,Biomarkers/Labs,
None routinely, troponin only if cardiac features.
SDEC—Musculoskeletal Chest Wall Pain / Costochondritis, First-Hour Actions
Rule out life‑threatening causes (brief ACS/PE/dissent screen as appropriate), give NSAID/analgesia
SDEC—Musculoskeletal Chest Wall Pain / Costochondritis, Initial Management
NSAIDs or paracetamol, topical NSAIDs/heat
SDEC—Musculoskeletal Chest Wall Pain / Costochondritis, Discharge Criteria
Pain controlled, serious causes excluded
SDEC—Musculoskeletal Chest Wall Pain / Costochondritis, Safety-Net & Follow-Up
Return if pain becomes exertional/central/pressure‑like, dyspnoea, syncope, fever. GP review if persistent >2–4 weeks or recurrent.
SDEC—Musculoskeletal Chest Wall Pain / Costochondritis, Clinical Pearls
Reproducible chest wall tenderness reduces but does not eliminate the chance of ACS—ensure appropriate initial screen.
SDEC—Musculoskeletal Chest Wall Pain / Costochondritis, Decision Rules
None required. Clinical diagnosis after exclusion of serious pathology.