language Flashcards

(31 cards)

1
Q

Who was Paul Broca and what was his major contribution to language neuroscience

A

Paul Broca was a nineteenth century neurologist who discovered that a small region in the inferior part of the left prefrontal cortex is crucial for speech production. This region is now known as Broca’s area. He proposed that articulatory programs are stored in this area and activate the nearby primary motor cortex to control the muscles of the mouth and face during speech.

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2
Q

What is Broca’s aphasia and what causes it according to the classical model

A

Broca’s aphasia, also called expressive aphasia, is caused by selective damage to Broca’s area. It results in slow, labored, effortful, and poorly articulated speech. Comprehension remains mostly intact. Speech is non fluent and telegraphic.

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3
Q

What is an example of the type of speech produced in Broca’s aphasia

A

A typical example is disjointed output such as “Yes Monday Dad and Dick Wednesday nine o’clock.”

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4
Q

Who was Carl Wernicke and what did he contribute to language neuroscience

A

Carl Wernicke discovered a second major language area in the left temporal lobe, posterior to the primary auditory cortex, located in the left planum temporale. He proposed that this region, now called Wernicke’s area, is the main cortical center for language comprehension.

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5
Q

What is Wernicke’s aphasia and what causes it

A

Wernicke’s aphasia, also called receptive aphasia, is caused by selective damage to Wernicke’s area. It results in severe deficits in comprehension of both spoken and written language. Patients produce fluent but meaningless speech filled with paraphasias and jargon, often termed word salad.

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6
Q

What is an example of speech produced in Wernicke’s aphasia

A

A typical example is “Mother is away here working her work to get her better, but when she’s looking at the two boys looking in the other part.”

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7
Q

What is conduction aphasia and what causes it in the Wernicke Geschwind model

A

Conduction aphasia is caused by damage to the arcuate fasciculus, the fiber pathway connecting Wernicke’s area to Broca’s area. Patients can understand speech and produce fluent spontaneous speech but have major difficulty repeating words or phrases they have just heard.

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8
Q

What did Joseph Jules Dejerine contribute to language research

A

Dejerine studied a patient who could speak and understand spoken language but could not read or write, a syndrome called pure alexia with agraphia. He concluded that the left angular gyrus is responsible for processing language based on visual input.

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9
Q

What is alexia

A

Alexia is the inability to read.

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10
Q

What is agraphia

A

Agraphia is the inability to write.

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11
Q

What is the Wernicke Geschwind model

A

It is a serial processing model of language created by combining the ideas of Broca, Wernicke, and Dejerine, later revived and systematized by Norman Geschwind in the mid nineteen sixties. It proposes seven left hemisphere components specialized for language and predicts the pathways involved in tasks such as reading and speaking.

PAP Worked At BP

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12
Q

What are the seven components of the Wernicke Geschwind model

A

The primary visual cortex, the angular gyrus, the primary auditory cortex, Wernicke’s area, the arcuate fasciculus, Broca’s area, and the primary motor cortex.

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13
Q

According to the model, what is the flow of information during speech comprehension and production while conversing

A

Sound is received in the primary auditory cortex, interpreted for meaning in Wernicke’s area, transferred to Broca’s area through the arcuate fasciculus, translated into articulatory programs in Broca’s area, and executed by the primary motor cortex as spoken output.

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14
Q

According to the model, what is the flow of information during reading aloud

A

Visual input is processed in the primary visual cortex, transformed into an auditory representation in the angular gyrus, interpreted in Wernicke’s area, sent to Broca’s area through the arcuate fasciculus, and converted into speech by the primary motor cortex.

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15
Q

What major prediction of the Wernicke Geschwind model failed empirical testing

A

That discrete cortical areas such as Broca’s area, Wernicke’s area, the arcuate fasciculus, and the angular gyrus each have sharply defined, exclusive language functions. Damage restricted to these areas often produces little or no lasting language impairment.

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16
Q

Why do discrete lesions in Broca’s or Wernicke’s areas often not cause lasting classical aphasias

A

Because language functions are widely distributed across cortical and subcortical areas. Aphasia usually arises from more extensive damage, including white matter pathways, rather than isolated cortical destruction.

17
Q

Why are the pure aphasia syndromes predicted by the Wernicke Geschwind model rarely observed

A

Because expressive and receptive symptoms usually co occur, and real brain lesions rarely match the clean, localized patterns assumed by the model.

18
Q

What evidence shows that aphasia can arise from damage outside the Wernicke Geschwind network

A

Aphasia has been documented following lesions in areas such as the medial frontal lobe, the basal ganglia, and the thalamus, which the classical model did not include.

19
Q

What general trend linking lesion location and aphasia type is supported by the research

A

Large anterior lesions tend to produce more expressive impairments, while large posterior lesions tend to produce more receptive impairments. This is a gradient rather than a strict mapping onto specific areas.

20
Q

What did electrical stimulation studies by Penfield and Ojemann reveal about language organization

A

Disruptions of speech were found across wide areas of frontal, temporal, and parietal cortices. The areas crucial for language varied from person to person, indicating an individual mosaic pattern rather than fixed boundaries.

21
Q

What is the current scientific status of the Wernicke Geschwind model

A

It has been largely abandoned in research because evidence does not support strict localization. However, it remains widely used in clinical and educational contexts as a simplified way to understand aphasia syndromes.

22
Q

Which elements of the Wernicke Geschwind model remain supported today

A

Broca’s and Wernicke’s areas still have important roles in language, and the anterior to posterior gradient for expressive versus receptive deficits is reliable.

23
Q

What is the cognitive neuroscience approach to language and how does it differ from the Wernicke Geschwind model

A

It assumes that language is created by many smaller cognitive processes rather than large general modules. It emphasizes distributed networks, shared functions, and small specialized cortical areas rather than large exclusive ones.

24
Q

What is the first major premise of the cognitive neuroscience approach

A

Language should be studied at the level of constituent processes such as phonological processing, grammatical analysis, and semantic analysis. Complex behaviors are combinations of these smaller components.

25
What does the Wernicke Geschwind model assume about the level of analysis
It assumes large, complex functions like speech production or language comprehension are housed in single cortical centers.
26
What is the second major premise of the cognitive neuroscience approach
Brain regions used for language are not exclusive to language. They also participate in general cognitive processes such as memory, attention, and working memory.
27
What did the Wernicke Geschwind model assume about exclusivity of function
It assumed that large parts of the left cortex are dedicated solely to language.
28
What is the third major premise of the cognitive neuroscience approach
Language areas are small, widely distributed, specialized, and embedded within broader functional networks rather than forming large, homogeneous regions.
29
What did the Wernicke Geschwind model assume about the size and distribution of language areas
It assumed that language areas are large, distinct, and circumscribed.
30
What methodological difference separates the two approaches
The classical model relies mainly on lesion studies in brain damaged patients, while the cognitive neuroscience approach relies heavily on functional brain imaging in healthy individuals along with other modern techniques.
31
Why must neuroimaging results be interpreted with caution when studying language
Neuroimaging shows correlation rather than causation. A brain area may become active during a language task without being essential for it, as demonstrated by the fact that right hemisphere activation appears during language tasks even though right hemisphere damage rarely causes persistent aphasia.