Strabismus Flashcards

(39 cards)

1
Q

Describe fully accommodative esotropia and how it’s managed

A

a strabismus secondary to refractive error onset between 2-5yrs (+2.00 to +7.00)

they manage visually sRx or they’d be bilateral refractive amblyopes

Give full Rx full-time wear and check post-cyclo, review in 1mth ensure no amblyopia/suppression

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2
Q

Describe partially accommodative esotropia

A

high AC:A with manifest deviation reduced by min.10D with specs

full rx doesn’t correct small strabismus; try exercises

if large consider surgery (be aware of divergence)

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3
Q

Describe convergence excess esotropia

A

ESOT@Near with corrected Rx due to >8:1 AC/A onset 2-5yrs
BSV for distance fixation
(+1.50 to +5.00)
noticeable when tired/ill

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4
Q

Describe infantile esotropia with its risk factors and management

A

onset <6mths with large 30∆ deviation
age-expected refraction, often crossed fixation

male, FOH, prematurity, systemic disorders/medication

refer for surgery, latent/manifest nystagmus or dissociated vertical deviation can persist post-surgery

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5
Q

Describe acquired non-accommodative esotropia

A

onset 6-24mths with 30-70∆ deviation, normal BV development until strabismus, normal AC/A

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6
Q

Explain intermittent exotropias at distance and near

A

Distance: likely asymptomatic, eye drift noticed by others (exercises for fusional reserves, over-minus)

Near: ≈myopic teenage with diplopia when reading (exercises/over-minus for small angles, surgery for large angles)

EXOT@D then EXOP@N vice versa

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7
Q

Describe microtropia, which is more common and its management

A

micro-strabismus less than
6∆/10∆ with mild amblyopia + anisometropia
Reduced stereo on Frisby with EF + HARC
Secodnary to larger treated deviation (surgery/exercises/specs)
Primary if no previous history (<3yrs old)

eso more common than exo
treat anisometropia/give exercises if <4/5yrs consider patching

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8
Q
A
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9
Q

Describe the significance of NMD on CT with nd without identity

A

With: HARC + absolute EF, Anomaly angle = Eccentricity angle (NMD)

Without: central/non-absolute EF, ARC can be ARC/NRC with central suppression/peripheral fusion or manifest deviation seen on CT

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10
Q

Define residual and consecutive tropias

A

Residual: from surgical under correction
Consecutive: from surgical over correction

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11
Q

Which 3 things will a strabismic px experience related to BV?

A

Diplopia/Visual confusion
Suppression
ARC

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12
Q

Describe the initial presentation of strabismus

A

if eyes are misaligned the sensory fusion can’t cope with the motor misalignment so there’s no longer a single percept
Perceived position from retinal location remains due to neural ‘wiring’ so percept is different from ‘real’ world

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13
Q

Explain how some diplopia is normal in terms of crossed + uncrossed

A

diplopic images on widespread NCP (outside Panum’s FA)
physiological diplopia

Crossed – focus @ distance near object will
have image on temporal retina each eye

Uncrossed – focus @ near, distance object
will have image on nasal retina of each eye

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14
Q

How is diplopia and visual confusion removed in young/older individuals?

A

Young: alter signalling/organisation of visual system
Binocular HARC or monocular suppression of strabismic eye’s binocular field

Older: occlude one eye, compensate for AHP, try to ignore image/confusion

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15
Q

Describe binocular suppression

A

a non-selective inhibitory process removing all stimuli n suppressed region from consciousness

occurs in plastic systems in response to strabismus/anisometropia/phorias

affected by fatigue/squint angles/fixation and can cause diplopia

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16
Q

When can normal suppression occur?

A

contradictory (non-fusable) supply each eye causing alternate suppression (binocular retinal rivalry)

physiological suppression against physiological diplopia

monovision CL wearers ignore non-required image

17
Q

How does suppression relate to the fovea?

A

persists in binocular conditions, suppression areas localised to poor eye fovea + non-foveal area creating diplopic area

18
Q

How is a Sbisa bar used to test for suppression?

A

filter bar of increasing density of sequential filters
medium density filter on good eye fixating a white spotlight
level of suppression measured with increasing filter density until 2 dots/white dot seen

19
Q

Describe the classification of HARC and UnHARC

A

HARC: sensory realignment counteracts motor misalignment exactly

UnHARC: sensory realignment can’t fully counteract motor misalignment exactly - usually an artefact/px always diplopic

20
Q

Describe the significance of HARC as a binocular adaptation

What are its advantages?

A

avoids diplopia/confusion, disappears on occluding 1 eye
natural viewing conditions with fixing eye’s fovea corresponding to retinal area at angle of squint in non-fixing eye

used alternatively to suppression

retains BSV while squinting and can achieve gross stereo depending on angle size/VA of non-fixing eye

21
Q

How is HARC investigated and differentiated from suppression?

A

strabismic px with stereo must have HARC, detect via Bagolini for minimal visual disturbance

using monocular markers in a binocular environment (Mallett but near markers too small/require polarising filter so unnatural)

22
Q

How can we grade depth of ARC?

A

Good ARC condition cause deep ARC otherwise shallow
ARC can break down into induced diplopia

assessed via ND filter bar in front of fixing eye, increase by 0.3 log steps, when streak/marker disappears suppression occurs

higher ND value ~ stable/deeper ARC

23
Q

Explain why HARC doesn’t completely compromise binocularity

A

in a plastic system the visual system suppress the squinting eye fovea and appropriate non-foveal points to avoid diplopia/confusion

left with no (para)central binocularity but under favourable conditions a sufficient system further adapts to achieve a degree of binocularity via cortex reorganisation in response to motor misalignments

this sensory realignment is an ARC

24
Q

Why don’t all strabismic have HARC?

A

ARC key requirement involve early onset squint (plastic system) and small, constant strabismus angle

25
Explain penalisation therapy vis atropine
1% atropine in good eye to blue near vision (+DV if no rx) encourages use of amblyopic eye for near
26
Give 4 diagnostic uses of optical management for strabismus
Modification + justification of rx Convergence excess ESOT Divergence excess EXOT@D
27
Give 4 therapeutic uses for strabismus management
Decompensated phoria Fully accommodative ESOT Convergence excess ESOT Divergence excess EXOT@D
28
Explain how to /justify assess an Rx for treatment
assess effects ESOT if <+2.00, review in 2wks assess effect of proposed rx modification on strabismus size
29
Explain decompensated phoria with signs and causes
asthenopia/blurred VA in effort to control phoria failure to maintain BV so diplopia/poor stereo can be due to poorly fitted specs inducing prism or incorrect Rx
30
Explain the cause and management for fully accommodative ESOT
due to hypermetropia cyclo refraction; give full + check CT on specs collection, review in 4-6 weeks to check VA/stereo improvement
31
Explain the management for convergence excess ESOT
Exercises + Bifocals (Exec) for 5yr+ as temporary use until successful surgery/exercises (prolonged use causes abnormal accom.) increases strength to until within ortho; aim for lowest strength, can trial Fresnel prisms CT/Bagolini/Stereo
32
Explain the management for distance EXOT
<-3.00DS lenses, increase strength till ortho in distance (aim for lowest strength) ~ <20∆ exo (high AC/A) less tolerable for older kids doing more near work
33
Explain the 3 stages of exercise therapy for strabismus
1. Overcoming suppression (if unaware of manifest diplopia, red filter tracing) & Diplopia awareness (R/G glasses) 2. Extension of fusion range (+exo/-eso) via 2 pens or dot card 3. Improved relative convergence reserves
34
Describe the use and procedure for Dot cards, Brock strings, Jump convergence
Dot cards: +ve fusion range for awareness of physio diplopia (effective, portable, cheap but convergence must be <30cm) Brock strings: physio diplopia & BSV at >33cm for severe CI Jump: +ve fusion reserve for later treatment regime, uses accom. target with dot card
35
Explain how to perform Prism Fusion Range tests
‘measure how well eyes converge/diverge keeping a single image’ lowest line read, move bar towards until blurred/double (blur/break) and then back until regained bifoveal fixation (recovery) perform for D & N
36
What 3 clinically significant points do you record for PFR?
Blur: prism when px can maintain/overcome via effort/limit of convergence Break: lowest prism to break fusion limit of px’s fusion range/loss of bifoveal fixation Recovery: prism power where eyes fuse again, regain bifoveal fixation
37
Explain the extension of relative convergence range and give 2 ways of exercising this
manipulate convergence to maintain BSV + stable accom. with a CLEARLY SEEN target +ve when convergence in excess of accom. (exo) -ve when convergence is relaxed compared to accom. (eso) stereograms (proximal pen clearer = +ve & distal pen =-ve)
38
Describe the procedure for MEM retinoscopy
dim lights with px distance rx looking at detailed target on retinoscope 40cm away, neutralise fast with movement with + lenses if >0.75/1.00 indicative of problems, if -ve then px overaccommodating
39
Explain prism adaptation in relieving symptoms and other uses
normal BV px adapt to prism but px with minor phoria and severe symptoms generally don’t adapt so prism prescribed to relieve symptoms of phoria/CI if exercises fail; image moved into suppressed retina always give minimum prism (cosmesis/cost) on preferred (non-dominant eye), split between eyes if needed prisms can aid nystagmus (congenital) via binocular BO prisms, (eyes deviate towards null region) and can deviate eye away from any field defects