Nursing interventions for absence of voiding for significant period of time postoperatively
Interventions (in proper order) if postoperative bleeding is assessed at the incision site
The blockage of pulmonary arteries by thrombus, fat or air emboli, or neoplastic tissue
PULMONARY EMBOLISM
Definition
PULMONARY EMBOLISM:
CLINICAL MANIFESTATIONS
PULMONARY EMBOLISM:
NURSING INTERVENTIONS - ACUTE CARE
A disorder involving a thrombus in a deep vein, most commonly the iliac and femoral veins.
DVT
(called Virchow’s triad)
(1) venous stasis
(2) damage to the endothelium (inner lining of the vein)
(3) hypercoagulability of the blood.
The client at risk for the development of venous thrombosis usually has predisposing conditions to these three disorders
DVT ETIOLOGY
May have no symptoms or have unilateral leg edema, extremity pain, warm skin, and erythema.
If the calf is involved, tenderness may be present on
palpation.
A positive Homans’ sign (pain on forced dorsiflexion
of the foot when the leg is raised) is a classic but very unreliable sign
If the superior vena cava is involved, there may be symptoms in the upper extremities, the neck, the back, and the face.
DVT CLINICAL MANIFESTATIONS
DVT NURSING INTERVENTIONS
Decrease in oxygen resulting in failure to nourish tissues at the capillary level
IMPAIRED TISSUE PERFUSION
WHAT DOES IMPAIRED TISSUE PERFUSION CAUSE?
SHOCK
A syndrome characterized by decreased tissue perfusion and impaired cellular metabolism. This results in an imbalance between the supply of and the demand for oxygen and nutrients. The exchange of oxygen and nutrients at the cellular level is essential to life. When a cell experiences a state of hypoperfusion, the demand for oxygen and nutrients exceeds the supply.
SHOCK
WHICH STAGE OF SHOCK?
aerobic switches to anaerobic, build up of lactic acid, metabolic acidoseous
STAGES OF SHOCK - Intital stage
WHICH STAGE OF SHOCK ?
neurologic changes, maintaining homeostasis, barrow receptors, vessels general vasoconstriction, trying to be maintained, trying to maintain the function of vital organs, kidneys renetangio rensen cycle, decreases urinary output, increases venus return to the heart, attempting to increase blood pressure, GI tract compromised, impaired motility, skin is cool and clamy.
STAGE OF SHOCK - COMPENSATORY STAGE
WHICH STAGE OF SHOCK?
compensating measures in general are failing, proteins leaking through to the interstital space, causes interstitial edema, anascara, blood flow to pulmunary capillaries, leaving vascular space, pulmonary edema, looking for crackles, fluid moves into aveoli, respiratory increase, cardiac input falls further, circulation decreases, cap refil low, temp cold, pulses weak, b/p low, low oxygen, dysrythmea, excemia, MI, complete deteriation of the cardiac system,GI tract mucousa barrier is decreasing, protects lining of bowels, causes major bleeding, increases with shock, ability to absorb nutrietns decreases, immune function compromised
STAGES OF SHOCK - Progressive Stage
• vomiting
• hemohharge
• diahhrea
• diabetes
• excessive urine result of high blood sugar acts like salt
* External loss of whole blood (e.g., hemorrhage from trauma, surgery,
* GI bleeding)
* Loss of other body fluids (e.g., vomiting, diarrhea, excessive diuresis, diabetes insipidus, diabetes mellitus - high blood sugar causes oligoria)
Hypovolemic shock symptoms
Absolute Hypovolemia
* Pooling of blood or fluids (e.g., bowel obstruction)
* Fluid shifts (e.g., burn injuries, ascites)
* Internal bleeding (e.g., fracture of long bones, ruptured spleen, hemothorax, severe pancreatitis)
* Massive vasodilation (e.g., sepsis)
Hypovolemic shock symptoms
Relative Hypovolemia (3rd spacing)
Common laboratory readings during progressive stage OF SHOCK
• Maintain patent airway
• Optimize oxygenation
with supplemental O2
• Intubation, mechanical
ventilation, if necessary
• Aggressive fluid
resuscitation with
colloids
• Antihistamines (e.g.,
diphenhydramine)
• Epinephrine (subQ, IV,
nebulized)
• Bronchodilators:
nebulized (e.g.,
albuterol)
• Corticosteroids (if
hypotension persists)
• Identify and remove
offending cause
• Prevention via avoidance
of known allergens
• Premedication with
history of prior sensitivity
(e.g., contrast media)
Interventions for anaphylactic shock
Common fluids used in fluid resuscitation
COLLOIDS & ISOTONIC CRYSTALLOIDS
Septic shock manifestations
Goal of care for cardiogenic shock
For a client in cardiogenic shock, the overall goal is to restore blood flow to the myocardium by restoring
the balance between oxygen supply and demand
Collaborative care for clients with MODS focuses on 4 THINGS
Collaborative care for clients with MODS focuses on
(1) prevention and treatment of infection
(2) maintenance of tissue oxygenation
(3) nutritional and metabolic support
(4) appropriate support of individual failing organs.
Dosage adjustment based on assessment of the adequacy of analgesic effect versus the side effects produced
TITRATION