Unit 3 Flashcards

(64 cards)

1
Q

Transduction

A

The process in the retina’s photoreceptor cells that converts light into electrical signals

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2
Q

Law of Specific Nerve Energies

A

Nerves are specific to their senses

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3
Q

Coding

A

How the brain translates light signals into meaningful visual information for perception and memory. Aspects: which neurons are active, & how many action potentials a neurons sends per unit of time

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4
Q

Identify the visual receptors. What is the collective name for the receptors and what are the individual names?

A

Photoreceptors: rods (faint light, periphery of eye), & cones (color, fovea)

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5
Q

How does light stimulate receptors?

A

Strikes the 11-cis-retinal (the protein complex that activates when hit by light waves) and causes transduction

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6
Q

Anatomy of the eye

A

Iris & pupil (light enters; pupil can constrict and dilate), cornea & lens (light focused onto the retina), retina (lined w/receptors), fovea (center of the macula), macula (contains cones; sharp details, recognize colors, and read)

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7
Q

What is the pathway and direction of impulses after the receptors are stimulated?

A

Bipolar cells - ganglion cells - optic nerve - brain

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8
Q

Extra cells

A

Amacrine cells get info from bipolar cells and send it to other bipolar, amacrine, and ganglion cells; horizontal cells make inhibitory contact onto bipolar cells, which in turn make synapses onto amacrine and ganglion cells

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9
Q

What does the optic nerve consist of and what causes the blind spot?

A

Consists of the axons of the ganglion cells that travel to the brain; has no receptors as it is the point at which the optic nerve leaves the back of the eye

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10
Q

Describe receptive fields and state how the receptive field combines when multiple receptors are funneled to one ganglion cell.

A

The point in space from which light strikes it; small group of rods or cones connect to a bipolar cell, several of those connect to a ganglion cell (receptive field becomes larger, and include both excitation and inhibition)

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11
Q

What is the adaptive purpose of rod cell neural convergence?

A

It maximizes visual sensitivity in low-light conditions; the summation enables perception of fainter lights in the periphery

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12
Q

What does the fovea contain and what is the purpose? What cells are in the peripheral aspect of the retina?

A

Filled with cones which allows for acute & detailed vision, and allows for the registering of the exact location of input; rods which respond to faint light

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13
Q

Compare and contrast foveal and peripheral vision.

A

Foveal: cones, each ganglion cell excited by a single cone, distinguishes among bright lights & responds poorly to dim, good detail vision and good color vision
Peripheral: rods increase, each ganglion cell excited by many receptors, responds well to dim light, poor detail vision and color vision

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14
Q

What is the range of wavelengths that humans can perceive? What does this have to do with color vision?

A

380nm - 750 nm; they are called light because the receptors in our eyes are tuned to detecting them

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15
Q

What is the trichromatic theory of color vision? What does this have to do with cones?

A

Relative rates of response by 3 kinds of cones (i.e., long, medium, & short) - each is most sensitive to a diff set of wavelengths & the ratio of activity determines color

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16
Q

Why have other theories been proposed, and what was proposed to explain the negative color after image?

A

Opponent process theory: perceive color in terms of opposites (brain has a mechanism that perceives color on a continuum from red to green, yellow to blue, and white to black) - an increase in response produces one perception, and a decrease produces a different perception

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17
Q

State the most common type of color vision deficiency. What is the genetic link?

A

Red-green color deficiency; is on the X chromosome (either the lack of a type of cone, or a cone w/abnormal properties)

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18
Q

Trace the pathway for vision from the optic chiasm to the lateral geniculate nucleus and then to primary visual cortex. What happens to the visual field information at the optic chiasm? What parts of the visual
field does each eye see?

A

Information from the nasal half crosses to the contralateral hemisphere, info from the temporal half goes to the ipsilateral hemisphere - percentage of crossover varies depending on the location of the eyes

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19
Q

What happens to visual info after going to V1?

A

1st processing (occipital lobe) - as imagining seeing something, activity increases in area V1 in a pattern similar to actually seeing object; V1 to V2 to other areas

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20
Q

Define and describe blindsight.

A

Ability to respond in limited ways to visual information without perceiving it consciously - no awareness of visual input (could be that small island of healthy tissue remain, or thalamus sends visual input to several other brain areas besides V1, including parts of the temporal cortex)

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21
Q

What functions are the ventral and dorsal streams associated with? What parts of the brain are these streams associated with?

A

Ventral: through the temporal cortex, “what” pathway - specialized for identifying and recognizing objects
Dorsal: through the parietal cortex, “where” pathway - helps the motor system locate objects

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22
Q

Why is V1 called striate cortex?

A

Because of its striped appearance, line of Gennari (thick band of myelinated axons)

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23
Q

Define visual agnosia. What is prosopagnosia and what part of the brain is it related to?

A

Visual agnosia is the inability to recognize objects and is caused by damage to the pattern pathway usually in temporal cortex - V1 is okay; prosopagnosia is the inability to recognize faces and occurs after damage to the right fusiform gyro of the inferior temporal cortex

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24
Q

What is motion blindness and what area of the brain is it related to?

A

Refers to the inability to determine the movement of objects (which direction & how fast); area MT (middle temporal cortex) & MST (medial superior temporal cortex)

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25
What is some evidence for some genetic coding of facial recognition ability?
Newborns who showed a strong preference for a right-side-up face over an upside-down face, regardless of whether the face was realistic or distorted - the face only requires the eyes to be on top but does not have to be realistic
26
Explain how experience with the physical world shapes the development of vision
Brain connections; learn about the world by observing how objects move and interact in their environment; depth perception, hand-eye coordination, object & face recognition
27
What happens to cortical connections when there is the lack of stimulation (i.e., vision is blocked) in one eye versus two during early development?
One eye: causes synapses in visual cortex to become unresponsive to input from that eye - synapses from open eye inhibit those of closed eye Both: cortical responses become sluggish & lose their well-defined receptive fields but does not cause blindness - visual ctx. starts responding to auditory & touch stimuli
28
Explain how critical periods are related to neuronal sprouting. What does this have to do with the early development and removal of cataracts (2 to 6 months) versus when they are removed as an adult?
The sensitive period ends with the onset of certain chemicals that stabilize synapses and inhibit axonal sprouting (cortical plasticity is greatest in early life, but never ends); during infancy, causes visual deprivation but in adulthood, can limit future vision (difficulty recognizing objects)
29
How does retinopathy relate to cognitive changes over time?
Eyesight can be clue to mental decline - decreases in blood supply may be damaging eyes and brain concurrently
30
What physical stimulus is responsible for hearing? How is this stimulus characterized?
Sound waves (periodic compressions of air); characterized by amplitude (height/intensity) & frequency (# of compressions per second)
31
Define the role of the following parts: tympanic membrane, ossicles (- state the Latin names), oval window, cochlea, basilar membrane, tectorial membrane, and hair cells
Tympanic membrane (eardrum): vibrates at the same frequency as the sound waves Ossicles (malleus, incus, & stapes): connects to the oval window Oval window: amplifies the waves Cochlea: contains 3 long fluid filled tunnels (Scala vestibule, Scala media, & scala tympani) Basilar membrane: translate sound vibrations into electrical signals by acting as a frequency analyzer Tectorial membrane: stimulate hair cells in the inner ear to enable hearing Hair cells: responds to displacement from cochlea fluid, thereby opening ion channels in its membrane
32
What causes transduction in the auditory system?
The hair cells that are being displaced
33
What are the frequencies that adults lose the ability to sense over time?
High frequency sounds, primarily losing sensitivity in the 2,000 Hz to 8,000 Hz range, hear sounds ranging from about 15 Hz to 20,000 Hz
34
State the pathway for sound stimuli after leaving the inner ear. Know brain stem nuclei (in general) to medial geniculate thalamus to superior temporal cortex (A1)
Auditory nerve - brain stem - medial geniculate - superior temporal cortex (A1)
35
What & Where Processing Pathways
What: patterns of sound, anterior temporal cortex Where: sound location, posterior temporal cortex & parietal cortex
36
Place Theory
Hair cells at a location are sensitive to one frequency of sound wave
37
Frequency Theory
Basilar membrane vibrates in synchrony (APs at the same frequency)
38
What is a tonotopic map and what does this have to do with sound and brain damage?
Primary auditory cortex cells are responsive to particular tones; when damaged, the map can reorganize leading to issues like tinnitus or distorted sound perception as neighboring neurons take over the job of the lost frequency (plasticity)
39
Why did I emphasize that nociception is adaptive?
It is an essential survival mechanism that alerts an organism to potential or actual tissue damage, thereby promoting behaviors to avoid injury
40
What are the two types of nocicceptors?
A-delta-myelinated (initial sharp pain), c-fibers (slow dull ache)
41
What is the relationship between the processing of physical and emotional pain in the brain?
Somatosensory cortex responds to painful stimuli, memories of pain, & signals that warn of impending pain; structures involved in emotional associations (hypothalamus, thalamus, amygdala, hippocampus, prefrontal cortex, cingulate cortex)
42
What is the periaqueductal grey area and what does it have to do with the proposed gate theory of pain?
Area of midbrain whose receptors opiates bind to; input from brain & skin can close the "gates" and decrease nociception
43
What do endorphins and Substance P have to do with decreasing the quantity of pain perceived in the brain?
Substance P increases intensity of pain; endorphins block pain signals particularly by inhibiting the release of Substance P
44
Where do the pain signals come from and what is interesting about the theory that the periaquductal (PAG) area is a pseudo-gate?
It comes from a bare, specialized, nerve ending; it's a key midbrain hub that can descend to the spinal cord to either block or amplify pain signals before they reach the brain
45
How is the blocking of pain signals adaptive under some circumstances?
It allows the body to prioritize immediate survival in extreme situations by overriding pain's warning, enabling focus on escape or action
46
Explain what placebos have to do with the cingulate cortex and somatosensory cortex.
Can decrease the emotional response but not the sensation (decreased response in the cingulate gyrus but not in somatosensory cortex)
47
Why is gustation classified as a chemical sensation?
It relies on specialized taste receptors that directly bind with chemical molecules dissolved in saliva, triggering neural signals to the brain, rather than responding to physical stimuli like light or pressure
48
What is the significance of the story "The Man Who Couldn't Eat?"
Chronic illness strips away identity, relationships, and joy by turning food into a toxic enemy
49
How many different tastes do we have?
Sweet, sour, salty, bitter, & umami
50
See “chemicals that alter the taste buds” in your book.
Miracle berry - temporarily changes sweet receptors, changing from acidic to sweet Sodium laurel sulfate - intensifies bitter tastes & weakens sweet ones by coating the sweet receptors Gymnema slyvestre - sugar becomes tasteless
51
What are papillae and where do you find them? What do they have to do with gustation?
It contains taste buds (10+) that are the receptor cells
52
What is special about bitter taste?
Sensitive to a wide range of chemicals w/varying degrees of toxicity; maybe biologically hardwired to distrust/dislike bitter things b/c most associated with toxicity
53
What did you learn about the neuropathways of taste?
Brain stem - 1. hypothalamus & amygdala, 2. thalamus, 3. insula (primary cortex) (other cortical areas); ipsilateral hemispheric representation
54
Where are the olfactory receptors located?
They line the epithelium in the rear of the nasal area
55
Provide the name for primary olfactory cortex. Where is it located?
Entorhinal cortex; anterior-medial temporal lobe and parts of the frontal lobe
56
What is the name for loss of smell? Why are the olfactory bulbs prone to damage?
Anosmia; they are exposed to air, average survival time of just over a month
57
What is the difference between pheromones and olfaction?
VNO (vomeronasal organ) receptors are specialized to respond only to pheromones (chemicals released by an animal that affect the behavior of other members of the same species)
58
Be able to explain why olfaction is adaptive. What is conditioned taste aversion?
Important for food choices - much of taste (flavor) is dependent on odor; organism learns to avoid a specific food after a single experience of becoming ill shortly after consuming it
59
Why do smells seem to trigger intense memories?
The brain forms strong memories and the amygdala triggers an emotional reaction (PS nervous system)
60
What is the organ that non-human mammals use to sense pheromones? What do we know about the organ in humans?
Vomeronasal organ, a set of receptors located near the olfactory receptors that are sensitive to pheromones; it is tiny and has no receptors, it is vestigial
61
Is there evidence for human chemosensation and emotional activation?
Smell of a woman's sweat increases a man's testosterone secretions, & male sweat increase release of cortisol; timing of women's menstrual cycles - women exposed to secretions became synchronized to the donor's menstrual cycle
62
Where do the senses of smell and taste combine in the brain?
In the orbitofrontal cortex
63
What did you learn about olfactory bulbs and stem cells?
Adult neural stem cells in the nose constantly generate new smell-sensing neurons that project to the olfactory bulb in the brain
64
Define synesthesia and state the common theory that explains why some people have this ability.
The experience of one sense in response to stimulation of a different sense; some of the axons from one cortical area branch into another cortical area