Congestive Heart Failure (CHF):
A chronic condition where the heart fails to pump sufficient blood to meet the body’s needs, resulting in fluid accumulation in tissues (edema) or body cavities (effusions).
Preload
amount of blood in the ventricles at the end of diastole (filling phase). Increased
preload stretches the myocardium and affects contractility.
Afterload
The resistance the heart must overcome to eject blood during systole (contraction
phase).
Myocardial Contractility (Inotropism)
The heart muscle’s ability to contract and generate force. Dilated Cardiomyopathy
Systolic Dysfunction
Impaired heart contraction, leading to reduced cardiac output.
Diastolic Dysfunction:
Impaired heart relaxation during diastole, reducing ventricular filling. Hypertrophic Cardiomyopathy
Dilated Cardiomyopathy (DCM):
the heart’s chambers (primarily the
ventricles) enlarge, and the myocardial walls become thin and weak, reducing the heart’s
ability to pump blood effectively.
Hypertrophic Cardiomyopathy (HCM):
thickening (hypertrophy) of the heart muscle, particularly the left ventricle, which impairs the heart’s ability to relax and fill appropriately during diastole.
Cardiac Output (CO):
blood volume the heart pumps per minute. CO = Stroke Volume ×
Heart Rate.
Hypertrophy:
Thickening of the heart muscle as a compensatory response to increased
workload.
Backward Failure:
inability of the heart to adequately empty during systole, leading to
venous congestion and fluid accumulation (e.g., pulmonary edema, ascites).
Forward Failure
Inadequate blood supply to tissues due to reduced cardiac output, leading to
poor perfusion (e.g., hypotension, weakness).
Pulmonary Edema
Fluid accumulation within the lungs, typically due to left-sided heart
failure.
Ascites
Fluid accumulation within the abdominal cavity, often associated with right-sided
heart failure.
Pleural Effusion:
Fluid accumulation in the pleural cavity (space surrounding the lungs).
Seen in: Cats with CHF.
. Renin-Angiotensin-Aldosterone System (RAAS)
A hormonal system activated in response
to decreased cardiac output, leading to fluid retention and vasoconstriction.
. Sympathetic Nervous System (SNS) Activation:
The body’s response to decreased cardiac
output, increasing heart rate, and contractility.
Edema:
Fluid accumulation in tissues, typically in the lungs (pulmonary edema) or extremities
(peripheral edema).
Valve Regurgitation
Incompetence of heart valves leading to backward blood flow
Endocardiosis:
A degenerative condition of heart valves, especially the mitral valve, leading
to regurgitation and CHF
Ventricular Remodeling:
Structural changes in the heart in response to chronic stress, including dilation or hypertrophy.
Cardiogenic Shock:
A severe form of heart failure with a critical reduction in cardiac output,
leading to inadequate perfusion of vital organs.
Furosemide
A diuretic used to manage pulmonary edema by reducing fluid overload.
Pimobendan
An inodilator (positive inotrope and vasodilator) that improves heart contractility
and reduces afterload.