(ADHD) HiTop Category
under disinhibited and antagonistic externalizing disorders under antisocial behaviour
ADHD
-motoric and verbal hyperactivity
-problems maintaining focus in conversations and activities
-impulsive or erratic behaviours
-not episodic, but a way of being in the world but symptoms do fluctuate depending on the circumstances
Untreated ADHD
if adhd is left untreated, lower educational attainment, early pregnancies, more run ins with the law etc and other lasting effects are more likely to occur
(ADHD) Onset
-symptoms almost always emerge in early childhood, some improve with age and brain maturation
-but at least ⅓ retain their diagnosis in adulthood
(ADHD) Specifiers
-specifiers: ADHD-I (inattention), ADHD-H (hyperactivity), or ADHD-HI (mixed)
(ADHD) George Still Definition
-lack self control
-showed symptoms of overactivity/inattention in school
-still noted they had poor inhibitory volition and defective moral control
ADHD & Influenza Outbreak
(ADHD) 1950s Definition
hyperkinesis
* attributed to poor filtering of stimuli entering brain
* Led to definition of hyperactive child syndrome
* motor overactivity seen as main feature
ADHD Contreversies
-skeptisicim about the diagnosis
-abnormal development (then persistent symptom into adulthood) or lag in dveelopment (would resolve by adulthood)
-ADHD in girls and women
(ADHD) Can’t Be Explained By
-a better suited disorder
-not present in just one setting, bc then behviour could be attributed to the setting
-must be an inability to do these things, not a refusal
Parents w/ADHD
Problems for Families w/ADHD
(ADHD) Prenatal Factors
(ADHD) Genetic Predisposition
about 1/3 of biological relatives of children with ADHD have the
disorder
(ADHD) Adoption Studies
(ADHD) Twin Studies
(ADHD) Heterotypic Continuity Explanations
1) failure models
2)shared etiopathogenic factors
(ADHD) Failure Model
Impulsivity/ hyperactivity/ inattention lead to higher levels of interpersonal conflict (with peers, teachers, parents), rejection, lack of support, and poor skills development
* All of these factors increase risk of subsequent depression and other forms of psychopathology
* Difficulties in social interactions significantly predict depression
* Peer rejection mediates the relationship between aggression and depression
(ADHD) Shared Etiopathogenic Factors
(ADHD) Homotypic Continuity
-having a disorder at one time point predicts having the same disorder at a a later time point
-same disorder evident at different points in development
-ex. ADHD in childhood is assc w/ ADHD in adolescene and or adulthood
(ADHD) Heterotypic Continuity
-having a disorder at one time poitn predicts having a different disorder at a later time point
-underlying processes or factors that contribute to one disorder can also lead to the development of a different disorder over time, behaviours and association with ADHD could make them predisposed
-ex: ADHD in childhood is associated with MDD or SUD in adoleadnce or adulthood
(ADHD) ALSPAC Study
ADHD in Adolesecne
-Some outgrow
* At least 50% of clinic-referred elementary school children don’t; sometimes problems get worse
* Significant impairments continue emotional, behavioral, and social functioning
* Children who met diagnostic criteria for ADHD in childhood (ages 5–11): in adolescence more likely to exhibit oppositional defiant behaviors, anxiety/depression, and involvement with the juvenile justice system
ADHD in Adulthood
-rates of ADHD diagnosis persistence ranged from 5.7% to 77%
-all studies observed high rates of symptomatic persistence ranging from 60% to 86%
-doesnt remit, or continues in a form not captured by criteria
-hyperactivity less relevant for adults
-adults may be able to better mask or make sympoms less relevant