Week 4 Flashcards

memory disorders (34 cards)

1
Q

example priming

A

when something in memory is closely associated with something else

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2
Q

brain structures

A

Different types of memory involve different brain structures

Damage to hippocampus
Poor episodic memory

Damage to frontotemporal para-hippocampal cortex
Poor semantic memory

Damage to both regions
Poor episodic + semantic memory

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3
Q

biological causes of amnesia

A

Surgery
Chronic alcohol abuse
Encephalitis
Brain tumours
Dementia
Closed head injury

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4
Q

types of amnesia

A

Retrograde
Anterograde
Global

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5
Q

retrograde amnesia

A

Poor recall for memories formed before onset of amnesia
Greater for episodic than semantic
temporal (time related) gradient

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6
Q

explanations for temporal gradient: consolidation theory

A

hippocampus leads to long lasting memory formation; consolidated memories stored elsewhere, protecting effects of hippocampus damage

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7
Q

explanations for temporal gradient: semanticisation

A

episodic memories become more like semantic memories over time = protected from effects of brain damage

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8
Q

explanations for temporal gradient: reduced learning opportunity

A

episodic memories depend on a single learning experience, reduced learning opportunity explains amnesia; semantic memories depend on several learning experiences

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9
Q

anterograde amnesia

A

Loss of ability to form new memories after onset of amnesia

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10
Q

global amnesia

A

Moderate retrograde amnesia + severe anterograde amnesia
Results from lesions of structures in medial temporal lobe, specifically the hippocampus

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11
Q

patient HM

A

Most studied amnesiac patient; suffered from severe epilepsy from 10yrs old
27 yrs old: surgery removed entire medial temporal lobe
Moderate retrograde + severe anterograde amnesia

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12
Q

Korsakoff’s Syndrome

A

Also known as diencephalic amnesia

Vitamin B1 deficiency from chronic alcoholism
Damage to mammillary bodies in hypothalamus
Poor ability to remember events both before and after; some new learning ability; slight impairment of STM

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13
Q

semantic dementia

A

Severe problems with semantic memory, but intact episodic memory
- Loss of info about meanings + facts
- Difficulty naming objects, single word comprehension, categorizing objects

Precise symptoms differ
Always involves degeneration of anterior (fronto)temporal lobe; where semantic memories are formed

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14
Q

dissociation

A

Identification of a brain region responsible for a cognitive process

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15
Q

single dissociation

A

Identify that brain damage to one structure disrupts one cognitive process, but not another

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16
Q

double dissociation

A

Identify that damage to one structure disrupts one cognitive process but not another
Identify that brain damage to different structure disrupts cognitive process Y but not X

17
Q

double dissociation example

A

Amnesia + semantic dementia point to double dissociation in LTM
Amnesia = hippocampus

Poor episodic memory (X)
Intact semantic memory (Y)

Semantic dementia = anterior/frontotemporal lobe
Poor semantic memory (Y)
Intact episodic memory (X)

18
Q

eyewitness testimony

A

the accuracy of an individual’s memory is sometimes of enormous importance

19
Q

decay

A

Memory trace fades over time
Less info available for retrieval as time passes

20
Q

interference

A

Similar info gets in the way of to-be-remembered info
Retroactive: recent info gets in the way of older info recall
Proactive: older info gets in the way of new info recall

21
Q

intrusions

A

mistakes in which elements not part of original memory trace get mixed up or intrude recall

22
Q

DRM paradigm

A

Named after Deese, Roediger & McDermott
Recall words semantically associated with list words that aren’t included in the list; leads to memory error
Background knowledge helps to link together the list of words according to a theme, which aids recall

23
Q

misinformation effect

A

Memories easily distorted by misleading info presented afterwards

Source misattribution
- Memory probe activates memory traces with overlapping info
- Need to decide on source of activated information
- Misattribution occurs when memories from one source resemble those from another

24
Q

Loftus + Palmer (1974)

A

Participants shown a film of a car accident
“How fast were the cards going when they […] into each other?”
SMASHED = 41mph
HIT = 34mph

One week later: “Did you see any broken glass?”
SMASHED = 34%
HIT = 14%

25
bias
Memory influenced by observer’s prior expectations Conformation bias Age Weapon focus Unconscious transference
26
conformation bias
Tendency to recall info in a way that confirms pre-existing beliefs Schemas can lead us to form specific expectations - Packets of knowledge stored in LTM - Causes us to reconstruct events based on what just have been true
27
Tuckey + Brewer (2003)
Participants shown film of a simulated bank robbery Recalled info relevant to bank robbery schema Less likely to recall info irrelevant to schema
28
face recognition
Eyewitness identification from line ups typically depends on face recognition Often fallible: 1. unconscious transference 2. other race effect 3. own age bias
29
unconscious transference
misidentify a familiar (but innocent) face as being the person responsible
30
other race effect
recognition for same-race faces generally more accurate than other-race faces
31
own age bias
more accurate when culprit is similar age to witness
32
anxiety + violence
Anxiety + stress generally impair memory Causes narrowing of attention on important stimuli Reduces ability to remember peripheral details
33
weapon focus effect
attend to weapon, reducing memory for other info Attend to unexpected stimuli inconsistent with schema of that situation
34
cognitive interview
strategies to aid retrieval 1. Mental reinstatement of the environment 2. Encourage the reporting of every detail 3. Describe the incident in several different orders 4. Reporting the incident from different viewpoints