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General Pathology Theory
> Q 65. Hypertrophy. > Flashcards
Q 65. Hypertrophy. Flashcards
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General Pathology Theory
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Decks in class (101)
# Cards
Q1 History of Pathology. History of Pathology in Bulgaria.
10
Q2 Subject, aim and significance of General Pathology.
9
Q3 -Methods of Pathology. Macroscopic examination
9
Q4 Methods of Pathology. Histological examination.
9
Q5 Methods of Pathology. Cytological examination.
9
Q 6. Methods of Pathology. Histochemical examination. Histochemistry of carbohydrates, lipids, proteins and enzymes.
8
Q 7. Methods of Pathology. Immunohistochemistry.
8
Q 8. Electron microscope examination.
7
Q9. Pathology of the damaged cell. Etiology and morphogenesis of the cellular damage.
9
Q10 . Cellular damage in hypoxia, physical and chemical factors.
7
Q11 . Cellular damage due to biological factors: bacteria and viruses.
9
Q 12. Ultrastructural pathology of the damaged cell. Ultrastructural pathology of the cell nucleus.
9
Q13. Ultrastructural pathology of the damaged cell. Ultrastructural pathology of the cell cytoplasm.
9
Q 14. Disorder of the fluid and electrolyte balance of the damaged cell: cellular swelling, vacuolar and hydropic degeneration.
9
Q 15. Excessive accumulation of proteins in the cell
9
Q 16. Excessive accumulation of lipids in the cell: adipose degeneration.
9
17. Excessive accumulation of lipids in mesenchymal cells: mesenchymal lipidoses.
9
Q18. Accumulation of pigments. Exogenous pigments.
9
19. Accumulation of pigments. Endogenous pigments. Haemoglobinogenic pigments: haemosiderin, ferritin, haematin.
9
20. Accumulation of pigments. Iron-free haemoglobinogenic pigments: bilirubin. Types of jaundice.
9
Q21 . Accumulation of pigments. Endogenous pigments. Autochthonous pigments: proteinogenic and lipidogenic pigments.
9
Q 22. Disorders of collagen synthesis.
10
Q23. Disorders of elastin synthesis.
7
24. Excessive accumulation of material in the intercellular substance of the connective tissue: fibrinoid accumulation.
3
Q 25. Excessive accumulation of material in the intercellular substance of the connective tissue: hyaline.
3
Q 26. Accumulation of amyloid /amyloidosis/.
9
Q 27. Disorders of calcium and copper metabolism
9
Q 28. Death – types, phases, morphological features. Signs of death.
10
Q 29. Necrosis – causes, types.
7
Q 30. Necrosis. Clinico-anatomical forms. Outcome of necrosis.
7
Q 31. Apoptosis
9
Q32. Left-sided heart failure.
8
Q 33. Right-sided heart failure.
7
Q 34. Disorders of the microcirculation.
0
Q 35. Shock. Clinico-morphological forms.
0
Q 36. Organ changes in shock.
0
Q 37. Local disorders in blood circulation. Arterial hyperaemia.
0
Q 38. Local disorders in blood circulation. Venous hyperaemia
0
Q 39. Ischemia – types.
0
Q 40. Infarctions – types.
0
Q 41. Haemorrhage – mechanisms. Haemorrhagic diathesis. Coagulopathies.
0
Q 42. Thrombosis. Conditions for thrombus formation. Types of thrombi.
0
Q 43. Thrombosis. Clinico-anatomical forms. Outcome.
0
Q 44. Intravascular coagulation. Clinico-anatomical fo
0
Q 45. Embolism. Types. Embolus pathways
0
Q 46. Thromboembolism
0
Q 47. Fat embolism. Bone marrow embolism.
0
48. Air, gas, tissue embolism. Microbial embolism and other rare types.
0
Q 49. Disorders of fluid exchange. Oedema. Types.
0
Q 50. Clinico-anatomical forms of oedemas. Hyperhydration and dehydration
0
Q51 Inflammation. Etiology and phases of development of the inflammatory reaction.
15
Q52 Plasma and tissue mediators of inflammation.
16
Q53 Inflammation: forms. Acute inflammation. Functional changes at the site of inflammation. Vascular changes. Types of exudate.
16
Q54 Cellular elements in acute inflammation.
13
Q55 -Morphological forms of acute inflammation: serous and fibrinous inflammation.
17
Q56 - Morphological forms of acute inflammation: purulent, haemorrhagic and gangrenous inflammation. Outcomes of acute inflammation.
15
Q57.Chronic inflammation. Morphological characteristics. Cellular elements in chronic inflammation. Forms. Outcome.
16
Q58 - Non-specific granulomatous inflammation. Types. Polypous inflammation.
15
Q59 - Specific granulomatous inflammation.
15
Q60 - Allergic reactions. Types.
0
Q61. Autoimmune diseases. Types.
0
Q 62. Immunodeficiency syndromes. Types. Morphological characteristics.
0
Q 63. Transplant immunity.
0
Q 64. Hyperplasia.
0
Q 65. Hypertrophy.
0
Q66. Atrophy.
0
Q 67. Reorganization of tissue and organ structures. Prosoplasia. Anaplasia. Metaplasia.
0
Q 68. Regeneration. Complete and incomplete regeneration.
0
Q 69. Factors affecting the growth processes.
0
Q 70. Regeneration of skin and mucous membranes.
0
Q71. Regeneration in cardiovascular system, liver.
0
Q72 Regeneration in kidneys, lungs, haemopoietic tissue, lymph nodes and muscles.
1
Q 73. Regeneration in nervous system, cartilage and bone tissue.
0
Q74 74. Tumors. Biological, morphological, biochemical and functional characteristics.
0
Q 75. Etiology and epidemiology of tumors.
0
Q 76. Precancerous conditions. Types. Significance.
0
Q 77. Morphogenesis of tumor growth.
0
Q 78. Biological manifestations of tumors – benign and malignant.
0
Q 79. Recurrence and metastasis. Types of metastases.
0
Q 80. Cellular biology of tumors. Grades and stages of tumor growth.
0
Q 81. Tumor-host interactions.
0
Q82. General morphology of tumors. Macroscopic and microscopic characteristics.
0
Q 83. Immunohistochemical and molecular-pathological diagnostics of tumors.
0
Q 84. Terminology and classification of tumors.
0
Q 85. Benign epithelial tumors.
0
Q 86. Malignant epithelial tumors.
0
Q 87. Benign and malignant tumors of muscle tissue.
0
Q 88. Benign and malignant tumors of connective tissue.
0
Q89. Benign and malignant tumors of adipose tissue.
0
Q 90. Benign and malignant tumors of blood vessels.
0
Q 91. Benign and malignant tumors of bone tissue.
0
Q 92. Benign and malignant tumors of cartilage tissue.
0
Q 93. Melanin-producing tumors.
0
Q 94. Tumors of lymph nodes and bone marrow.
0
Q 95. Teratomas.
0
Q96. Tumors of the nervous system.
0
Q 97. Fetal malformations. Etiology and morphogenesis of congenital malformations.
0
Q 98. Types of malformations in relation to the period of teratogenic exposure. Names and classification of congenital malformations.
0
Q 99. Congenital malformations of the foetus (most common and affecting the cardiovascular, nervous, gastrointestinal and urinary systems).
0