1
Q
case studies that we’ve seen IVIg in (4)
A
XLA
Asplenia
DiGeorge syndrome
AID deficiency
2
Q
what is AID activated by
A
CD40-CD40L signalling
3
Q
AID deficiency is part of the __________ syndrome family
A
hyper-IgM
4
Q
what is a hyper IgM syndrome
(2)
A
too much IgM
absence of class switched antibodies
5
Q
what can express AID
A
immature B cells
6
Q
whats an alternative methode to increase AID (2)
A
stimulation of TLR9 with CpG
7
Q
TLR9 / BCR (IgM) signalling can induce what
A
AID
BMIS 321
flashcards
Decks in class (23)
# Cards
-
Lecture 1 Cells of the immune system and their functions57
-
Lecture 2 Molecular mediators of innate immunity34
-
lecture 3 - chronic granulomatous disease32
-
lecture 4 - innate immune cell sensing and surveillance67
-
lecture 5 - innate immune sensitivity and surveillance 227
-
lecture 6 - activation of innate immunity and inflammation41
-
lecture 7 - hereditary periodic fever syndromes19
-
lecture 8 - steady state immune cell circulation43
-
lecture 9 - immune cell recruitment and trafficking12
-
lecture 11 - post midterm55
-
lecture 1236
-
lecture 13 - hematopoietic stem cells and tissues of the immune system72
-
lecture 14 - Asplenia30
-
lecture 15 - VDJ46
-
lecture 16 - T cell development43
-
lecture 17 - DiGeorge24
-
lecture 18 - 9a T cell activation58
-
lecture 9b19
-
lecture 20- B cell development25
-
X-linkes Agammaglobulinemia13
-
B cell activation46
-
B cell differentiation70
-
AID difficiency7
