adult etiologies
respir failure w/ refractory hypoxia
ARDS
alveolar hemorrh
PNA
lung tx
respir failure w/ refractory hypercapnia
copd
pediatric specific etiologies
respir failure w/ refractory hypercapnia
asthma
refractory hypoxemia
ards
PNA
mediastinal mass
PE
neonate specific etiologies
meconium aspiration
PNA
persistent air leak
severe respir failure (genetic lung dis)
congenital diaphragmatic hernia
function of VV
perform ventilation and oxygenation
anatomical drain sites
R/L IJ
R/L Femoral Vein
anatomical return sites
R/L IJ
R/L Femoral Vein
All locations transport blood to the RA**
Does VV impair native cardiac function
Depends..
native blood mixes with ecmo blood in the RA- then Tricuspid, RV, pulmonary valve, pulm arteries, lungs, pulm veins, LA, mitral, LV, aortic valve, aorta
Goal pt SVo2/ SCVo2
> 60-85%
trend only- do not use to titrate flow
represents mixing of pt venous blood = recirc pump arterial blood
T/F recirculation is always present
T
4 main causes recirc
CO
pump flow
RA size
cannula position
s/s recirc
pt spo2/sao2 dec
pre oxy ABG SVo2 inc
blood darker
Recirc- RA size
smaller RA/ dec preload and intravasc volume = inc r/f recirc
RA needs to be full enough that the ecmo blood can mix well w/ the native
trtmnt
give volume
blood/fluids
stop pulling crrt
recirc- pump flow
ECMO blood pressure so high that it shoots past SVC/RA junction and directly down to IVC and drain cannula
fix
turn down flow
recirc- CO
poor contractility or arrhythmias can impede foward flow of mixed ecmo blood through pulm circ and to the body
fix
control hr
improve oxygenation
inc BP
echo/XR to eval for tamponade/ inflammation
recirc- cannula position
cannula opening twisted towards wall of vena cava
moved up/down and away from tricuspid valve
fix
cxr
echo
goal pt spo2
> 85%
neonate max flow/ kg
main indicator value
pt spo2
goal pt Sao2
> 85%
measures percentage of hgb in blood that is fully bound to o2 vs unbound hgb
contraindications (adults)
terminal dis
pao2/fio2 <100 for >5 days
significant cns injury
mod- severe chronic lung dis
benefits of vv
dec pulm inflamm
reduces ischemic lung injury
no cardiac suppression (doesn’t dec preload, pulm blood flow, LA or LV output)
no LV stun
inc o2 supply to coronary circ
parameter used to adjust pt Pao2
FiO2/ flow
parameter used to adjust PCo2
sweep
reasons for conversion to VA
cardiac arrest
unresponsive pulm failure leading to cardiac compromise
inability to meet pt perfusion needs