Using the lowest FiO₂ possible prevents nitrogen washout and alveolar collapse.
Huff coughing is effective for mobilizing secretions without airway collapse.
Repositioning and mobilization expand alveoli, reducing risk of collapse.
The incentive spirometer directly promotes alveolar inflation and prevents collapse.
Regular deep breathing and coughing keep alveoli open and clear secretions.
Repositioning and deep breathing can reopen collapsed alveoli before escalating treatment.
Bronchoscopy can remove mucus plugs or foreign bodies causing obstruction.
Adequate pain relief enables effective deep breathing and coughing.
Incentive spirometry re-expands alveoli and relieves tachypnea from atelectasis.
Chest physiotherapy mobilizes secretions and improves lung aeration.
Positive pressure ventilation (CPAP/PEEP) reopens alveoli when oxygen alone is insufficient.
Better oxygenation and auscultation show interventions are effective.
Immobility worsens secretion stasis and atelectasis progression.
Breathing techniques improve alveolar expansion and reduce atelectasis risk.
Atelectasis impairs oxygen delivery, making gas exchange the priority diagnosis.
Absent breath sounds suggest lobar collapse, requiring immediate provider notification.
Bronchoscopy clears secretions or obstruction, restoring ventilation.
Anxiety worsens dyspnea; reassurance and guided breathing improve outcomes.
Continued breathing exercises and daily activity prevent recurrence.
Humidified oxygen loosens secretions, aiding clearance and alveolar expansion.
Tachypnea is an early sign as the body attempts to compensate for reduced gas exchange.
Chest X-ray is the standard initial tool, showing volume loss, opacity, or mediastinal shift.
Collapsed alveoli produce diminished or absent breath sounds on auscultation.
Low-grade fever and tachypnea are common bedside findings due to inflammation and impaired ventilation.