What is Vertigo?
What are some characteristics of peripheral vertigo?
What are some characteristics of central vertigo?
What is the etiology of Peripheral vertigo?
What is the etiology of Central Vertigo?
What is Benign paroxysmal positional vertigo (BPPV) comprised of?
Comprised of repeated episodes of vertigo that occur subsequent to changes in head position
Describe BBPV
What is the etiology of BPPV?
Usually otoconia (carnalith) that loosens and travels into the posterior semicircular canal, causing vertigo.
What is the treatment for BPPV?
Treated with carnality repositions maneuvers which are passive movements used to remove the otoconia from the canals, thus remediating vertigo
What is the Dix-Hallpike test?
Maneuver that is a vertiginous position test used in assessment and treatment
What does the Dix-Hallpike test stimulate?
Stimulates the posterior semicircular canal and attempts to determine if otoconia exist within the canal
What is Dix-hallpike test used for?
If pt experiences nystagmus and vertigo, the test is performed to determine if a pt presents with BPPV or a central lesion
Steps of the Dix-Hallpike
What is a Nystagmus?
When observing Nystagmus what should the PT be observing?
Eye movement: Horizontal, vertical, rotatory or mixed movements
Type of eye movement: Pendular or jerk
Direction: Bidirectional or unidirectional
Nystagmus movement: Binocular or monocular with symmetrical or dissociated movement. Effects of change of position of the head or posture on Nystagmus
What is congenital Nystagmus?
Typically mild and does not change in severity over the person’s lifetime. It is not usually associated with other pathology
Spontaneous nystagmus
imbalance of vestibular signals to the oculomotor neurons that causes a constant drift in one direction that is countered by a quick moment in the opposite direction.
When does a spontaneous nystagmus typically occur?
Occurs after an acute vestibular lesion and will last approximately 24 hours
Peripheral nystagmus
Occurs with a peripheral vestibular lesion and is inhibited when the pt fixates their vision on an object
Central nystagmus
occurs with a central lesion of the brainstem/cerebellum and is not inhibited by visual fixation on an object
Positional nystagmus
Induced by a change in head position. The semicircular canals stimulate the nystagmus that typically lasts only a few seconds
Gaze-evoked nystagmus
Occurs when the eyes shift from a primary position to an alternate position. The nystagmus is caused by patient’s inability to maintain the stable gaze position
Typically indicative of CNS pathology and is associated w/ brain injury and multiple sclerosis
Compare the direction of a central lesion vs peripheral lesion nystagmus
Central lesion: Bidirectional or unidirectional
Peripheral lesion: Unidirectional w/ the fast segment of movement indicating the opposite direction of lesion
Compare visual fixation of a central lesion vs a peripheral lesion
Central lesion: No inhibition with fixation
Peripheral Lesion: Will inhibit nystagmus and vertigo