disorders 2 Flashcards

(48 cards)

1
Q

what happens before DSM is published?

A
  • clinician consultations
  • advocacy groups
  • general public
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2
Q

what is our perception of psych disorders impacted by?

A
  • biased media portrayal
  • mainly extremes are portrayed on TV and films
  • can increase fear of those w/ mental illnesses
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3
Q

dyslexia in DSM 4 vs 5 + over the lifetime

A
  • in DSM 4, it was a single disorder
  • in DSM 5, it is in category of specific learning disorder
  • in preschool: hard to learn new words/rhyme
  • in school: below reading level spelling, following instructions
  • in teen/adult: can’t summarize story, memorize, time management = problems w/ independent living
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4
Q

etiology of major depressive disorder can be understood through…

A

can be understood through hypotheses and treatments

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5
Q

mindfulness based cognitive therapy (MBCT)

A
  • combines CBT with mindfulness
  • present-focused therapy designed to prevent depressive relapses
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6
Q

emotion brain regions involved in depression

A
  • amygdala (emotional processing + regulation)
  • middle frontal gyrus (cognitive control + emotions)
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7
Q

one study let half continue w/ SSRIs and other half included MBCT in an eight week program. results?

A
  • functional activation between MFG and amygdala was measured
  • combined w/ MBCT causes even better communication w/ those two regions
  • depressive symptoms decreased more than just the pharmacological treatment AND has a longer lasting effect
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8
Q

three main categories in which MBCT improved depression symptoms

A
  • decrease in repetitive negative thoughts abt future and past
  • decrease in emotional and cognitive reactivity
  • increase in self compassion (self kindness, recognize selfs humanity, recognizing negative thoughts as a part of life)
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9
Q

auditory visual synesthesia

A
  • auditory system can be connected to visual system
  • when listening to music we can see it
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10
Q

auditory taste synesthesia

A
  • when someone can taste the names of others
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11
Q

colour graphing synesthesia

A
  • most common
  • instead of seeing black letters they see colour patterns
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12
Q

defining traits of synesthesia

A
  • involuntary/automatic
  • consistent
  • highly memorable
  • laden with affect
  • spatially extended
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13
Q

time space synesthesia

A

can visualize a ring around them that represents their calendar with months/dates that can be expanded

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14
Q

neural development and synesthesia

A

that connections between neurons don’t get pruned, so cross-activation occurs

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15
Q

hunger is…

A
  • a multisensory experience
  • taste + smell = flavour
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16
Q

letters and colours have…

A
  • natural associations
  • like kiki bouba
  • we also know what colours letters have
    = we all have a little bit of synesthesia
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17
Q

etiology

A
  • cause, or set of causes, of a disorder
  • not well understood for any given disorder
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18
Q

three etiological models

A
  • biological
  • behavioural
  • cognitive
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19
Q

biological model
- also known as medical/disease model

A
  • assumes that psych. disorders result from malfunction in the brain
  • points to genetics, atypical neurotransmitter activity, or abnormal brain structures
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20
Q

how do we gain insight into genetic etiology

A
  • twin studies
  • if two twins share a trait, they have concordance
  • heritability of disorders can be estimated by comparing concordance rates of identical/fraternal twins
  • IF identical twins are likelier to share a specific disorder, can be concluded that genetics play a larger role
21
Q

what do researches do after determining that a genetic component exists to a disorder?

A
  • identify the specific genes involved
  • genome wide association studies
22
Q

genome wide association studies + problems

A
  • involve scanning of 1000s of genetic markers to find variations that show up more often in affected individuals

problems:
- can be contradictory/inconclusive
- associations are rarely as direct/straightforward
- someone could have alteration but not show disorder or vice-versa

23
Q

which neurotransmitters are implicated in depression

A
  • low lvls of dopamine and norepinephrine
  • seratonin + GABA implicated in etiology of depression/anxiety
24
Q

struggles with drug therapies for altering levels of neurotransmitter activity

A
  • varied and unpredictable success
  • emerging view is that neurotransmitter activity is part of the explanation, not the whole story
25
patients with anxiety disorders tend to have hyperactive ______
- amygdalas - correlation is not causation: anxiety could’ve caused the amygdala not other way around
26
environmental model
- considers environmental factors - i.e. where we live, who we socialize with, what we consume
27
prominent etiological explanation for schizophrenia
diathesis stress model
28
diathesis stress model
- genetic predisposition: diathesis pairs with… - environmental stress - ppl w/ high diathesis need less environmental stressors to develop schizophrenia - ppl w/ no diathesis and high stress would not lead to the disorder
29
environmental risk factors that reoccur across near every disorder except for bipolar/eating disorders
low socioeconomic status
30
problem with diathesis stress model
- many risk factors exist (born in winter, exposure to cats, etc) - ultimately, schizophrenia is very complex - can’t just rely on stress model bc it can’t encapsulate alllll risk factors
31
researchers aren’t sure what environmental events trigger schizophrenia. what is the leading candidate?
- stress and problems in relationships with others (esp immediate family) - those with schizophrenia live in homes with higher levels of dysfunction - HOWEVER. hard to tell if dysfunction existed before disorder or if it appeared as family tried dealing with symptoms
32
behavioural model
- external factors aren’t the issue, but rather our response to them - view disordered behaviours established though classical/instrumental conditioning - rewards, punishments, and contingencies received for actions influence our behaviour - behaviours can be inappropriately generalized from situations where they have appropriate function to situations where they don’t
33
what does the behavioural model suggest about depression?
- arises in individuals who lack social skills - this makes in hard for them to elicit normal positive social reinforcement from others - this can lead to lowered mood and self-blame of depression
34
negative feedback loop in depression
- depressive symptoms may elicit sympathy, attention, and concern - leads to reinforcement of the symptoms
35
somatic symptom disorders
- individuals experience genuine anxiety/fear and become afflicted by potentially serious health condition - real physical suffering, but there’s no physical basis - i.e. neurological issues, general pain, skin rashes
36
if a physical symptom is produced purely through psychological stress, it is said to be…
psychogenic
37
glove anesthesia
- absence of sensation/feeling in hand ending abruptly at wrist - makes no sense bc sensory nerve fibers tht supply hand also supply lower arm
38
what does the behavioural model say about somatic system disorders?
a method tht individuals unknowingly use to receive positive attention and nurturance from caring medical professionals
39
how many cases do somatic symptom disorders make up?
25% of general medical patients and 5% of neurology referrals
40
what is a strong reason for why the behavioural model is still relevant in the modern context?
psychosomatic symptoms
41
- dogs were exposed to mild electric shocks in cages they couldn’t escape - at first, they tried to avoid the shocks but eventually became passive - then, when the same dogs were put in cages with open doors, what happened?
stayed inside passively
42
how is the theory of learned helplessness used in depression? what r the downsides?
- depressive symptoms are the result of a more sophisticated learned helplessness - however…. - not all behaviours can be learned (i.e. hallucinations) - doesn’t always transfer to all environments - treats people as simple reflexive beings vs. ppl w/ ability to plan/rmbr/predict things
43
cognitive model
- suggests that mental disorder results from the maladaptive or inappropriate ways of selecting/interpreting information from the environment - not bc of events, but how we interpret the events
44
what does the cognitive model explain about maladaptive thoughts?
- that they are reinforced by self-generated misperceptions about reality - misperceptions are called cognitive distortions
45
catastrophizing
- extreme distortion of a situation - when we overemphasize the negative outcomes of a rather mundane situation
46
cognitive distortions occur in the absence of ______
- any external evidence - individual relies on ONLY their self-generated misperceptions
47
according to the cognitive model, depression arises in what sorts of individuals?
- ppl who view neutral/positive events as being negative due to their own actions
48
depressnogenic schemata
- the particular maladaptive way depressed individuals evaluate themselves/experiences - these people develop unrealistically negative/demeaning interpretations of those events - leads to a negative world view of themselves, the world, and their future