what cause burns
Thermal, chemical, electrical, and radioactive agents
results in
cellular destruction of the skin layers, and underlying tissue.
- destruction of body tissue, a burn injury results in the loss of temperature regulation, sweat and sebaceous gland function, and sensory function.
- Metabolism increases to maintain body heat as a result of burn injury and tissue damage.
dry heat injuries
result from open flames and explosions
moist heat injuries
injuries result from contact with hot liquid or steam.
contact burns
occur when hot metal, tar, or grease contacts the skin.
chemical burns
result from exposure to a caustic agent
cleaning agents
in the home (drain cleaner, oven cleaner, bleach) and agents in the industrial setting (caustic soda, sulfuric acid) can cause chemical burns.
thermal burns
result when clothes ignite from heat or flames that electrical sparks produce.
flash (arc burns)
result from contact with electrical current that travels through the air from one conductor to another.
conductive electric injury
results when a person touches electrical wiring or equipment.
radiation
burns most often result from therapeutic treatment for cancer or from sunburn.
severity of burn based off of
1st and 3rd degress
1st: sunburns
3rd: into tissue
rules of nines
Quick method to approximate the extent of burns by dividing the body into multiples of nine. The sum equals the TBSA.
emergent (resuscitative)
Begins with the injury and continues for 24 to 48 hr.
- Priorities include securing the airway, supporting circulation and organ perfusion by fluid replacement, managing pain, preventing infection through wound care, maintaining body temperature, and providing emotional support.
acute
rehabilative
older adults
Inhalation Injury: Clinical Findings
Carbon monoxide inhalation
(from burns in an enclosed area) findings include headache, weakness, dizziness, confusion, erythema (pink or cherry red skin) and upper airway edema, followed by sloughing of the respiratory tract mucosa
what can result from inhalation injury
Hypovolemia and shock can result from fluid shifts
Additional findings include hypotension, tachycardia, and decreased cardiac output.
Resuscitation phase on inhalation injury lab values:
glucose
bun
hct and hgb
sodium
potassium
Initial fluid shift (occurs in the first 12 hr and continues for 24 to 36 hr)
●Glucose: elevated due to stress
●BUN: elevated due to fluid loss
- Hct and Hgb: elevated (hemoconcentration) due to the loss of fluid volume and the fluid shift into the interstitial space (third spacing)
Electrolytes
- Sodium: decreased due to third spacing (hyponatremia)
- Potassium: increased due to cell destruction (hyperkalemia)
inhalation injury fluid remobilization labs:
hgb hct
sodium
potassium
wbc
blod glucose
abgs
total protein and albumin
(starts at about 24 hr; diuretic stage begins at 48 to 72 hr after injury)
% of head
9