week 6 Flashcards

(51 cards)

1
Q

What do we mean by low lung volumes?

A
  • reduced tidal volume (shallow breathing)
  • reduced vital capacity (inability to take a deep
    breath)
  • reduced FRC (generalised loss of volume)
  • reduced TLC (generalised loss of volume)
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2
Q

Reduced Tidal Volume (VT) results in

A
  • less gas moving in & out of the alveoli
  • less fresh gas in the alveoli
  • less O2 to move into the blood
  • O2 movement problem
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3
Q

Reduced VT (shallow breathing) without Sighs results in

A

less gas moving in & out of the alveoli
reduced stretch of the alveoli
reduced surfactant production
increased surface tension/more inwards recoil
Smaller alveoli (some may even close)
General loss of volume (FRC) & Changed distribution of ventilation

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4
Q

Reduced VC results in

A

reduced Inspiratory volume/deep breath in
reduced volume to exhale
reduced expiratory flow rate
reduced cough effectiveness
Secretion movement problem

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5
Q

FRC is

A
  • The amount of air left in your lungs after a normal breath out
  • The inward recoil of your lungs equals the outward recoil of the chest wall
  • Allows gas exchange to occur continuously in the clungs
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6
Q

FRC is reduced in

A

Chest wall and /or lungs less compliant
* Supine position
* Obesity
* Anaesthesia
* Thoracic & abdominal surgery
Chest wall more compliant/floppy so lung recoil pulls chest wall in
* Neonates
* Acute cervical spinal cord injury (SCI)

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7
Q

what is Closing Capacity

A

The point at which dependent small airways close during inspiration & expiration

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8
Q

Most common causes of increased CC (closing capacity)

A

when airways lose rigidity (become more floppy), as in
– Increased age
– Smoking

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9
Q

what happens when CC > FRC

A

small airways close during normal
breathing

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10
Q

what happens when FRC > CC

A

gas exchange to continuously occur

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11
Q

common causes of Localised Volume Loss

A
  • Lobar or segmental lung collapse
  • Pleural effusion
  • Rib #
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12
Q

Consequences of a Pleural Effusion

A
  • Disassociates the chest wall from lung (reduces intrapleural pressure)
  • Underlying lung tissue is not expanded
  • Eventuates in closure of underlying alveoli ->
    localised O2 movement problem
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13
Q

Consequences of Fractured Ribs

A
  • Pain causes a decrease in tidal volume
  • Chest wall may be unstable due to # -> reduced local chest wall movement
  • Chest wall may be stiff due to soft tissue injury
  • Eventuates in secondary atelectasis or collapse
    PLUS
  • Any bruising of underlying lung tissue alters the surfactant production and directly stiffens the lung
  • Consequently impairments can include
    – General O2 movement
    – Localised O2 movement problem
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14
Q

Effect of Reduced Lung Volumes on
Secretion Clearance

A

increased risk of infection

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15
Q

Signs & Symptoms of Reduced Volumes

A
  • May complain of shortness of breath
  • Observation/Palpation:
    – reduced expansion of chest wall
    – increased RR
    – +/- use of accessory muscles
  • Cyanosis (if PaO2 reduced significantly)
  • Auscultation:
    – reduced or absent breath sounds
    – fine end inspiratory crackles
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16
Q

Relaxed Breathing aim

A

encourage relaxation of the upper chest and
shoulders

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17
Q

Techniques to Increase Lung Volumes

A
  • increase inspired volume
    – Deep breathing exercises including breath holds
  • increase FRC
    – Upright positioning
  • Re-expand localised area of loss of volume
    – Specific positioning
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18
Q

Aims of Deep Breathing Exercises

A
  • Increase gas movement (O2) by increasing tidal volume and FRC
  • Prevent possible secondary effects on secretion clearance
  • Prevent possible secondary effects on respiratory load
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19
Q

Factors that affect distribution of ventilation during deep breathing exercises

A

– Volume of inspiration
– Muscle recruitment
– Inspiratory flow rate
– Addition of inspiratory hold (end inspiratory
pause)
– Alveolar interdependence

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20
Q
  • Intercostal / accessory muscle recruitment: leads to gas going
A

to non-dependant regions

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21
Q

Diaphragm recruitment: leads to gas going

A

to dependant regions

22
Q

what is the impact of a slow inspiratory flow rate

A

reduces the effect of airway resistance on where gas goes

23
Q

Inspiratory Hold

A
  • Adding a 2-3 second pause at the end of a deep breath
  • Allows gas to travel through collateral channels, opening up adjacent closed alveoli
24
Q

Alveolar Interdependence

A
  • Alveoli are connected to each other and therefore a change in volume in one exerts a stretch on those around it
25
How does specific positioning help/work?
* Place the affected region uppermost * Gravity will stretch the uppermost area open (more than the lowermost) * The greater the distance between the uppermost and lowermost regions of the lung, the greater the effect
26
Mucociliary Clearance (MCC) is
– Primary clearance mechanism of the airways – Protection mechanism of the lungs
27
* MCC apparatus consists of:
MUCUS * 2 layers * Functions as a mechanical, chemical and biological barrier CILIA * Moves particulate material caught in mucus towards the pharynx
28
what are the 2 layers of the mucus
Sol layer Gel layer
29
Sol layer (apart of the mucus layer) is
– Thin, watery/aqueous fluid that “bathes” the cilia – Provides minimal resistance to cilial movement – Has very low viscosity & elasticity (it behaves as a liquid)
30
Gel layer (apart of the mucus layer) is
– Is both elastic and viscous (more like a gel) – Moved towards the mouth
31
what is the normal amount of secretions
10 - 100 ml/day
32
Mucus produced by
– Submucosal glands – Goblet cells – (Clara cells) – Type II alveolar cells (surfactant)
33
Mucus consists of
– H2O – DNA, cellular debris – Trapped foreign matter (eg bacteria) – Glycoproteins – Lipids
34
MCC can be increased by
* Posture (use of gravity) * Exercise * Environment
35
MCC can be decreased by
* Increasing age * Sleep * Disease/injury
36
what is the reason if there is secretion production is normal or increased (but not excessive)
– Impaired MCC (back up cough) – Ineffective cough – Impaired MCC + ineffective cough
37
what is the reason If excessive secretion production (> 30ml/gm)
– Impaired MCC (inherent) – Ineffective cough
38
increased size & number of secretory cells caused by
* Smoking * Chronic bronchitis
39
increased volume mucus produced caused by
infection
40
Where decrease depth Sol (periciliary fluid) layer caused by
cystic fibrosis
41
* Where rheological properties are altered caused by
cystic fibrosis, asthma
42
what causes Destruction of cilia
* Smoking * Infection * Mechanical damage: endotracheal tube (ETT) * Burns * Chemical damage:
43
what causes cilia action altercation
aging, anaesthetic
44
what causes immotile cilia
Genetic disorders such as primary cilial dyskinesia/ Kartagener’s syndrome
45
Components of an effective cough
1. Deep inspiration 2. Breath hold against closed glottis 3. Contraction of the expiratory muscles resulting in large increase in intrathoracic pressure 4. Glottis opens and there is a rapid expulsion of air
46
Two-Phase Gas-Liquid Flow
Acceleration of air flow (gas) across the secretions (liquid) lining the airway walls is responsible for the cephalad (mouthward) movement of secretions (transfers the momentum from the gas to the liquid)
47
what is MIST FLOW
the liquid is carried as small droplets in the gas (associated with coughing)
48
ANNULAR FLOW
the surface of the liquid layer moves in waves (associated with huffing)
49
Reasons for an Ineffective Cough
1. Inability to take deep breath 2. Inability to close glottis 3. Weak expiratory muscles 4. Excessive dynamic compression (closure) 5. Depressed cough reflex eg sedation
50
Equal Pressure Point (EPP) is
EPP is the point at which the pressure inside the airway wall is equal & opposite to the pressure outside the airway
51
Summary of forced expiration
1. +ve pressure with expiratory muscle contraction 2. Greater +ve pressure occurs in alveoli with expiratory muscle contraction 3. +ve pressure drops in the airways as air flows out 4. EPP occurs at some point along the airway 5. Dynamic compression and increase EFR in front of EPP (towards mouth) will move the mucus via annular two- phase gas-liquid flow