“JAAOS 2018 - Os Acromiale (JAAOS ‘18)”
“-common in blacks<div>-bilateral in 33%</div><div>-closure of physis at 25 years</div><div>-meso-acromion most common</div><div>-US can be 100% sensitive</div><div>-Treatment</div><div><img></img></div><div>-Tension banding decreases nonunion rate</div>”
Minimizing complications in Pilon Fractures
“<div>-early fixation = no increase in infx, but decrease LOS/cost<br></br></div><div>-primary fusion</div><div>-staged sequential posterior and anterior fixation</div><div>-acute shortening</div><div>-transsyndesmotic fibular plating</div><div>-upgrading (change a C-type to B-type: buttress oblique apex fracture while doing ex-fix)</div><div><br></br></div><div><div> <div> <div> <div>Summary</div> <ul> <li>Techniques for diminishing risk of soft-tissue and osseous sepsis:</li> </ul> <div>Early ““immediate”” fixation - decreases surgical time and LOS</div> <div>Staged ““delayed”” fixation</div> <div>Upgrading</div> <div>Primary arthrodesis</div> <div>Staged sequential posterior and anterior fixation</div> <div>Acute shortening</div> <div>Transsyndesmotic fibular plating</div> <ul> <li>Degree of articular reduction relates to arthrosis (but no clinical correlation</li> <li>Clinical outcome related to</li> <ul> <li>Social factors</li> <li>Medical comorbidites</li> </ul> <li>Open fractures: risk of vascular injury</li> <ul> <li>I and D, antibiotic beads, soft tissue coverage, staged bone graft</li> </ul> </ul> </div> </div></div></div>”
interbody fusions: open vs MIS
“<ul> <li>Kambin triangle: must do a facectectomy to get here.</li> <ul> <li>Borders: exiting nerve root, superior endplate of inferior vert body, traversing nerve root (articular process)</li> </ul> </ul> <div><img></img></div> <div></div> <ul> <li>MIS: less blood loss, less narcotic use, quicker ambulation, decreased LOS, less infection risk</li> <li>Open: less radiation exposure</li> <li>Equivalent: OR time, fusion rates, patient reported outcomes, complications</li></ul>”
Perc pedicle screw stabilization
<div> <div> <div> <ul> <ul> <li>Technique</li> <ul> <li>Good xray showing level endplate, SP bisecting pedicles</li> <li>Incision with lateral margin of pedicle</li> <li>Guidewire/Screw</li> <ul> <li>Startpoint: centered on lateral pedicle margin</li> <li>Trajectory: stay within laterral 2/3rds of pedicle</li> <li>Aim to be just past posterior vertebral wall</li> <li>Remove stylet, pass guide wire through trocar to anterior 1/3rd body</li> </ul> <li>Notes</li> <ul> <li>Cannulated screws have decreased pullout strength</li> <li>T-spine: TP can block trajectory so may need more medial startpoint, with more vertical trajectory</li> <li>Use of excessive rod contouring as sole method of indirect # reduction may result in screw stripping (especially w/ shallow purchase of cannulated screws) </li> </ul> <li>Reduction</li> <ul> <li>Pedicle screws at # level maximizes # reduction (Requires intact pedicles)</li> <li>3 phases of reduction: pt position, direct manip through endplate, indirect through ligamentotaxis viad distraction</li> </ul> <li>Screw types</li> <ul> <li>Monoaxial better for compression fractures to reduce anterior wedging and maintain anterior height</li> <li>Polyaxial easier for rroad passage and can achieve compression posterior for flexion distraction</li> </ul> <li>ROH at 3-4 months maintains motion</li> <li>Contraindications</li> <ul> <li>Severe comminution</li> <li>Nonintact pedicle/facet</li> <li>Inability to get startpoint/trajectory</li> </ul> <li>Outcomes: perc vs open</li> <ul> <li>Less EBL, maybe shorter OR time</li> <li>Deformity correction same</li> </ul> </ul> </ul> <div></div> <div></div> <div></div> </ul> </div> </div></div>
Cervical Laminoplasty
<div> <div> <div> <ul> <li>Summary</li> <li>Theory: Dorsal element-preserving, motion-preserving, posterior decompression via spinal canal expansion </li> <ul> <li>Originally for OPLL</li> </ul> <li>Consideration</li> <ul> <li>Cervical kyphosis is a contraindication</li> <ul> <li>Recommend lami + fusion for >15 deg kyphosis C2-C7</li> </ul> <li></li> </ul> <li>Techniques</li> <ul> <li>Unilateral open door</li> <ul> <li>Hinge on lamina-facet junction</li> </ul> <li>Bilateral double door laminoplasty</li> <ul> <li>Osteotomy at lamina-pedicle junction</li> </ul> </ul> <li>Outcuomes</li> <ul> <li>Pre-existing kyphosis >5 deg improve less</li> <li>Laminoplasty better without fusion</li> </ul> <li>Complications</li> <ul> <li>C5 palsy 6% (vs ACDF 2%)</li> <ul> <li>67% resolve by 4 months</li> <li>If severe - consider foraminotomies</li> </ul> <li>Axial neck pain</li> <ul> <li>Preserve semispinalais cervicis mm to decrease post-op neck pain</li> </ul> <li>C2-C3 stiffness from inadvertent interlaminar fusion</li> <li>Loss of lordosis</li> <ul> <li>Dissect between paraspinal mm and avoid going lateral to lateral masses</li> </ul> </ul> </ul> </div> </div></div>
Spinopelvic fixation
<div> <div> <div> <div>Summary</div> <ul> <li>SPF: powerful technique to resist flexion/cantilever forces that cause pseudarthrosis</li> <ul> <li>Fixation to pelvis provides stability anterior to pivot point which resists cantilever bending</li> </ul> <li>Adult sacrum: poor fixation: cancellous consistency, short/capacious S1 pedicles, sacral slope = shear forces vs. L5</li> <li>Options</li> <ul> <li>Iliac screws/bolts</li> <ul> <li>23/67 require ROH</li> </ul> <li>S2 Alar Iliac fixation</li> <ul> <li>Less ROH</li> <li>low profile, ability to align with rostral instrumentation, not require use of offset connectors</li> <li>long-term consequences of crossing SI joint with S2AI = unknown</li> </ul> </ul> <li>Outcomes: beneficial for:</li> <ul> <li>Patients: osteoporotic, with 3 column osteotomies, require fusion from TL junction to sacrum</li> </ul> <li>both iliac/S2AI screws fusion at LS junction</li> </ul> <div></div> </div> </div></div>
Discoid Meniscus: Classification, Investigation findings, tx
<ul> <li>Watanabe (arthroscopic classification)</li> <ul> <li>Type I - stable, complete discoid meniscus (covers the entire plateau)</li> <li>Type II - stable, partial discoid meniscus (covers up to 80% of the plateau)</li> <li>Type III - unstable, Wrisberg variant (Wrisberg is the only thing that attaches), lack of any other posterior meniscotibial attachments</li> </ul><li>Xray</li> <ul> <li>Squaring of lateral femoral condyle</li> <li>Widening of joint space</li> <li>Hypoplastic lateral tibial spine</li> </ul> <li>MRI</li> <ul> <li>Transverse meniscal diameter > 15mm</li> <li>Continuity between the anterior and posterior horns of the menisci noted on at least three consecutive 5mm thick sagittal slices</li> <li>Selective MRI no better than physical exam</li> </ul><li>Tx</li><li>meniscal rim preservation (6-8 mm) with arthroscopic saucerization</li> <li>After saucerization, assessment of meniscal stability is performed, because peripheral stabilization is necessary in patients with unstable discoid variants. Stabilization may be achieved through all-inside, inside-out, or outside-in suture repair. Stabilizing the hypermobile meniscus contributes to the ultimate goal of meniscal preservation</li> <li>In young patients a formal inside-out meniscal repair may be performed, using a posterolateral incision to protect the peroneal nerve, popliteal vessels, and tibial nerve during suture retrieval.</li></ul>
PMC injury of the knee
“<ul> <li>associated with anteromedial rotatory instability (AMRI)<br></br></li><li>components:POL, semimembranosus, OPL, capsule, PH of MM</li><li>injury: valgus and ER</li><li>P/E: AMRI (subluxation of AM plateau on femoral condyle), anterolateral drawer test, posteromedial drawer</li><li>Repair: Best for avulsion injuries (Stener lesions with MCL retracted prox to pes)<br></br></li><li>Reconstruction: of sMCL and POL</li></ul><div><div> <div> <div><img></img></div> </div></div></div>”
Shoulder Injections
“<ul> <li>Subacromial</li> <ul> <li>Pain relief with subacromial injection doing Neer’s test</li> <li>Ultrasonography – no difference in accuracy between injections into subacromial space with or without u/s </li> </ul> <li>AC</li> <ul> <li>Accuracy 39-67%</li> <li>U/S can help given oblique joint</li> <li>No difference b/t peri-arituclar vs articular injection</li> </ul> <li>GH</li> <ul> <li>Anterior more accurate than posterior</li> </ul> <li>Suprascap nerve</li> <ul> <li>Use U/S to get to suprascapular notch to avoid neurovasc injury</li> </ul> <li>Biceps injection</li> <ul> <li>False positive for inj into groove as fluid can travel into GH joint</li> <li>Inject into sheath not tendon itself</li> </ul></ul>”
Weight Bearing Shoulder: considerations
<ol> <li>Higher prevalence of shoulder pain and rotator cuff pathology in weightbearing shoulders</li> <li>Weight bearing shoulders are stronger than healthy counterparts</li> <li>Improving external rotation strength = strongest factor in reducing shoulder pain</li> <li>Common MRI findings:</li> <ol> <li>Cuff pathology</li> <li>CA ligament thickening / edema</li> <li>AC degeneration</li> </ol> <li>Patients cannot tolerate non-WB after surgery</li> <ol> <li>use double row techniques</li> <li>Leave long head of biceps if possible, otherwise tenotomize</li> </ol> <li>No data on reverse total shoulders in this population, options are total vs hemi</li> <li>Rehab is aggressive</li></ol>
Teres Minor Review
<ol> <li>Tm tears are rare</li> <li>Tm tears are NEVER seen in isolation</li> <li>Most active in higher levels of ER, but contribute minimally if IS/SS intact</li> <li>Most accurate clinical test is ER LAG</li> <ol> <li>Elbow 90, FE 20, max ER, pt must maintain</li> </ol> <li>Important functionally in massive RCT to maintain ER</li> <li>Hypertrophies when IS/SS deficient</li> <li>Integrity affects outcomes in RTSA and Lat dorsi transfer</li> <li>Site of compression QUAD space</li> <ol> <li>Lesions can be vascular or neurologic</li> </ol></ol>
Psych Factors in Shoulder Surgery
“<ol> <li>Post op pain associated with depression</li> <li>Psychological Distress affects outcomes in </li> <ol> <li>Shoulder</li> <ol> <li>TSA: Depression independent risk factor for delirium 2.3x, anemia 1.7x, infection 2.1x, alternative discharge 1.5x.</li> <li>Cuff: high subjective pain tolerance was bigger predictor of outcome in one study</li> </ol> <li>Spine</li> <li>Arthroplasty</li> <li>ACL</li> </ol> <li>Very high rates of depression and new depression in trauma 42-48%</li> <li>Surgical factors less associated with outcome than psyche</li> <li>There are many PROs that can detect depression and that are affected by psyche distress</li> <ol> <li>These are more sensitive than routine clinical evaluation</li> </ol> <li>Depression could affect compensation in USA, despite good surgery because PROs will be one factor</li></ol><div><div> <div> <div><img></img></div> </div></div></div>”
U/S as a tool…
<ul> <li>Longitudinal sinusoidal sounds waves that reflect off of soft tissue</li> <li>Piezoelectric effect</li> <ul> <li>Reflected echoes turned into electric signals</li> </ul> <li>As good as MRI for RCT, not as good for partial tears RCT/Biceps</li> <li>Good for long bone fractures</li> <li>Better in peds elbow fractures that XR</li> <ul> <li>In hands of ED doc</li> </ul> <li>Knee</li> <ul> <li>ACL</li> <ul> <li>88% sens, 98% spec</li> </ul> <li>LCL/MCL = good</li> <li>2x as good as MRI in acute mensical tear </li> <ul> <li>?just detecting it</li> </ul> </ul></ul>
Throwers Shoulder
“<div>Key Points</div> <ul> <li>Injuries during late cocking/acceleration</li> <li>Acceleration: Pect Major, Lat Dorsi, Triceps and Serratus anterior; subscap does very little</li> <li>Deceleration: RC at risk</li> <li>Changes/Adaptations</li> <ul> <li>Late cocking leads to anterior capsule stretch/laxity (AIGHL) –> laxity -> Bankart</li> <li>PIGHL contracts -> GIRD -> risk of injury</li> <li>Increased humeral retroversion</li> </ul> <li>Throwing injuries</li> <ul> <li>(1)Internal impingement </li> <ul> <li>at 90/90 - posterosup RC contacts posterosup labrum - ie pinching of PL cuff</li> <li>PIGHL contracture can lead to GIRD</li> </ul> <li>(2) Internal impingement and anterior instability</li> <ul> <li>Repetitive micro trauma may lead to loose anterior capsule and AIGHL</li> <ul> <li>May lead to symptomatic shoulder instability</li> </ul> <li>Increased anterior translation can lead to anterior labral tearing</li> </ul> <li>(3) Primary anterior or MDI</li> <ul> <li>Extreme fatigue/dead arm </li> <li>As cuff fatigues, subluxation episodes occur</li> <li>Often MRI grossly normal</li> </ul> </ul> <li>Tx: sleeper’s stretch</li> <ul> <li>RC repair</li> </ul><li><div> <div> <div><img></img></div> </div></div></li></ul>”
LCL Injury
<ol> <li>LCL Function</li> <ol> <li>primary resistor to varus</li> <li>Secondary stabilizer to ER in early flexion, some IR throughout ROM</li> <li>Resists anterior translation in concert with ACL</li> </ol> <li>Isolated injuries are very RARE, generally PLC injury</li> <ol> <li>Can be treated non-op</li> </ol> <li>Types 1, 2: non-op with early mobilization</li> <li>Type 3: reconstruction</li> <ol> <li>Anatomic reconstruction most reliable method, lower failure</li> <li>Better knee scores at 2 years</li> <li>Better outcomes in MLKI after recon cf repair</li> <li>Non-op leads to quicker RTS</li> </ol> <li>Recon techniques</li> <ol> <li>Anatomic</li> <li>Isometric: BF tenodesis</li> </ol></ol>
ALL of the knee
<div> <div> <div> <div>Summary</div> <ul> <li>ALL restrains Internal Tibial Rotation</li> <li>Anterolateral complex</li> <ul> <li>ITB: sup + deep + capsulo-osseous layer</li> <li>Anterolateral capsule: coronary ligaments (meniscofem/tib)</li> <li>ALL = refers to either mid-third capsular ligament, capsulosseus layer of the ITB or combination of both</li> </ul> <li>Inv</li> <ul> <li>US sucks</li> <li>MRI: not reproducible</li> </ul> <li>Tx (no outcome data)</li> <ul> <li>Lateral extra-articular tenodesis or ALL reconstruction = decreases internal tibial rotation but can result in overconstraint of internal rotation at knee flexion angles >30 degrees </li> <li>Addition of LET to ACL reconstruction – lowers risk of post-op pivot shift </li> <li>Indications: young, active patients with pathology rotator laxity and imaging suggest of anterolateral complex injury, and in the setting of revision for failed ACL reconstruction with no additional features of failure identified </li> </ul> </ul> <div></div> <div></div> </div> </div></div>
Elbow US
<div>Summary</div>
<ul> <li>Epicondylitis: anechoic/hypoechoic, increased vascularity on dop</li> <li>UCL: see ligament and medial joint space widening as well</li> <li>Biceps: partial tear can be seen</li> <li>Injection possible</li></ul>
Elbow P/E
“<div> <div> <div> <div>Summary</div> <ul> <li>Milking maneuver</li> <ul> <li>positive test: patient experiences a subjective feeling of apprehension and instability along with medial elbow pain</li> <li>SN 87.5%, NPV 100%</li> </ul> <li>Moving valgus stress</li> <ul> <li>Pain between 120-70 deg flexion</li> <li>SN 100%, SP 75%</li> </ul> <li>Valgus Extension Overload Test:</li> <ul> <li>-patient seated, shoulder slightly FF</li> <li>-slight flexion at elbow, examiner applies valgus stress while bringing elbow into flexion; attempting to cause impingement of the medial tip of the olecranon on the medial wall of the olecranon fossa</li> <li>-positive = posteromedial pain</li> <li>-varus may decrease pain</li> </ul> <li>Posterolateral rotatory instability</li> <ul> <li>Lateral pivot shift</li> <li>Chair push up</li> <li>Prone push up</li> <ul> <li>Prone and chair push up more sensitive than lateral pivot shift</li> </ul> <li>Table-top relocation</li> <li>Posterolateral rotatory drawer</li> </ul> </ul> <div>-elbow flexed to 40 degrees, a-p force on lateral aspect of proximal radius and ulna applied</div> <div>-looking for translation of forearm away from humerus on lateral side, pivoting about intact medial side</div> <div>-positive = apprehension, skin dimple</div> <ul> <li>Proximal median nerve entrapment (pronator syndrome)</li> <ul> <li>Loss of pinch, fine motor skills</li> <li>differentiate from carpal tunnel: PS = numbness/paresthesias in palmar cutaneous branch of median nerve and absence of positive provocative maneuvers at the wrist</li> </ul> <li>Radial tunnel/Supinator syndrome</li> <ul> <li>Weakness and decreased sensation in SRN</li> </ul> </ul> <div></div> <div><img></img></div> </div> </div></div>”
Glenoid Rim Reconstruction
<ul> <li>Glenoid and humeral bone lesions are increasingly recognized as key risk factors in recurrent anterior glenohumeral instability - they affect the concavity-compression mechanism</li> <li>Bone defects of 20-30% of glenoid fossa --> decrease GH stability</li> <li>Glenoid on-track: when the humeral head defect is confined within the margins of the glenoid contact area in abd/ER</li> <li>CT best for estimation of defect size</li> <li>Tx: if bone loss>10% and patient RFs: do bony reconstruction</li> <ul> <li>Laterjet: Increased stability from: increased A-P diameter, dynamic sling of conjoint tendon, capsular reinforcement with coracoacromial ligament remnant </li> <li>The congruent- arc Latarjert can improve articular surface restoration </li> <ul> <li>Do not oversize graft - it will just resorb</li> </ul> <li>Iliac crest grafts can be used in arthroscopic and subscapularis-sparing procedures(go through rotator interval)</li> <li>Implant-free techniques, such as the J-bone procedure, have shown accurate graft remodeling and good clinical outcomes </li> <li>Osteochondral allografts (lateral tibial plafond) aim to restore the cartilaginous surface without the morbidity that can result from graft harvesting, but concerns remain regarding availability, contamination, and viability </li> <ul> <li>Do not resorb</li> </ul> </ul></ul>
Periprosthetic tibia fractures
<div> <div> <div> <div>Summary</div> <ul> <li>XR: # pattern, stability, bone stock</li> <li>Aspirate: cell count <1100 not reliable in # setting</li> <li>Tx</li> <ul> <li>Non-op for stable and well aligned, Cast X 6 weeks</li> <li>Locking plates</li> <li>Nail (<9mm diameter)</li> <li>Revision if unstable</li> </ul> </ul> </div> </div></div>
Bearing Surfaces in THA
<div> <div> <div> <div>Key points:</div> <ul> <li>Harder bearings have lower wear rates due to lower surface roughness and less vulnerability to deforming forces</li> <li>Lubrication is useful as it disperses pressure</li> <ul> <li>Lubrication factor: Ceramic on ceramic > metal on metal > hard on soft</li> </ul> <li>Implant factors leading to wear</li> <ul> <li>Position: Vertical cup (edge loading)</li> <li>Material: large CoCr heads (>32) with poly have high wear rates</li> <li>Time: wear rates highest in first 1 million cycles</li> <ul> <li>Osteolysis from polyethylene debris is </li> <ul> <li>0.1mm/year for linear wear</li> <li>80mm3/year for volumetric wear</li> </ul> </ul> </ul> <li>Adverse local tissue reactions</li> <ul> <li>Common with metal on metal bearings but also due to fretting and corrosion at junction of trunion and head</li> <li>RFs for local tissue reaction</li> <ul> <li>Large femoral head</li> <li>Long trunnion length, small diameter</li> <li>Long neck length</li> <li>High taper angle</li> <li>Low rigidity</li> <li>Dissimilar alloys</li> <li>Note: trunnion corrosion is lowest with ceramic heads</li> </ul> </ul> <li>XLP</li> <ul> <li>Undergoes two processes to decrease free radical (+ vitE may help):</li> <ul> <li>High dose radiation in inert gas or vacuum</li> <li>Annealing or re-melting</li> <ul> <li>Melting --> removes all free radicals but affects crystalline structures</li> <li>Annealing --> heat to just below melting point</li> <ul> <li>Removes most free radicals, no effect to overall structure</li> </ul> </ul> </ul> <li>XLP has LOWER WEAR, SMALLER DEBRIS, but is more brittle</li> </ul> <li>All Zirconia ceramic bearings taken off market for late phase transformation </li> <ul> <li>tetragonal to monoclinical transformation</li> </ul> </ul> </div> </div></div>
Young Adult Hip
<div> <div> <ul><li>Patients with structural hip abnormalities may undergo premature hip joint degeneration. However, although the relative risk of joint degeneration in patients with camtype FAI or hip dysplasia is elevated, the absolute risk is still relatively low</li> <li>FAI</li> <ul> <li>CAM lesions lead to an increased risk of developing hip OA over time, but many hips with FAI morphology will never develop OA</li> <li>Pincer: unclear</li> <ul> <li>one study suggesting that acetabular overcoverage may protect against the development of OA</li> </ul> <li>CAM</li> <ul> <li>Modified Dunn View</li> <ul> <li>Alpha angle > 55 deg</li> <li>femoral head/neck ratio < 0.17</li> </ul> <li>AP Pelvis</li> <ul> <li>The triangular index is a measure of the pistol-grip deformity on the AP pelvis radiograph that quantifies the degree in which the lateral cortex of the femoral head/neck junction diverges from the best-fit circle of the femoral head</li> <li>A value of >1 is indicative of cam-type FAI morphology </li> </ul> </ul> <li>Pincer</li> <ul> <li>Can be focal (Anterosup) or global</li> <li>X-rays: </li> <ul> <li>lateral and anterior center edge angles > 40 deg</li> <li>downsloping or negative acetabular index</li> <li>the presence of a crossover sign or acetabular protrusio/profundal or by presence of global acetabular retroversion</li> </ul> </ul> </ul> <li>Acetabular dysplasia</li> <ul> <li>Anterolateral uncoverage leads to transmission of hip forces through smaller surface aread of weight-bearing cartilage/labrum</li> <li>Deformity: coxa valga, anterveted femoral neck, lateralized hip COR</li> <ul> <li>Can lead to hypertrophied labrum and stabilizing muscles</li> </ul> <li>X-ray:</li> <ul> <li>Lateral CEA < 20 – dysplastic</li> <li>Anterior CEA (on false profile view) < 20 deg – dysplastic</li> <li>Tonnis > 10 – abnormal</li> </ul> <li>Strong evidence supports an increased risk of developing hip OA associated with acetabular dysplasia. </li> <li>Although dysplasia is commonly defined by an LCEA of <20°, the relative risk of developing OA extends into the 25°to 30°LCEA range.</li> </ul> <li>SCFE</li> <ul> <li>Cam-type deformity from anterior metaphyseal prominence</li> <li>Severity related to initial slip</li> <li>The alpha angle (or the severity of residual proximal femoral deformity) was strongly correlated with OA development and poor outcomes</li> </ul> <li>Perthes</li> <ul> <li>Coxa magna, vara, plana and secondary acetabular dysplasia/retroversion</li> <li>Severity of deformity correlated with OA</li> </ul> <li>Surgical Interventions</li> <ul> <li>For now, no evidence exists that any of these procedures are preserving the hip joint, but uncontrolled series of patients show improved pain and function at short- to mid-term follow-up. </li> <li>Surgical Indications: symptomatic disease, </li> <ul> <li>no indication for prophylactic surgery in the asymptomatic hip with structural deformity at present</li> </ul> </ul> </ul> </div> </div>
Flexor Tendon Injuries
<ol> <li>Repair strength depends on number of core sutures, more is better, 2 is not enough. </li> </ol>
<ul> <li>Authors use a 4 core cross-locked cruciate, 8 core = least risk of gapping</li> </ul>
<li>Epitendinous repair increases strength 10-50% and reduces gapping</li>
<li>Partial tears <50% can be trimmed and left alone, > 50 % require repair</li>
<li>A2/A4 pulleys can be released 25%</li>
<li>ZONE 1 </li>
<div>Acute injuries (< 6 weeks)</div>
<div>> 1cm stump = primary repair</div>
<div>< 1cm stump = pullout button or anchor</div>
<div>Chronic injuries (> 6 weeks)</div>
<div>Non-op</div>
<div>if become symptomatic = dip arthrodesis</div>
<ol> <li>ZONE 2 </li> </ol>
<div>Repair acutely or plan for 2 stage procedure</div>
<ol> <li>ZONE 3-5 </li> </ol>
<div>Direct repair acutely</div>
<div>Release Carpal Tunnel in Zone 4 injuries</div>
<ol> <li>Get patients flexing fingers early to maximize tendon excursion</li> </ol>
<div>Modified Duran protocol</div>
<div>Place-and-hold exercises. Splint with extension block</div>
<div>Kleinert protocol</div>
Thumb arthritis
<div> <div> <div> <ul><li>Anatomy</li> <ul> <li>Volar oblique (deep anterior) beak ligament becomes taut with abduction, pronation, extension (this is reduction maneuver for bennett fracture)</li> <ul> <li>New studies dispute the theory that beak ligament degeneration contributed to disease progression</li> </ul> <li>Dorsal ligaments are robust, whereas volar ligaments are weak and have minimally regular struction</li> </ul> <li>Pathoanatomy</li> <ul> <li>Thumb metacarpal pulled into adduction with MCP hyperextension</li> <li>Volar-ulnar quadrant of trapezium has greatest compressive load</li> </ul> <li>Epi</li> <ul> <li>6F:1M (ligamentous laxity)</li> <li>Labourers with repetitive thumb use</li> </ul> <li>Dx</li> <ul> <li>Grind test</li> <li>Ix: robert view</li> <li>Classification</li> <ul> <li>Eaton</li> <li>Ladd</li> </ul> </ul> <li>Tx</li> <ul> <li>Non-op</li> <li>Early OA: MC extension osteotomy, debridement, hemitrapeziectomy</li> <li>Late: trapeziectomy with sling (FCR, hematoma, suture)</li> <li>Severe Instability or young: fuse</li> <li>Lrti>fusion for females>40 years</li> </ul> </ul> </div> </div></div>