ch 3 Flashcards

(34 cards)

1
Q

what is the normal tidal volume ranges in mL/kg of ideal body weight

A

7-9mL/kg ideal body weight

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
2
Q

define the normal inspiratory to expiratory (I:E) ratio and explain why expiration takes longer than inspiration

A

(1:2) duration of inhalation to exhalation

takes longer because its a passive process

*in copd pt ratio 1:3 (longer to exhale due to obstruction)

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
3
Q

what is subjective signs of dypnea

A

experiences felt/symptoms felt by pts

ex shortness of breath,labored breathing

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
4
Q

objective signs of dypnea

A

observable signs

ex. hyperventilation, tachypnea, use of accessory muscles, distress facial expressions, flared nostrils

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
5
Q

describe what happens when “demand to breath” exceeds “ capability to breath”?

A

respiratory system must work harder to supply O2 and remove CO2. cant meet metabolic needs

pt experiences dyspnea

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
6
Q

explain the relationship between lung compliance and elasticity.

A

high compliance=low elasticity, lungs are easily stretched but hard to go back to normal, causes prolonged exhalation

low compliance=high elasticity lungs are thick/stiff more effort to expand, causes rapid/shallow breathing

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
7
Q

what is lung compliance

A

lungs ability to stretch and expand

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
8
Q

what is elastance

A

(recoil) lungs need to go back to original form after being stretched

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
9
Q

which diseases are associated with increased lung compliance?

A

emphysema, COPD

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
10
Q

what diseases are associated with decreased lung compliance

A

pulmonary fibrosis, ARDS

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
11
Q

list 3 factors that can increase airway resistance

A

*changes in airway-secretions—>narrowing in airway
*changes in the wall of the airway-asthma, bronchoconstriction
*changes outside of the airway-tumors compressing the airways

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
12
Q

what is airway resistance (Raw)

A

resistance created by airway to the flow of gas

high=more obstruction or narrowing of airways-harder for air to pass

low=clear airways, air flows freely

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
13
Q

what are peripheral chemoreceptors

A

detect low O2(PaO2)levels and send signals to the medulla to increase RR and depth

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
14
Q

how do peripheral chemoreceptors respond to hypoxemia: include the crainal nerves

A

hypoxemia is detected–>peripheral chemoreceptors to activate–>send signals(afferent) via (carotid)glossopharyngeal nerve(IX) and (aortic)vagus nerve(X) –>to the medulla–>send commands (efferent) to the respiratory muscle–> tells the body to increase depth of breathing (RR)

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
15
Q

what is the difference in sensitivity between central and peripheral chemoreceptors?

A

peripheral= PaO2 (oxygen)
central=PaCO2 (carbon dioxide)*located in pons and medulla

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
16
Q

explain why patients with chronic COPD may rely on peripheral chemoreceptors to drive ventilation?

A

“hypoxic drove”-peripheral primary receptors for ventilation control in COPD pts that have a high PaCO2 and low PaO2, helps maintain O2 levels by adjusting breathing rate in fluctuating gas levels

17
Q

give two examples of acute onset respiratory conditions

A

(happens within mins/hr)
pneumonia, pulmonary edema & ARDS-rapid immediate medical attention

18
Q

give examples of chronic conditions?

A

(happens over days/months/yrs)
COPD, TB, pulmonary fibrosis- requires ongoing management of longterm complications

19
Q

which accessory muscles assist with inspiration

A

*pt has difficulty breathing and body trys to compensate to maintain ventilation
-scalene
-sternocleidomastoid
-pectoralis major
-trapezius muscle

20
Q

which accessory muscles assist with expiration

A

*pt has difficulty w/passive exhalation
-rectus abdominis
-external oblique
-internal oblique
-transversus abdominis

21
Q

what is the purpose of pursed lip breathing and how does it benefit pt with obstructive lung disease?

A

-offsets airway collapse and air trapping during exhalation
-(+) pressure creates airway stability
-slows ventilatory rate
-generates better gas mixing

22
Q

why is nasal flaring a concerning sign particularly in neonates?

A

neonates naturally are nose breathers so cant breath out of mouth, needs immediate intervention to prevent respiratory distress

23
Q

what is pleuritic chest pain and give one example

A

-sudden, sharp, or stabbing pain
-increases during deep inspiration and coughing
-ex:pneumonia, pneumothorax,tb, pleural effusion

24
Q

what is non pleuritic chest pain

A

-constant pain located centrally
-doesn’t worsen with deep inspiration
-pain may radiate
-ex: myocardial ischemia, pericardial inflammation

25
3 abnormal extremity findings associated with chronic respiratory or cardiac disorders
(chronic respiratory)-digital clubbing * swelling& rounding of finger&toes ( think ET fingers) (heart)- peripheral edema * swelling in extremities due to fluid in tissue (lower legs and ankles) (CHF) JVD-jugular vein distention
26
what information should be included when assessing sputum production
-sputum volume -appearance -odor -viscosity
27
define hemoptysis and list two causes
-coughing up blood or blood tinged sputum -causes: TB, bronchiectasis
28
list 3 common triggers of cough reflex
-inflammation -infectious agent -excessive secretions -noxious gases -obstruction of airway -mechanical stimulation (suctioning airway)
29
compare productive cough vs non productive cough
productive=produces sputum, clears secretion non productive= dry cough, unable to produce any sputum
30
a patient with low compliance what breathing pattern would you expect to observe?
rapid and shallow
31
you are observing a pt with severe asthma who is using accessory expiratory muscles, what does this indicate about airway resistance?
increased airway resistance
32
why might administering high concentration of O2 to chronic CO2 retain be dangerous
- can knock out hypoxic drive
33
if a neonate is displaying nasal flaring and intercostal retractions, what should your next step be?
-provide O2 support -suction nose -bronchodilator
34
a pt presents with sharp chest pain that worsens with inspiration. which conditions should you suspect
pleuric chest pain