VASOMOTOR RHINITIS
It is nonallergic rhinitis but clinically simulating nasal allergy with symptoms of nasal obstruction, rhinorrhoea and sneez- ing.
The condition usually persists throughout
the year
the tests of nasal allergy
negative
PATHOGENESIS
Nasal mucosa has rich blood supply. Its vasculature is similar to the erectile tissue in having venous sinusoids or “lakes” which are surrounded by fibres of smooth muscle which act as sphincters and control the filling or emptying of these sinusoids. Sympathetic stimulation causes vasoconstriction and shrinkage of mucosa, while parasympathetic stimula- tion causes vasodilation and engorgement. Overactivity of parasympathetic system also causes excessive secretion from the nasal glands.
Autonomic ner vous system is under the control of
hypothal- amus and therefore emotions play a great role in vasomotor rhinitis. Autonomic system is unstable in cases of vasomotor rhinitis.
Nasal mucosa is also
hyper-reactive and responds to several nonspecific stimuli, e.g. change in temperature, humidity, blasts of air, small amounts of dust or smoke.
SYMPTOMS
Paroxysmal sneezing
Excessive rhinorrhoea
Nasal obstruction.
Postnasal drip.
Paroxysmal sneezing
Bouts of sneezing start just after getting out of the bed in the morning.
Excessive rhinorrhoea
accompanies sneezing or this may be the only predominant symptom. Pofuse and watery and may even wet several handkerchiefs. The nose may drip when the patient leans forward and this may need to be differentiated from CSF rhinorrhoea
Nasal obstruction
marked at night. It is the dependent side of nose which is often blocked when lying on one side
SIGNS
Nasal mucosa over the turbinates is generally congested and hypertrophic. In some, it may be normal.
COMPLICATIONS
nasal polypi, hypertro- phic rhinitis and sinusitis.
MEDICAL TREATMENT
SURGICAL TREATMENT