week 3 Flashcards

(52 cards)

1
Q

What is mood?

A

A pervasive and sustained emotional response

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2
Q

what are mood disorders?

A

are defined by mood episodes in which a person’s behavior is dominated by “the ups” or “the downs”

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3
Q

What is the spectrum of the downs?

A
  • Euthymia (normal mood)
  • Dyshoria/dysthymia (Experience of unpleasant (usually low) mood)
  • depression (pervasive and sustained low mood & related behaviours & symptoms)
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4
Q

What is the spectrum of the ups?

A
  • Euthymia (normal mood)
  • Euphoria (Intense feeling of well-being, excitement, over-
    confidence & over optimism)
  • hypomania (increased energy but less severe features than
    mania)
  • Mania (Elevated mood, inflated self-esteem & associated
    symptoms)
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5
Q

How do mood disorders affect a person?

A

Emotionally
* can cause Dysphoria or Euphoria (and associated mood extremes)

Cognitively
* Abilities – e.g., disturbed concentration
* Appraisals – e.g., grandiosity & inflated self-esteem OR depressive /‘negative’ triad: hopeless view of self, environment, futur

Somatic
* Fatigue/energy level, pain threshold, appetite, sleep,

Behavioural/Affective
e.g., Psychomotor slowing versus agitation; limited behaviour vs ceaseless activity

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6
Q

Distinguishing depression & ‘normal’ sadness

A

Mood change is persistent and pervasive, not lifting even during enjoyable activities.

It may occur without a clear trigger or seem out of proportion to events.

The mood impairs daily functioning, affecting work, social life, or routine roles.

It’s accompanied by additional symptoms (cognitive, physical, or behavioural), not just sadness alone.

The quality of the mood differs from ordinary sadness — deeper and more enduring.

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7
Q

How was mood disorders conceptualised in the DSM-4 compared to the DSM-5

A

DSM-4
- The DSM dealt with depressive and bipolar disorders in one chapter called the mood disorders chapter

DSM-5
- In the DSM -5 the conditions were split up into deperate chapters. One focuses on depressive disorders and another for bipolar and related disorders

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8
Q

What is the common features of depressive disorder?

A
  • Presence of “sad, empty or irritable mood, accompanied by somatic and cognitive changes”
  • What differs is their duration, timing, severity, and presumed aetiology
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9
Q

What is Major Depressive Episode/Disorder (MDE, MDD)

A

1 major episode in the absence of any history of manic episodes

A. An episode is ≥5 of 9 symptoms indicative of change over 2-week period; including either (1): depressed mood, or (2): loss of interest or pleasure.

PLUS

B. Cause significant distress or impairment
C. Not attributable to other disorders/substances

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10
Q

Criteria for Major Depressive Episode/Disorder

A

Depressed mood most of the day, nearly every day.

Loss of interest or pleasure in almost all activities.

Significant weight or appetite change.

Sleep problems — insomnia or oversleeping.

Noticeable restlessness or slowed movement.

Fatigue or low energy.

Feelings of worthlessness or guilt.

Poor concentration or indecisiveness.

Recurrent thoughts of death or suicide

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11
Q

MDE/MDD and Bereavement (grief)

A

DSM-IV: Excluded depression within 2 months of a loved one’s death (no MDE/MDD diagnosis).

DSM-5: Bereavement can cause deep grief but usually isn’t MDD. If both occur, symptoms may be more severe, recovery slower, and antidepressants may help those with underlying vulnerabilities. –> believed that people should be able to be diagnosed and that depression can develop after loosing a loved one

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12
Q

What is Persistent Depressive Disorder [PDD] (“Dysthymia”)

A

A more chronic, mild presentation

  • Over a period of >2 years, exhibit a depressed mood for most of the day, more days than not
  • ≥2 symptoms of 6 symptoms
  • Poor appetite or overeating
  • Insomnia/hypersomnia
  • Low energy or fatigue
  • Low self-esteem
  • Poor concentration/decision making
  • Feelings of hopelessness
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13
Q

We would NOT diagnose PDD if:

A
  • Symptoms absent for more than 2 months at a time during
    2-year period
  • If at any time during first 2 years meets criteria for MDE/MDD, then given MDD diagnosis
  • Presence of manic episode
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14
Q

What are some other depressive disorder?

A

Disruptive Mood Dysregulation Disorder
* Children 6-18 yo
* Chronic, severe, persistent irritability and frequent episodes of extremely out-of-control behaviour

Premenstrual Dysphoric Disorder (PMDD)
* Moved from DSM IV ‘further study criteria’ to a recognised condition in the DSM 5
* Severe form of PMS, characterized by mood lability, irritability, dysphoria, anxiety, difficulty concentrating, changes in appetite and sleep, pain, etc

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15
Q

What was Allen francizes most notable criticism in the DSM-5

A
  • he disagreed with adding ‘Disruptive Mood Dysregulation Disorder” into the DSM. He said ‘ DSM 5 will turn
    temper tantrums into a mental disorder, which could be bad for the child long-term.
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16
Q

Whatwas Allen francizes second most notable criticism in the DSM-5

A

Criticised taking out the bereavement clause in the DSM5

He believed that Normal grief will become Major Depressive Disorder,

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17
Q

What is the hallmark or primary feature of Bipolar Disorder

A

Primary impairment involving mood but with
a manic/hypomanic component

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18
Q

What are the three types of Bipolar?

A

Bipolar I [Depressive episode +]
* At least one manic episode

Bipolar II [Depressive episode +]
* Hypomania: episodes of increased energy, not severe enough to qualify manic episodes
* Severity and duration

Cyclothymia
* Chronic, but less severe form of bipolar
* Symptoms of mania and depression rather than ‘episodes’

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19
Q

What is a manic epsiode?

A

A. At least a week of (or any period of time if hospitalisation due to) abnormally & persistently elevated, expansive, or irritable mood and persistently increased goal-directed activity/energy: feature is present for most of the day, nearly every day of this period

B. 3 or more of the 7 symptoms (≥ 4 if mood is irritable)

C. Sufficiently severe to cause marked impairment in functioning, OR to necessitate hospitalisation, OR with psychotic features

D. Not attributable to effects of a substance or due to another medical condition

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20
Q

What are the manic episode criteria?

A

3 or more of these symptoms (or 4 if mood is ‘irritable’):

  1. Inflated self-esteem or grandiosity
  2. Decreased need for sleep
  3. Increased talkativeness or pressure of speech
  4. Flight of ideas or racing thoughts
  5. Distractibility
  6. Increased goal-directed activity / psychomotor agitation
  7. Excessive involvement in activities with high potential for painful consequences
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21
Q

What is a Hypomanic Episode

A

Same symptoms as manic episode (3 or more) except:
* Lasts at least 4 consecutive days
* Unequivocal change in function but not severe enough to cause ‘marked impairment’ in function or trigger hospitalisation

22
Q

What is Cyclothymic Disorder

A
  • Chronic, fluctuating mood disturbance for 2 years or more

*Numerous periods of hypomanic & depressive symptoms (not enough to meet criteria for episodes)

  • Never symptom free for more than 2 months
  • Onset usually in adolescence or early adulthood
23
Q

What is a specifier?

A

When diagnosing bipolar disorder, clinicians can add specifiers to describe additional features or patterns present in the condition. These specifiers provide more detail about the specific characteristics of the disorder.

24
Q

What are common specifiers in Bipolar

A

With anxious distress

With mixed features

With melancholic features

With atypical features

With mood-congruent psychotic features

With mood-incongruent psychotic features

With catatonic features

With peripartum onset

With seasonal pattern

With rapid cycling (Bipolar I or II)

25
What is Reification
Reification means treating a mental disorder label as if it were a real, separate thing that exists on its own — instead of remembering that it’s just a description of symptoms. so if someone has depression - its not that they have depression, instead they have multiple symptoms that have been grouped and given the name depression
26
How prevalent are depressive disorders?
* One of the most common forms or psychopathology * Meanage of onset = 32 years (Kessler et al., 2007) * Comorbidities: anxiety, substance abuse (and ‘physical’ illness)
27
What is the course & outcome of depression?
Also one of the most treatable, although episodes can re-occur * 50% recover within 6 months of beginning of an episode * Typically, 5 to 6 lifetime episodes * Risk of recurrence * 1 episode = 50% * 2 episodes = 70% * 3 episodes = 90%
28
Course and outcome of Bipolar
Manic episode lasts ~2–3 months Course is unpredictable - 90% with one manic episode → have further episodes More lifetime episodes than depressive disorders Earlier onset (18–22 years) High comorbidity (esp. substance use; WHO 2024) Mixed long-term prognosis → often lifelong meds Gender: roughly equal overall Women have increased risk for Bipolar II, hypomania, rapid cycling, mixed episodes (Diflorio & Jones, 2010)
29
Prevalence of both depressive disorder and Bipolar disorder
According to the ABS, 43% had a lifetime mental disorder 21% had a 12-month mental disorder * 17 anxiety disorders (most common) * 7.5% “affective” [mood] disorders * 4.9% Depressive Episode * 1.5% Dysthymia * 2.0% Bipolar I / II
30
How do these disorders impact Australia
* $12.6 billion in costs per year * 6 million working days of lost productivity * Accounts for approximately 10% of all disability * 80% of suicides preceded by a mood disorder
31
Potential life factors causing depressive disorder
* Negative /stressful life events: * Interpersonal loss * Loss of “social roles” * Feelings of entrapment, humiliation, defeat * Stress generation: increase prevalence of stressful life events For specific groups, such as Indigenous peoples, there may be additional significant social influences such as dispossession, cultural genocide, displacement, segregation, “stolen generation.”
32
What are some cognative factor that may cause depressive disorder?
Cognitive vulnerability Maladaptive schemas Causal attributions Response styles Goal attainment
33
What are Maladaptive schemas (factors that increase risk for depression)
1. Assign global, personal meaning to failures 2. Overgeneralise conclusions about self from events 3. Drawing arbitrary inferences about self without supporting evidence 4. Selective recall of events with consequences
34
What are 'causal attributions' (factors that increase risk for depression)
Causal attributions are the explanations people give for why things happen, especially for their own successes and failure * Depressogenic attribution style: internal, stable, global
35
What are 'response style' (factors that increase risk for depression)
How do people respond to thing/life events? * Ruminative style - tend to revisit issues, can't let go of situations, which feeds a sense of hopelessness * Distracting style
36
What are 'goal attainment' (factors that increase risk for depression)
Do people feel like they are moving toward their goals or away from them * Generalized goals * Pessimistic attitude
37
What is the behavioural factors that may increase risk of depression?
* Get depressed when we have Less positive reinforcement (verbal, social) * Get depressed if we have More negative events * Reduced behaviour -> when we get negative responses from others we may reduce our behaviour by taking fewer risks, socialising less often etc.
38
What are some psychological factors that may increase risk of bipolar?
Precipitating factors (things that may trigger onset in a vulnerable person) * Schedule-disrupting events (changes in routine) * Goal attainment events Social factors can influence recovery/relapse * Emotional climate within families * Social support
39
What are some Biological factors that may influence risk of depression and Bipolar
* Genetic contribution especially influential in bipolar 1 disorder * Genetic risk & ‘sensitivity’ to stressful events * Genetic relations are complex * HPA axis (stress system): Abnormal cortisol response; “fight or flight” system may function differently. * Neurotransmitters: Serotonin, norepinephrine, and dopamine involved; complex interactions. * Sleep & biological rhythms: Often disrupted. * Hormones: Possible influence, but evidence is mixed
40
How does a Psychological approach treat depression?
* Cognitive therapy * Interpersonal therapy * Cognitive-Behavioural Therapy / Behavioural therapy: e.g., new response / altered contingencies / pleasant event scheduling
41
How does a social approach treat depression?
* e.g., meaning making, life roles
42
How does a biological approach treat depression?
* antidepressants (& non-medication e.g., TDCS/TMS, ECT, bright light therapy). Types of drugs: * Selective Serotonin Reuptake Inhibitors * Tricyclic antidepressants (TCAs) * Mono-amine oxidase inhibitors (MAOI)s
43
Is treatment of depression effective?
For moderate depression, combining medication and therapy gives the best results and keeps people most engaged in treatment.
44
How common is suicide?
Each year a million people suicide worldwide [more people die each year by suicide than by all other forms of violence combined] * A history of psychopathology (many types, but especially depression) is found in about 90-95% of people who die by suicide
45
Suicide and age
Older Australians have fewer suicides overall, but a higher suicide rate because their age group makes up a smaller share of the total population.
46
Suicide for Indigenous people
* Suicide rate for young Aboriginal and Torres Strait Islander men is about 40% higher than that for the young men in the general population
47
What are some of the risks that may cause suicide
Mental disorders – esp. depression & substance dependence Depression = strongest risk factor (≈19% lifetime suicide risk in mood disorders) Recent stressors: illness, loss, relationship breakdown, isolation History: previous attempts or expressing intent Psychological factors: feeling like a burden + lack of belonging (Joiner’s model
48
Suicide Prevention – Key Levels of Intervention
Population level: - gun control to reduce access to lethal means. Individual (emergency): - Crisis support (helplines, hospitalization, removing access to means), even involuntary treatment if needed. Individual (non-emergency): - Psychotherapy: challenge suicidal thinking, address causes, and strengthen protective factors (e.g., social support, belonging). - Use medication (e.g., antidepressants) and treat comorbidities such as substance use. - Ongoing monitoring and risk assessments are essential.
49
What are the suicide warning signs
*Threatening to self-harm of suicide * Seeking access to means of suicide * Talking or writing about death, dying or suicide * Hopelessness * Rage, anger, revenge * Recklessness * Feeling trapped * Increasing substance use * Social withdrawal * Anxiety, agitation, sleep disturbance * Dramatic mood changes * Feeling purposeless in life/no reason for living
50
Suicide prevention for older adults
Build resilience for loss & life changes Support carers and those facing bereavement Encourage retirement planning Strengthen social connections with peers & community Promote positive ageing through public awareness Reduce risk factors: Limit isolation & living alone Review transport/living needs Remove access to means of suicide Encourage healthy activities (e.g., walking groups) Community programs: improve wellbeing (e.g., raise pensions to reduce poverty)
51
Suicide intervention in younger adults
Traditional school suicide education can be unhelpful - May increase anxiety or depression - Can make suicide seem more common than it is Focus instead on: - Emotional well-being & resilience - Self-esteem, coping, and life skills - Supportive environments (school & home) - Identify at-risk youth early and provide targeted interventions
52