Vestib 1 Practical COMP Flashcards

(76 cards)

1
Q

If you are looking at an ABR for retrocochlear and conductive, how do you tell the difference?

A

You can not tell from just the graph, you must do bone conduction or reflexes. If they add bone to the graph and it falls into the gray range, then it would be a conductive hearing loss because of the air-bone gap.

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
2
Q

Tell me how latency and amplitude change with intensity (ABR) ?

A

Louder Intensity = higher amplitude & shorter latency
Smaller Intensity = shorter amplitude & longer latency

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
3
Q

The latency of wave 5 between ears should differ by no more than?

A

0.2 to 0.4 msec

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
4
Q

Why is latency important?

A

it is the most robust parameter in the clinical interpretation of the ABR

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
5
Q

Review what the test results look like for an uncompensated vestibular loss case

A
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
6
Q

The ABR is not sensitive …

A

to all central nervous system disorders, it is only sensitive from ear to brainstem

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
7
Q

which tests require you to task?

A
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
8
Q

what is tasking?

A

“tasking” refers to keeping the patient mentally occupied — usually by giving them something to think about or say — while their eye movements (nystagmus) are being measured.

Because if the patient starts paying attention to their dizziness, they might suppress their nystagmus (especially during tests like calorics). This makes the response weaker or inaccurate.

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
9
Q

true or false: water caloric irrigations have greater variability and are more prone to operator error than air caloric irrigations?

A

false

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
10
Q

list 2 reasons why you might perform ice water caloric irrigations

A
  • may be used when traditional bi-thermal irrigations are very low or to help confirm diagnosis of bilateral loss
  • may be useful for confirmation of successful abalative procedures such as gentamycin injections or vestibular nerve section
    may be useful when there is a strong pre-existing spontaneous nystagmus (reversing the bias in the system)
  • may be useful to determine if there is any residual vestibular function in the system (even minimal) which - may help determine further course of treatment either surgically or rehab
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
11
Q

true or false: caloric reversal is the phenomena where oblique or vertical nystagmus is recorded with caloric irrigation

A

false - caloric perversion

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
12
Q

what is the caloric position? why do we place patients in the “caloric position?”

A

the caloric position (supine head elevated 30 degrees) brings the horizontal SCC into an orthogonal relationship with the gravity vector thereby ensuring maximum amount of stimulation during irrigation. perpendicular to the floor for max stimulation.

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
13
Q

what are the BSA recommended temperatues and irrigation time for AIR caloric irrigations. Warm air _____ degrees. Cold air ______ degrees. Irrigate for _____ seconds.

A

warm air = 50 degrees
cold air = 24 degrees
irrigate for = 60 sec.

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
14
Q

what are the BSA recommended temperatues and irrigation time for WATER caloric irrigations. Warm water _____ degrees. Cold water ______ degrees. Irrigate for _____ seconds.

A

warm air = 44 degrees
cold air = 30 degrees
irrigate for = 30 sec.

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
15
Q

what does COWS stand for? what does it mean?

A

COWS = Cold Opposite Warm Same

a cold irrigation will produce a nystagmus that beats in the opposite direction of the ear being stimulated and warm irrigation will produce a nystagmus that beats in the same direction of the ear being stimulated. this has to do with the direction of endolymph flow (ampullopetal=less density = excitatory flow) (ampullofugal = more dense = inhibitory flow). the stimulus doesnt transfer to the other side of the head. it only tells you direction of the nystagmus based upon endolymph movement toward or away from ampullla (occurs on same side as irrigation).

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
16
Q

what is the equation for unilateral weakness?

A

(RW+RC) - (LW+LC) ÷ (RW+RC+LW+LC) x 100

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
17
Q

what is the equation for directional preponderance?

A

(RW+LC) - (LW+RC) ÷ (RW+RC+LW+LC) x 100

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
18
Q

what are the vestibular nerve and canal connections?

A

SVN: lateral and anterior canals
IVN: posterior canals

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
19
Q

what is normal for unilateral weakness

A

below 20-25% is normal for unilateral weakness

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
20
Q

what is normal for directional preponderance?

A

below 30-35% is normal for directional preponderance

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
21
Q

what is abnormal for total eye speed?

A

anything less than 26

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
22
Q

whats alexanders phenomenon?

A

when the patients nystagmus increases when they look the same way of the beating & decreases or is eliminated when they look the opposite way. in order for it to be alexanders law they MUST have central gaze nystagmus

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
23
Q

what is the epley

A

most treatment for canalithiasis AC/PC BPPV

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
24
Q

what is gain? (vestib)

A

eye movement relative to something else

head impulse →eye movement relative to head movement

rotary → eye movement relative to chair movement

pursuits → eye movement relative to target moving in front

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
25
tell me about the romberg
screens the body’s proprioception (somatosensory) system and the amount of disequalibrium caused by central vertigo, peripheral vertigo, and head trauma. the patient stands with their feet together and arms out to the side, we ask them to stand still for 10-15 seconds first with eyes open them again with their eyes closed. If you want to make it tandem they put one foot infront of the other.
26
what does a positive romberg mean? (what will you see the patient do)
it means the patient swayed or fell with non-fluid motions. they will typically lean toward the side of the problem.
27
what is the dix-hallpike
a test that assess for PC/AC BPPV
28
what is the supine head roll
it is a positional test that assesses for HC BPPV
29
what is the static positional nystagmus criteria ?criteria needed
Nystagmus that changes direction in any head position It is present in at least 3 head positions It is intermittent in all head positions (little here, little there) One or more positions that has a slow phase magnitude (velocity) >/=4-6 deg/s
30
what is the semont
treatment for cupulolithiasis AC/PC BPPV
31
what is the BBQ roll?
treatment for horizontal canal BPPV used for the geotropic/canalithiasis variant
32
what is the gufoni
treatment for horizontal canal BPPV used for the ageotropic/cupulolithiasis variant
33
what is the difference between canalithiasis and cupulothiasis symptoms
canalithiasis is a delayed onset and lasts for less than a minute cupulothiasis is an immediate onset and doesn’t fatigue
34
if you are performing dix hallpike and see Delayed onset, upward & rightward torsional fast phases, & <1min response duration, what type of BPPV is it and what is the treatment?
canalithiasis, posterior SCC treatment = epley
35
if you are performing dix hallpike and see Immediate onset, upward & rightward torsional fast phases, >1min response duration, what type of BPPV is it and what is the treatment?
cupulothiasis, posterior SCC treatment = semont
36
if you are performing dix hallpike and see Delayed onset, downward & leftward torsional fast phases, <1min response duration, what type of BPPV is it and what is the treatment?
canalithiasis, anterior SCC treatment = epley
37
if you are performing dix hallpike and see Immediate onset, downward & leftward torsional fast phases, >1min response duration, what type of BPPV is it and what is the treatment?
cupulothiasis, anterior SCC treatment = semont
38
if you are performing the supine head roll and see Horizontal, geotropic fast phases (toward down ear), stronger w/ R ear down, may have delayed onset & will fatigue over time, what type of BPPV is it and what is the treatment?
canalithiasis, horizontal SCC treatment = BBQ roll
39
if you are performing the supine head roll and see, Horizontal, ageotropic fast phases(beats away from ear down), stronger w/ L ear down, more immediate onset & longer response duration, what type of BPPV is it and what is the treatment?
cupulothiasis, horizontal SCC treatment = modified semont
40
how do you interpret calorics
41
what does the fukuda/stepping fukuda assess?
screens laryrinthine function via vestibulospinal reflexes
42
how to you perform the fukuda/stepping fukuda?
tell the patient to hold their arms out straight in from of them and close their eyes, then ask them to march in place for 50 steps
43
how do you interpret the fukuda/stepping fukuda results?
if the patient is normal they wont rotate more than 45 degrees. if the patient is abnoral they will rotate more than 45 degrees and this is considered a positive fukuda
44
how to you perform the active/passive headshake test?
the patient has their eyes closed or is vision denied under goggles. shake their head back and forth and after 20 sec have them open their eyes and watch for nystagmus
45
how do you interpret the results of the active/passive headshake?
if they are normal you will not see nystagmus, if they are abnormal nystagmus will be present after the headhshake or enhanced
46
what is ocular tilt?
47
how do you perform the dynamic visual acuity test?
the patient stands the proper distance from the eye chart (letters) and you ask them to read the lowest line they can. then stand behind them and move their head back and forth and ask them to read the lowest line possible while you move their head.
48
how to interpret the results of the dynamic visual acuity test?
if there was no line change or a slight line change they are normal indicating their VOR is functioning properly. if they have a line change of 2 or greater they are abnormal, indicating oscillopsia
49
what does dynamic visual acuity assess?
screens for oscillopsia (often caused by vestibular loss). looks at the VOR
50
what do we look at during saccades
gain velocity acuracy
51
what do we look at during smooth pursuits
gain symmetry
52
What does the dix hallpike assess?
anterior and posterior canal BPPV abnormal = torsional nystagmus
53
What are the treatments for BPPV?
habituation techniques & excersises particle repositioning maneuvers surgical intervention (rare)
54
What is direction changing nystagmus?
key points is that it beats in one direction. Slows down and stops. Goes in the other direction. BUT it has to happen just in center gaze they cannot be moving positions and that happen or its something else
55
What are the tests used for central findings?
oculomotor testing ENG/VNG having them fixate if they come in with center nystagmus
56
What are the positional nystagmus criteria that make it pathological (abnormal) ?
it changes direction in any head position, or it is persistent in at least 3 head positions, or it is intermittent in all head positions, or it’s slow phase velocity exceeds 6 degree/sec in any head position
57
What are saccades?
rapid shift in gaze, saccadic testing is the ability to track a fast moving target
58
what is torsional/rotary nystagmus?
when the eyes rotate about the central axis of the globe (twist in circular motion). pure torsional is a sign of central problems but if we see it with positional testing it indicates a peripheral finding.
59
nystagmus will beat?
away from the affected side
60
how do you perform the halmalgi head thrust?
hold the patients head and instruct them to keep their eyes open during the test. then slowly move their head back and forth, then rapidly move the head to each side. after the rapid movement hold it there briefly to check for refixation saccades
61
how do you interpret the results of the halmalgi head thrust?
if they are normal there will be no corrective saccades, if they are abnormal corrective saccades will be present. you will have refixation saccades in the direction of the damage
62
what does the halmalgi head thrust assess?
screens SCC dysfunction in all canals & the detection of peripheral vesibulopathy
63
what is caloric testing?
the most informative subtest of the VNG by being able to excite only 1 vestibular organ while the other one stays at rest
64
what is directional preponderance?
comparing the slow phase velocity (SPV) of eye movements in the same direction, so using RW and LC to compare to LW and RC -typically seen with a preexisting spontaneous nystagmus
65
video head impulse (vHIT) (halmagli made)
instrumented version of the bedside technique used to diagnose reduction in vestibular function -can detect SCC dysfunction in all canals and is useful for the detection of peripheral vestibulopathy
66
advantages of vHIT
can be performed from 10 months to elderly, gives a real world measure, evaluates HF reactivity, gives canal specific information, high sensitivity to vestibulopathy, can help track VRT progress and is site specific
67
two key aspects to remember when conducting vHIT
use quick acceleration and keep it unpredictable
67
limitations of vHIT
only analyses HF deficits, some studies suggest insensitvity to dysfunction secondary to menieres disease, techniques can be challenging, will miss mild unilateral vestibular origins
68
2 parameters for vHIT
gain and presence of refixation saccades
69
common peripheral signs
sudden onset, intense severity, paroxysmal and intermittent pattern, worse with movement, will often show nausea, horizontal nystagmus, fatigue of signs, HL/tinnitus may occur
70
common central signs
sudden or gradual onset, less intense severity, constant pattern, variability nausea, multi directional or vertical nystagmus, no fatigue of signs, typically no HL/tinnitus, CNS signs are typically present (such as headaches, changes in speech)
71
with rotational testing, we are looking at ….
how the VOR functions in regard to movements of the individual -observing the function through head and eye velocity movement
72
what are signs of insufficient VOR
nystagmus, oscillopsia, retinal slip
73
what are the slow and fast phase of nystagmus driven by
slow phase is driven by the ears = peripheral vestibular fast phase is driven by the CNS = central
74
when looking at a nystagmus video on a screen what do you need to remember
our left is the patients right the video is opposite
75