Unit 2 Week 1 Flashcards

(35 cards)

1
Q

what type of adaptation should we consider hypomobility to be?

A

a physiological adaptation (the body is trying to get better at what it is doing)

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2
Q

what is mechanotransduction?

A

the process by which the body converts mechanical loading into cellular responses which promote structural change

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3
Q

what are the 3 ways collagen contributes to adaptations for hypomobility?

A

cross-linking
adhesions (change in lubrication, harder to move in its sheath - less movement)
disorganized orientation (layed down randomly where there is no movement due to immobilization)

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4
Q

what is the physiological adaptation of the muscle when it is immobilized?

A

there is a reduced number of sarcomeres in series when a muscle is immobilized, to adapt to the shortened length of the muscle

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5
Q

why would the body add or remove sarcomeres to a muscle?

A

it does this to adapt to a muscle’s length, the muscle wants to make sure that sarcomeres are sitting in their optimal positions

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6
Q

what is tone and where is it common?

A

tone is the passive state of muscle contraction or a muscles’ resistance to passive tension at rest (it is common close to an area that has recently been injured)

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7
Q

what is muscle hypomobility largely due to?

A

neural control (e.g. changes in muscle tone)

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8
Q

what is the theory of connective tissue mobility prescription?

A

it explains the connective tissue response to tension (viscoelastic) which is rate dependant and time dependant
- in the elastic deformation phase, there will be no lasting change in the tissues (will cause stress relaxation, creep, and temporary change in mobility)
- in the plastic deformation phase, you want to cause just enough microscopic failure to create permanent change, but not enough to create macroscopic change (will cause microscopic failure, macroscopic failure, and permanent change in mobility)

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9
Q

what is the practical application of the connective tissue mobility prescription theory?

A

think PUDDY
- you want to do a low load prolonged stretch with total end range time (TERT)
- we want to LIMIT force, but increase TIME to avoid damage

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10
Q

what is the best practice for connective tissue mobility?
low load prolonged stretch

A

low load prolonged stretch
greater than 3x per day
into stiffness, not pain
position of TENSION
5-20 mins per session
increase volume and intensity to progress (often seen with post-surgery ACLs and TKRs)

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11
Q

what is the best practice for connective tissue mobility?
mobility

A

greater than 3x per day
into stiffness not pain
slow PROM, AROM, AAROM into position of tension
5-10 reps for 10-30 secs for 1-3 sets
increase volume (TERT) to progress

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12
Q

what is the theory of muscle flexibility prescription?

A

explains the muscle’s response to tension (stretching) - explaining how there is an activation of the muscle spindle, leading to stimulation of myotactic/stretch reflex, stimulating the alpha neuron, and leading to a muscle contraction
- this theory explains in short how the feeling of muscle tension should fade as you hold the muscle in it’s extended contracted position

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13
Q

what is the sensory theory portion of the muscle flexibility prescription theory?

A

it states that there are no lasting changes seen in muscle length following static stretching and that temporary increases in muscle flexibility following static stretching may be due to an increased TOLERANCE of tension and pain

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14
Q

what is the golgi tendon reflex?

A

it explain the tendon’s response to tension, the golgi tendon organ will be activated when a muscle nearly is contracted - this will cause stimulation of the golgi tendon reflex which tells the muscle to relax and helps to reset the body

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15
Q

what is the neuromuscular relaxation theory?

A

it explains autogenic inhibition (aka PNF stretching) and how the golgi tendon is responsible for telling the muscle to relax - this a action helps facilitate an additional stretch

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16
Q

what are the 3 types of muscle stretching

A

static
dynamic
pre-contraction (proprioceptive neuromuscular facilitation PNF)

17
Q

what does dynamic stretching increase?

A

range of motion
performance (if immediately before activity)

18
Q

what is the impact of static stretching on ROM and performance?

A

increase ROM
decrease performance (if immediately before)
* this activity gets you ready to relax rather than perform

18
Q

what is the best practice for dynamic stretching?

A

3x per week
into tightness not pain
controlled concentric contraction of opposing muscle into position of tension of target muscle
volume is 1-3 sets of 5-10 reps

19
Q

what is the best practice for static stretching?

A

2-3 per week
into tightness
slowly and passively assume position of tension in target muscle
10-30 seconds or 30-69 for OA
1-3 reps or 60 seconds total

20
Q

what are the effects of eccentric training on mobility?

A

comparable to static stretching
increases fascicle length (more sarcomeres)

21
Q

does foam rolling improve mobility?

A

yes there is a temporary change in the tone of tissues, comparable to static stretching, best when followed by static stretching
due to sensory & neuromuscular mechanisms (not structural)

22
Q

can unilateral stretching improve mobility in contralateral non-stretched joints

A

yes, there are temporary sensory changes to the body

23
Q

what are the benefits of resisted exercise in rehabilitation?

A

increase strength, improve function, reduce pain

24
what is the repetition continuum theory?
proposed that resisted exercise that produces fatigue in specific repetition ranges develops specific characteristics: low rep ranges increase strength moderate rep ranges increase hypertrophy high rep ranges increase endurance
25
is hypertrophy dependant on load?
NO, low moderate and high rep ranges cause hypertrophy
26
what is muscle disuse muscle atrophy?
states that episodes of inactivity or muscle disuse following illness/injury leads to loss of muscle size (muscle atrophy) - disuse atrophy is greater in ages/unwell people - occurs initially rapidly and then decreases over time - differs between muscle groups
27
how can resisted exercise help will disuse muscle atrophy?
it can mitigate disuse atrophy, strength loss, and function when implemented before, during and following episodes of disuse (e.g. illness injury surgery)
28
what are the methods for prescribing resisted exercise intensity?
rep max (RM) RM range RPE repetitions in reserve (RIR)
29
what is repetition max range?
max amount of load an individual can lift within a range of reps using standardized technique
30
what is repetitions in reserve?
an estimation of the remaining repetitions an individual feels capable of prior to failure
31
what is "failure"?
refers to temporary inability to continue a resisted exercise set using standardized technique - it induces neuromuscular fatigue, muscle damage, and perceived discomfort which may influence performance, recovery, and adherence
32
what is periodization?
it is the planned manipulation of exercise prescription variables (e.g. intensity, volume) to maximize physiologic adaptations - periodization resisted programs have a greater effect on strength
33
what is true about strength loss after 50 years of age?
it is believed that you lose up to 5% of your strength per year once youre 50, this leads people to function at their strength limits (ex 1RM living)
34
how can OA minimize the loss of strength?
it is recommended that resisted exercise is initiated early and maintained