Mechanical Bowel Preparation - rationale
To empty the intestines prior to the procedure, to provide a clear view of the bowel
Mechanical Bowel Preparation
Mechanical Bowel Preparation - Nursing considerations
Mechanical Bowel Preparation - After the procedure
Surgical Fasting Times
Bowel Cancer (Ca) - Prevalence
:
Bowel Cancer (Ca) - Risk factors
Age Bowel diseases Previous history Lifestyle (overweight, red meat, alcohol, smoking) Family history Rare genetic disorders polyps
Bowel Cancer (Ca) - Symptoms
Living with bowel cancer
Surgical complications -colectomy
Congestive Cardiac Failure Patho
occurs when the heart is unable to pump oxygenated, nutrient rich blood out at a rate that meets the metabolic demands of the body, causing a back-up of blood in the venous circuit and leading to oedema.
Results from any structural or functional cardiac disorder that impairs the ability of the ventricle to fill or eject blood.Diagnosed when LVEF < 40%
Congestive Cardiac Failure Patho - Causes
Determinants of Cardiac function
preload
afterload
contractility
heart rate
preload
represent the stretch on the ventricles as a consequence of ventricular filling. (venous return relates directly to end diastolic volume) i.e intravascular blood volume
Afterload
resistance downstream to the left ventricle which it has to overcome in order to eject blood from the heart. i.earterial vasoconstriction increases resistance
Contractility
the force of contraction to ensure the adequate stroke volume is ejected. The degree of myocardial fibre shortening
Captopril 50mg
Class: Antihypertensive, ACE inhibitor
Indications: hypertension, Heart Failure (+diuretic)
Mechanism of Action: highlyspecific competitive inhibitor of angiotensin I converting enzyme, theenzyme responsible for the conversion of angiotensin I to angiotensin II.:
Nursing assessment(s) prior to the administration: Check BP
Nursing assessment(s) after the administration: Monitor Serum potassium levels
Potassium 1200mg
Indications: Treatment of all types o fpotassium deficiencies, particularly hypochloraemic or hypokalaemic alkalosis, associated with prolonged or intensive diuretic therapy, e.g. in hypertension,cardiac failure
Mechanism of Action: sustained release potassium supplement
Interactions: Span-K should be used with caution, if at all, in patients receiving drugs that increase serum potassium concentrations. These include potassium sparing diuretics, angiotensin convertingenzyme (ACE) inhibitors
Nursing assessment(s) prior to the administration: Know patient serum potassium with hold if > 5.5mmol/L
Nursing assessment(s) after the administration: Monitor Serum potassium levels
Surgical risks for CCF
Heart failure is a major risk factor.
HF patients have substantially higher risks of postoperative mortality than those with coronary artery disease undergoing the same procedures
Patients with heart failure (HF) are at risk for hypotension, hypertension, and arrhythmias during surgery.
Due, in part, to the stress response induced by surgery, with release of catecholamines, steroids, and inflammatory mediators, which increase metabolic demand.
ACE inhibitor and surgery
Generally oral antihypertensive medications should be continued up to the time of surgery,with few exceptions. taken with small sips of water on the morning of surgery.
However, we typically withhold angiotensin-converting enzyme (ACE) inhibitors and angiotensin II receptor blockers (ARBs) for a period of 24 hours prior to surgery.
Some anesthesiologists may prefer to withhold these medications on the morning of surgery based on concerns about possible hypotension particularly if significant perioperative fluid shifts are anticipated.
We suggest individualizing the decision to continue or discontinue ACE inhibitors based on the indications for the drug, the patient’s blood pressure, and the type of surgery and anesthesia planned.
T2DM Pathophysiology
Chronic Progressive condition
Main problem is the body becomes resistant to the normal effects of insulin and/or the body loses the capacity to produce enough insulin in the pancreas to meet demand.
There is a decreased intracellular reaction to insulin causing a decreased uptake of glucose by the tissue
Causing an unchecked regulation of glucose production/release by the liver (gluconesis & Gluconeogenesis)
To overcome this there needs to be an increase in the amount of insulin secreted.
However if the beta cells cannot keep up with the increased demand for insulin, glucose level rises above normal level and T2DM develops.
T2DM Risk factors - modifiable
Weight Sedentary lifestyle diet HTN Apple shaped body
T2DM Risk factors - Non –modifiable
T2DM Symptoms / Clinical manifestations
Polyphagia
Polyuria
Polydipsia