How does muschle change extensibility?
What other things impact soft tissue mobility?
Nerves, tendons, fascia, blood vessels, skin, lymphatics
Think of how deep surgeons cut
Properties of Musculotendinous units
PEC=Parallel elastic component
SEC=Series elastic component
CC=Contractile component
NEURAL COMPONENTS:
- Resistance of stretch may come from the CC (gamma motor neurons) or from PEC/SEC
- Muscle Guarding/Tone
What tissues are we targeting?
What conditions are associated with soft tissue mobility deficits?
Long Term Immobilization
Acute onset of pain with associated muscle guarding
Increase blood flow, ROM, tissue mobility
Sensitivity of Myofascial Trigger Points associated with persistent (chronic) pain
Trigger Point Identification
* Specific area of muscle that is sensitive
* Palpation elicits response:
– May elicit twitch response
– Localized tenderness
– Typical referral pattern
* Active vs. Latent (feel it but not sending pain along pathway)
* More sensitive in tonic mm (more likely to have referral)
* Associated with “joint dysfunction”
* Goal: decrease sensitivity of trigger point (Varying levels of vigor for STM and stretching); Want to make it more latent than active
What muscles are more prone to “irritable” trigger points?
Tonic Muscles!
Upper Crossed Syndrome
* Upper trapezius
* Levator scapulae
* Suboccipitals
* SCM
* Scalenes
* Latissimus dorsi
* Pec major / minor
Lower Crosse Syndrome
* Iliopsoas and rectus femoris
* Adductors
* Latissimus dorsi
* Erector spinae
* Iliopsoas
* Hamstrings
* IT band
* Hip adductors
* Gastrocnemius
Address postural Faults
Improve flexibility (decrease tone) of “tight” (“tonic”) muscles to enhance posture and enhance agonist activation (associated with trigger points – Varying levels of vigor)
High Anxiety and Stress
^All Short Term Examples
Sunlight is long term.
Soft tissue swelling with impaired lymphatic activity / venous return
Tissue Adaptation
Not Enough
- Stagnation, Dysfunction, Pain
Just Right
- Growth and Healing
Too Much
- Irritation, Oversuse Injury, Pain
Where does STM and Stretching “fit” in PT Interventions?
IF YOU DON’T ADDRESS THE ISSUE AFTER FIXING IT WILL RESULT IN THEM REGRESSING BACK; Need exercise
Examination findings associated soft tissue mobility deficits
Subjective: Tightness and/or referres pain in similar pattern with muscle, Pain with static posture
AROM: Loss of motion with feeling of tightness on opposite side of direction of motion (antagonist mm); Ex: Quads with knee flexed.
PROM/Overpressure: Increase muscle guarding/Elastic end feel. Reproduce pain (“their pain”)
Palpation:
– Tenderness areas of muscle with or without pain referral that REPRODUCES patient’s pain OR increase muscle tone to palpation
– Decrease soft tissue/scar mobility following trauma/surgery (and appropriate healing has occurred); Ex: Try to move it and it stays tethered and adherred in place
Motor: Poor activation of agonist mm; Ex: Pec is tight, test rhomboids and middle/lower trap.
Irritability Levels - Soft Tissue Levels
What are some intervention options for Soft Tissue Mobility Deficits?
Soft Tissues Mobilization Mechanisms
Biomechanical/structural effects
Physiological Effects
Neurological Effects
Peripheral mechanisms
* Decrease pain through gate control system
* Stimulation of large diam. (A-beta) afferent fibers via mechanoreceptors
* Stimulation of substantia gelatinosa in SC inhibits smaller (A-delta, C) afferent fibers, blocking part of pain message
* Decrease tone in the muscle
Central pain processing effect
* Affects supraspinal centers of brain diminishing pain perception
Psychological Effects
Lymphedema
Patechia
Blood vessels bursting, purple or little dots, try to blanch will not change