Hf Flashcards

(45 cards)

1
Q

What are the two main priority concepts related to Heart Failure?

A

Perfusion and Gas Exchange.

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2
Q

What is ejection fraction (EF) and its normal range?

A

Percentage of blood ejected from the ventricle during systole; normal range 50–70%. EF <50% indicates heart failure.

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3
Q

Define preload.

A

The degree of stretch in the ventricles at the end of diastole, just before contraction.

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4
Q

Define afterload.

A

The pressure or resistance the ventricles must overcome to eject blood through the semilunar valves and into circulation.

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5
Q

What is heart failure (HF)?

A

The inability of the heart to pump effectively to meet the body’s metabolic needs. It leads to inadequate tissue perfusion and fluid overload.

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6
Q

What are common causes of left-sided heart failure?

A

Hypertension, coronary artery disease (CAD), and valvular disease.

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7
Q

What are common causes of right-sided heart failure?

A

Left ventricular failure, right ventricular MI, and pulmonary hypertension.

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8
Q

What are the three types of heart failure?

A

Left-sided, right-sided, and high-output failure.

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9
Q

How does the body initially compensate for decreased cardiac output?

A

Baroreceptor activation increases HR and vasoconstriction; kidneys retain sodium and water via the RAAS; ADH increases water retention.

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10
Q

What is ventricular remodeling?

A

Structural changes in the ventricles (dilation, hypertrophy) that reduce the heart’s pumping efficiency.

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11
Q

List key signs of left-sided heart failure.

A

Dyspnea, orthopnea, fatigue, pulmonary congestion (crackles, frothy pink sputum), cough, decreased perfusion.

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12
Q

List key signs of right-sided heart failure.

A

Jugular vein distention, dependent edema, ascites, hepatomegaly, weight gain, and increased abdominal girth.

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13
Q

What is the most reliable indicator of fluid gain or loss?

A

Daily weight.

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14
Q

What are key assessments for suspected HF?

A

History of cardiac disease, activity tolerance, dyspnea, urinary patterns, psychosocial factors.

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15
Q

What laboratory tests help diagnose HF?

A

BNP (elevated when ventricles are stretched), serum electrolytes, urinalysis, renal function tests.

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16
Q

What imaging is most useful in diagnosing HF?

A

Echocardiogram (determines ejection fraction).

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17
Q

What is the normal BNP value?

A

<100 pg/mL. Elevated BNP indicates worsening HF.

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18
Q

What ECG findings may occur in HF?

A

Dysrhythmias such as atrial fibrillation or ventricular ectopy.

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19
Q

Using the Clinical Judgment Model, what are key hypotheses for HF?

A
  1. Decreased gas exchange due to ventilation/perfusion imbalance. 2. Potential for decreased perfusion due to inadequate cardiac output. 3. Potential for pulmonary edema due to left-sided HF.
20
Q

What interventions promote gas exchange in HF?

A

Auscultate breath sounds, provide oxygen therapy, position upright (high-Fowler’s), monitor SpO₂, consider CPAP if needed.

21
Q

What are signs of pulmonary edema?

A

Crackles, dyspnea at rest, tachycardia, cough with frothy pink sputum, anxiety, low SpO₂, reduced urine output.

22
Q

How is pulmonary edema treated?

A

Administer diuretics (furosemide), nitrates (nitroglycerin), morphine to reduce anxiety and preload, and provide oxygen. Ultrafiltration may be used for severe fluid overload.

23
Q

What is the goal of drug therapy for HF?

A

Improve cardiac output, reduce preload and afterload, and enhance contractility.

24
Q

What are the main drug classes used for HF?

A

ACE inhibitors, ARBs, ARNIs, beta-blockers, diuretics, nitrates, and digoxin.

25
What do ACE inhibitors do?
Vasodilate and reduce afterload; improve survival. Watch for dry cough and hyperkalemia.
26
When are ARBs used in HF?
As alternatives for patients who cannot tolerate ACE inhibitors (e.g., due to cough).
27
What is the purpose of ARNI (sacubitril/valsartan)?
Combines RAAS blockade with neprilysin inhibition to improve outcomes and reduce mortality.
28
Why are diuretics used in HF?
They reduce preload by promoting sodium and water excretion; loop diuretics (furosemide) are most common.
29
What should be monitored with furosemide use?
Monitor potassium levels and for signs of dehydration or orthostatic hypotension.
30
What is digoxin’s mechanism of action?
Increases contractility (positive inotrope) and decreases HR. Watch for toxicity (blurred vision, halos, bradycardia).
31
What are signs of digoxin toxicity?
Bradycardia, nausea, vomiting, blurred or yellow vision (halos), and confusion.
32
What role do beta-blockers play in HF?
Reduce HR and myocardial workload, improve ventricular filling time (e.g., metoprolol, carvedilol).
33
What is the function of nitrates in HF?
Vasodilate veins to decrease preload and relieve pulmonary congestion.
34
What dietary recommendations are given to HF patients?
Low sodium (<2 g/day), fluid restriction (<2 L/day), small frequent meals, and referral to a dietician.
35
What is the purpose of weight monitoring in HF?
Detect fluid retention early; report gain of >2–3 lbs in 24 hours.
36
What education should HF patients receive for self-management?
Daily weights, medication adherence, activity pacing, avoiding smoking/alcohol, and recognizing worsening symptoms (e.g., dyspnea, swelling, weight gain).
37
What are symptoms of worsening HF that should be reported immediately?
Increased shortness of breath, swelling, rapid weight gain (>2–3 lbs in 24 hr), or reduced urine output.
38
Why is psychosocial support important in HF management?
HF is a chronic, progressive disease that can lead to depression and anxiety; addressing mental health improves adherence and outcomes.
39
What surgical options exist for advanced HF?
Heart transplant, left ventricular reconstruction surgery, or a ventricular assist device (VAD).
40
How does the nurse evaluate improvement in HF?
Improved gas exchange (SpO₂ ≥ 92%), stable weight, decreased edema, no crackles, patient verbalizes understanding of management plan.
41
What is the expected outcome for a patient with controlled HF?
Maintains adequate perfusion, stable weight, no pulmonary congestion, adheres to medication and lifestyle regimen.
42
CJM Case: Recognize cues for HF.
History of cardiac disease, activity intolerance, dyspnea, weight gain, fluid retention, abnormal BNP, low EF.
43
CJM Case: Analyze cues and prioritize hypotheses.
1. Decreased gas exchange. 2. Decreased perfusion. 3. Risk for pulmonary edema.
44
CJM Case: Generate solutions and take action.
Administer meds (ACEI, diuretics, digoxin), promote oxygenation, monitor weights, teach self-management.
45
CJM Case: Evaluate outcomes.
Improved perfusion and gas exchange; patient reports decreased dyspnea and understands management plan.