acute fatique
decrease in maximal force or power production in response to contractile activity
anaerobic pathways for ATP
pcr and glycolysis
Pcr re-synthesis of ATP depends on
creatine levels
type 1s use
mitochondria and some glycolsis and PCr
fuel used for type 1s
fat
glycogen depletion rate for type 1 fibers
slow
type 2 use
both mitochondria and glycolysis (some pcr)
fuel used for type 2
both cho and fat but mainly CHO
type 2x use
Pcr and glycolysis
glycogen depletion rate for type 2x
very fast
glycogen is depleted more from which fiber
fiber 2
traid is
the area of ATP demand/use
where do we use ATP
CA Pump
NA - K pump
myosin ATPase
SR
AP
what pathway is relied upon for high intensity exercise
glycolysis
if glycolysis isnt produced fully then
fat becomes ATP source but it is too slow for high intensity exercise
the 3 things involved in the triad is
1, membrance - Ca pump and Na-K pump
2. SR Ca pump
3. sarcomere - myosin ATPase
if atp cant resynthesize fast enough, what builds up
Pi
excessive build up Pi inhibits
cross bridge cycling by reducing myofibillar sensitivity to CA
extra Pi seeps into
SR and binds to CA so that it can leave to initiate cross bridge cycling
-so troponin cant bind to CA
Ca apperance stimulates
glycogenolysis
low Ph
high amount of H
-acidic
high ph
low amount of H
-basic
what does LDH do
converts pyruvate to lactate
hydrogen is being produced when
pyruvate is being converted to lactate and when ATPase releases energy from ATP