How many lead needed for V4R
11
Main ions in cardiac myocyte
Potassium
Sodium
Calcium
Ions intracellular
High K
Low Na
Low Ca
Ions extracellular
Low K
High Na
High Ca
Stage 0
Cardiac Action Potential
Rapid depolarization caused by influx of sodium
Stage 1
Initial repolarization
Phase 2
Plateau phase due to slow Ca influx
Phase 3
Restores the resting potential
Repolarisation due to delayed rectifier K channels
Phase 4
Stable period - resting potential
P wave
Atrial contraction
Initiated from SA node
Proceeds inferiorly to AV node
First bit of P wave is R atrium, 2nd bit is Left atrium
PR
Signal down through bundles of His to ventricles
QRS Complex
Ventricular contraction
<120ms (3sq)
T wave
Ventricular repolarisation
T wave inversion V1-V3 until about 10 years ‘Juvenile T wave pattern’
U wave
Hypocalcaemia
Hypokalaemia
more visible in bradycardia
Tall P waves
P Pulmonale
Right atrial enlargement
<3sq normal
Wide P waves
Left Atrial enlargement
<90ms normal
Bifid P wave
Asynchrony between R + L atriums
Short PR
WPW - delta wave. Risk of VF
Glycogen storage disease
Long PR Interval
AV block (first degree)
e.g. in Ebsteins (half R ventricle functions as R atrium)
- ASD
Variable PR interval
Mobitz type 1 - type wone - widens (second degree heart block)
Wolf Parkinson White
Short PR
Associated with SVT (80%)
Verapamil and Digoxin contraindicated
Delta waves common (50%)
Lateral
I, aVL, V5, V6
Inferior
II, III, aVF
Anterior
V1,V2,V3, V4