Week 3 - ECGs + Basic Dysrhythmias Flashcards

(51 cards)

1
Q

What heart changes might be seen in someone who is a heavy smoker?

A

Atrial enlargement - abnormal P-waves

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2
Q

What does an inverted T-wave indicate?

A

ischemia

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3
Q

What ECG changes indicate cardiac injury?

A

ST Elevation
–> NSTEMI may also occur

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4
Q

pathological Q waves indicates what?

A

History of infarction

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5
Q

How do pacemakers affect ECGs?

A

It might be visible before the area that it is correcting - pacer spike

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6
Q

What patient will spend more time in the relative refractory period?

A

A person with a lower HR will see longer Q-T intervals
–> Higher risk of entering a lethal rhythm

Set up 12-lead and look at QTs to determine if it is truly prolonged

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7
Q

What is sinus rhythm?

A

Regular 60-100 bpm

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8
Q

What is the inherent rate of the AV junction?

A

40-60 bpm
–> Junctional rhythms start here

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9
Q

What is the inherent rate of the Purkinje fibers?

A

20-40 bpm

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10
Q

One small square on an ECG is how many seconds?

A

0.04 seconds
–> 5 boxes = 0.2 seconds

5 large boxes = 1 second.

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11
Q

The P-wave is related to atrial depolarization. What does a good P-wave look like?

A

Upright, round, uniform
–> Followed by QRS complex

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12
Q

What does a PR interval represent? How long should it be?

A

Represents the time taken for the impulse to travel from the SA to the AV node.
–> 0.12-0.20 seconds

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13
Q

What ECG changes indicate a 1st degree heart block?

A

A PRI longer than 0.20 seconds

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14
Q

What does the QRS complex represent? How long should it be?

A

Ventricular Depolarization + Atrial repolarization
–> 0.06-0.12 seconds

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15
Q

What does the T-wave represent? What does a healthy one look like?

A

Ventricular Repolarization
–> Upright, round, uniform. Follows QRS complex

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16
Q

What does a QT interval represent? Why is a prolonged QT concerning? How long should a QTi be?

A

Length of time it takes for the ventricle to depolarize and repolarize
–> Increased time in relative refractory period means increases susceptibility for ventricular dysrhythmias
–> 0.36-0.44 seconds

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17
Q

What are the steps of interpreting an ECG?

A
  1. Rhythm
  2. Rate
  3. P-waves
  4. Determine PRI duration
  5. Determine QRS complex duration
  6. Evaluate T wave
  7. QT interval
  8. Other components (ectopic beats, ST segment)
  9. Assess: stable or unstable
  10. interventions
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18
Q

How long does an ECG need to be in order to be interpreted?

A

6 seconds - 30 big boxes

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19
Q

How can you measure regularity of an ECG?

A

Map r-wave to r-wave on a paper and determine if they are equal (Ventricles)

Then do the same for p-waves (Atria)

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20
Q

How can you measure the HR of a regular HR on ECG?

A

Multiply the 6 second strip by 10.

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21
Q

What is the 1500 method? When is it used?

A

Used to determine rate of regular ECGs. Most accurate, useful for atrial flutters/tachy
–> Count small boxes between P-waves. Divide 1500 by the distance between p-waves.

22
Q

What does a premature atrial contraction look like?

A

A p-wave that looks different than others or is not followed by a QRS complex

23
Q

What does a premature junctional beat look like?

A

Absence or inversion of a p-wave.

24
Q

What is a premature ventricular complex?

A

Absent p-wave and QRS complex > 0.12 seconds

25
What is considered ST elevation or depression?
1-2 mm outside of normal range that lasts for 1 minute with or without symptoms
26
What are some possible etiologies of sinus bradycardia?
--> Sleeping, well conditioned athletes --> Inferior or posterior MI --> Vagal stimulation --> Carotid sinus pressure --> Increased ICP --> Obstructive sleep apnea --> CCB, digitalis, b-blockers, amiodarone Hypo...xia, thermia, thyroidism, K. HyperK
27
If someone has sinus bradycardia what should you ask?
Wonder if this is normal for the person --> Sudden drops can indicate decompensation Assess for symptoms (Chest pain, SOB, LOC changes, HypoTN)
28
If someone is symptomatic with sinus brady what interventions should be done?
--> O2 --> IV access --> Notify Physician --> Administer atropine --> Consider pacemaker (transcutaneous, transvenous, permanent)
29
What are potential etiologies of sinus tachycardia?
Increased workload --> Fever, exercise, pain, anxiety, hypoxia, HF, acute MI, infection, sympathetic stimulation, hyperthyroidism Medications --> Epi, atropine, dopamine, dobutamine, caffeine, nicotine, cocaine, amphetamines, cannabis Hemodynamics --> Shock, dehydration/hypovolemia
30
What interventions should be taken if someone has sinus tachy and is symptomatic?
--> O2 --> Ensure IV access --> Call physician --> Correct etiology
31
Why is sinus tachy more concerning in someone with CAD/cardiac pathology?
Less time in diastole --> Less coronary perfusion in someone who has underlying perfusion issues.
32
What is the visual difference between a-flutter and a-fib?
Saw tooth pattern vs fibrillatory waves
33
What is considered a controlled ventricular rhythm when there is an uncontrolled atrial rhythm?
Less than 100.
34
What is meant by 4-1 controlled a-flutter?
Atrial flutter where there are four atrial contractions per ventricular contraction. It is regular. If it were not regular it would be considered variable a-flutter
35
What is a superimposed t-wave?
When another wave is hidden within the t-wave - such as a p-wave in a-flutter
36
When do we want to cardiovert someone with new-onset atrial fib/flutter?
Within 48 hours --> Prevent ischemic stroke/PE
37
What is cardioverting?
Applying pressure on the R-wave to convert someone back into a normal atrial rhythm
38
What are the two rhythms that can be defibrillated?
V-tach without pulse V-fib
39
If someone is in pulseless V-tach, what are the interventions?
--> Call the code --> CPR --> Delegate for crash cart, compression board, airway --> defibrillate --> O2 (Bag or intubate) IV EPI, amiodarone or lidocaine, and mag Treat reversible causes (Hs and Ts)
40
What do you do if someone is in stable V-tach/wide complex tachy?
--> Notify physician --> O2 --> IV access - amiodarone/procainamide
41
What are the interventions for asystole?
--> CPR --> Crash cart --> O2 (FiO2 100%) --> Ensure IV Access - Epi, atropine --> Treatment reversible causes
42
What are some possible causes of cardiac arrest?
Hypovolemia, hypoxia, acidosis, K, hypothermia, hypoglycemia Tamponade, toxins, tension pneumothorax, thrombus, trauma
43
What is supraventricular tachycardia?
Tachys originating from above the ventricles - junction, atria
44
What are the phases of repolarization?
0 - Rapid depolarization 1 - Early repolarization 2 - Plateau 3 - Rapid depolarization 4 - Resting potential
45
What do you do if someone is in unstable wide complex tachycardia?
Call MD stat /+ RACE team O2 IV access Cardioverting
46
What might a rapid HR with narrow QRS complexes indicate? What can you do to treat it?
Hypovolemia or... --> Administer blood/fluids Tamponade - also presents with JVD and muffled heart sounds --> Treat with a pericardiocentesis, which will require a thoracotomy
47
What might brady with cyanosis indicate? How is it treated?
Hypoxia --> Airway, ventilate with O2
48
What might a might a low amplitude QRS indicate? How is it treated?
Occurs with acidosis --> Treated with ventilation, sodium bicarb, or dialysis
49
What might flat T waves and a U wave indicate? How is this treated?
HypoK --> Give potassium
50
What might a peaked T wave with wide QRS complexes indicate? How is it treated?
HyperK --> Shift or dialyze
51
How can we treat a PE?
Embolectomy, fibrinolytics, anticoagulation