Define acute AF? How would you approach the management?
Acute AF is AF that started <48 hours ago
1) Are they haemodynamically stable?
If no –> emergency cardioversion (2nd line = amiodarone)
If yes –> 1) Rate control = b-blocker or diltiazem
2) Rhythm control (ONLY if <48h) = DC cardioversion or amiodarone
3) Start LMWH if chronic
4) Treat underlying cause
Define paroxysmal AF? How would you manage it?
DEF: self limiting + lasts <7 days
Mx:
Acute: Medical
Anticoagulate + rate/rhythm control
Rate control: diltaziam or a beta blocker (Eg Bisoprolol)
Rhythm control: amiodarone
Treat underlying cause
Mx of Ventricular tachycardia
If haemodynamiacally unstable, ie pulseless? –> CPR Adverse signs (Chest pain, HF) –> sedate + synchronised cardioversion!
If haemodynamically stable (ie no signs, pulse present)
-Beta blockers (Bisoprolol)
Ix:
ECG changes in hypokalemia
TIA - best brain imaging?
Diffusion weighted MRI
TIA - management? 5 aspects
What scoring system is used to determine risk of stroke in TIA patients?
ABCD2 Age >60 BP >140/90 Clinical Findx: unilateral weakness (2), speech disturbance w/o weakness (1) Duration Diabetes
Fluid resuscitation formula in burns? Which fluid?
Parkland formula:
4 x wt x % surface area
Give half in first 8 hours HARTMANN’S, warmed
How to determine % BSA involvement in burns?
1 arm: 9%
Head and neck : 9%
Front torso: 18%
Back torso: 18%
1 leg: 18%
Perineum: 1%
1 hand: 1%
Tx of burns (exc fluid resus)
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“Burns, a dress could split, tits exposed”
DKA management summary
(Assuming its confirmed with pH, blood glucose and ketones)
INSULIN: 0.1 U/kg/hr Actrapid infusion with 0.9% Saline
-Aim ketones reduction >0.5mM/hr
FLUIDS:
If SystolicBloodPressure < 90 = 0.9% Saline 1L stat (Over 15 minutes)
If SBP > 90 = 0.9% Saline 1L over 1hr –> add K+ (potassium) if <5.5 (20mM/L)
Consider LMWH
Definition of DKA
pH < 7.3
glucose > 11.1
ketones > 3.0 (or 2+ on dip)
Definition and Mx of HONK
Hyperglycemia (>35) w/o ketones
Fluids Potassium in 2nd bag of fluids
LMWH
?consider insulin
In upper GI bleed, how is a pt managed AFTER endoscopy?
Depends on cause:
/////////////OG answer
NBM + stop NSAIDs IV omeprazole Daily bloods H. pylori testing + eradication
ABCDE approach: How would you assess the airway?
Ensuring a patent airway
If speaking –> patency assumed.
Look –> for airway secretions, angioedema
Listen –> gurgling Breathe Sounds or stridor Feel –> for expired air -
Any evidence that airway is not patent –> HEAD TILT + CHIN LIFT (Jaw thrust if C-spine injury) -
Consider airway adjuncts eg Guedel or nasopharyngeal airway - If still not achieved –> check pulse + periarrest call!!!
ABCDE approach: How would you assess breathing?
Ensuring target saturations are met
OBS –> Sats, RR,
administer 15L 02 non-rebreather mask
Inspection –> accessory muscle breathing, nasal flaring Palpate –> Tracheal deviation, equal chest expansion Percussion –> dullness?
Auscultate –> equal air entry, wheeze, crackles
ABG + CXR
ABCDE approach: assessment of circulation?
Obs: CRT
pulse - rate and rhythm,
BP in both arms
Ask nurse to help w 12 lead ECG
Bloods: FBC, U+Es, glucose, CRP, Xmatch, G+S, clotting, blood cultures, troponin
ABCDE approach: assessment of disability?
Causes of shock
“Could Someone Hurry On down?”
CARDIOGENIC:
SEPTIC:
HYPOVOLAEMIC:
OBSTRUCTIVE:
DISTRIBUTIVE (ie 3rd spacing):
Causes of hypovolemic shock:
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How to differentiate between cardiogenic vs hypovolemic shock?
Cardiogenic: high JVP
Hypovolemic: low JVP
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Cardiogenic: JVP: high, peripheries: cold
Sepitc: JVP: low, peripheries: warm
Hypovolaemic: JVP: low, peripheries: cold
How to differentiate between septic shock vs hypovolemic shock
Septic: warm peripheries
Hypovolaemic shock: cold peripheries
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Cardiogenic: JVP: high, peripheries: cold
Sepitc: JVP: low, peripheries: warm
Hypovolaemic: JVP: low, peripheries: cold
Pathophysiology of anaphylaxis
Type 1 IgE mediated hypersensitivity reaction Mast cell degranulation –> histamine release –> inc vascular permeability + bronchoconstriction
Dose of adrenaline in anaphylaxis?
IM adrenaline 500micrograms