DLF lesion
Ipsi loss of P+T, total of 3 segment , affected level + 2 below
NP lesion
Ipsi loss of P+T, 2 segment down only
VWC lesion
Bilateral loss of P+T, 2 segment down only
LSTT lesion
Contra loss of P+T, 2 segment, 2 segments and all below
FC lesion
T6 and above
FG lesion
Below T6
Coricospinal Tract before cross lesion
Contra spasticity
Corticospinal tract after cross
Ipsi spasticity
Corticospinal tract V horn lesion
Ipsi flaccidity
Corticospinal tract LMN
Ipsi flaccidity
Lesion of reticular formation lesion
Cornea/death
Dorsal root
Sensory information
Ventral roots
Motor information
Lesion nucleus ambiguous
Dysphonia/dysphagia/
Lesion dorsal cochlear
Hearing loss
Lesion olivocerebellar fibers
Contra alaxia
UMNs
Start in higher centers of the CNS cerebral cortex and make up the Corticospinal tract (CST)
Corticospinal tract
Travels from the cerebral cortex to the spinal
Upper motor neurons
Send “command” to lower motor neurons (LMNs)
Hyperflexia
Body’s reflexes are more pronounced /exaggerated than normal, which can result in strong jerky movements
Lesion to UMNs
Spasticity paralysis or paresis, characterized by hyperreflexia and hypertonia
Hypertonia
Means that the muscles reflexes are more pronounced than normal, which can result in strong jerky movement
Lower mother neurons
Start in the spinal cord and brainstem and controlled by upper motor neurons
Lower motor neuron function
Directly synapse with muscles and command to contract via cranial and spinal nerves