Endocrine System Flashcards

(31 cards)

1
Q

Dietary trace element required for thyroid hormone synthesis; deficiency enlarges thyroid

A

Iodine

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2
Q

Testosterone

A

Primary androgen responsible for development of male sexual characteristics.

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3
Q

Principal adrenal medulla catecholamine; acute stress response.

A

Adrenaline (Epinephrine)

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4
Q

What hormone increases blood calcium by mobilizing calcium from bone and enhancing reabsorption?

A

Parathormone (PTH)

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5
Q

What posterior pituitary hormone stimulates uterine contractions in labor?

A

Oxytocin

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6
Q

What promotes breast tissue development and milk secretion?

A

Prolactin

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7
Q

What gland regulates Ca exchange bone↔blood and raises serum Ca; and what is the hormone associated with the gland?

A

Parathyroid, parathormone(PTH)

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8
Q

_____ needs idoine, if there is a deficiency the goiter enlarges. Hyperstate of the deficiency often shows irritability and the most common hormone associated with this gland is ______?

A

Thyroid, thyroxine(T4)

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9
Q

Main function of _____ is blood sugar control?

A

Pancreatic Islets

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10
Q

What hormones are associated with the Pancreatic Islets?

A

Insulin and glucagon

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11
Q

_____ principal hormone = adrenaline (emergency “fight or flight”).

A

Adrenal Medulla

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12
Q

Master gland; location helps with imaging & neuro sx correlation and has multiple tropic hormones.

A

Pituitary gland (attached to brain)

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13
Q

What gland’s key function is: anatomy clue for imaging

A

Adrenals (atop kidneys)

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14
Q

3 hormones associated with the Adrenals

A

Cortisol, aldosterone, and catecholamines

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15
Q

Prolactin develops breast tissue & stimulates milk; what gland is associated with prolactin and FSH.

A

Anterior Pituitary Gland

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16
Q

Develops primary male sexual characteristics, Testosterone

A

Male Gonads (Testes)

17
Q

Thyroid enlarges when iodine is lacking in diet or environment—watch for neck fullness, dysphagia complaints.

A

Goiter (iodine deficiency)

18
Q

Muscle spasms/cramps; treated with calcium. Recognize in postsurgical parathyroid cases.

A

Tetany (hypocalcemia)

19
Q

Insulin deficiency from destruction of pancreatic β cells; affects urinary (polyuria), endocrine, and circulatory systems over time.

A

Diabetes Mellitus

20
Q

Palpitations, tachycardia, cardiomegaly common; patients often report irritability.

A

Graves’ Disease (hyperthyroidism)

21
Q

Most commonly due to autoimmune destruction of hormone‑producing cells. Monitor for fatigue, weight changes, hypotension; labs per protocol.

A

Adrenal Insufficiency (e.g., Addison disease)

22
Q

Severe hypothyroid state—results from too little thyroid hormone. Note mental slowing, skin changes, facial puffiness.

23
Q

Overgrowth of cartilaginous & connective tissues from excess growth hormone (usually pituitary). Watch for enlarged hands/feet, jaw.

24
Q

__________ assesses pancreatic endocrine function; critical in diabetes workups and medication adjustment.

A

Blood sugar testing

25
______ evaluates thyroid gland function; part of hyperthyroid vs. thyroiditis evaluation pathways.
Radioactive iodine uptake (RAIU)
26
Vomiting, pregnancy, and exercise can all change needs; weather doesn’t directly affect insulin dose. (Illness may change patterns indirectly—check protocol.)
Insulin dosing factors
27
Glucagon release signals the liver to dump stored glycogen—why glucagon emergency kits matter in insulin‑treated patients.
Low blood sugar response
28
What cell produces insulin and majority of islets are these cells?
β cells
29
What cells produce glucagon?
α cells
30
Glucagon causes liver to release stored _____ when BS is ____.
glycogen, low
31
________ is a chronic condition in which your adrenal glands don't produce enough of the hormones cortisol and aldosterone.
Addison's disease